Putka相关文献与解析，生知库 | 链接生命科学的每一步

ASOs are an effective treatment for disease-associated oligodendrocyte signatures in premanifest and symptomatic SCA3 mice.

ASO 可有效治疗 SCA3 前驱期和症状期小鼠的疾病相关少突胶质细胞特征

doi:10.1016/j.ymthe.2024.02.033

Schuster Kristen H, Zalon Annie J, DiFranco Danielle M, Putka Alexandra F, Stec Nicholas R, Jarrah Sabrina I, Naeem Arsal, Haque Zaid, Zhang Hanrui, Guan Yuanfang, McLoughlin Hayley S

细胞生物学

发表日期：2024

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Revisiting Glutamate Excitotoxicity in Amyotrophic Lateral Sclerosis and Age-Related Neurodegeneration

重新审视肌萎缩侧索硬化症和年龄相关性神经退行性疾病中的谷氨酸兴奋性毒性

期刊:International Journal of Molecular Sciences

影响因子:4.9

doi:10.3390/ijms25115587

Arnold, Frederick J; Putka, Alexandra F; Raychaudhuri, Urmimala; Hsu, Solomon; Bedlack, Richard S; Bennett, Craig L; La Spada, Albert R

Myelinating Glia: Potential Therapeutic Targets in Polyglutamine Spinocerebellar Ataxias

髓鞘形成胶质细胞：多聚谷氨酰胺脊髓小脑性共济失调的潜在治疗靶点

期刊:Cells

影响因子:5.2

doi:10.3390/cells12040601

Putka, Alexandra F; Mato, Juan P; McLoughlin, Hayley S

Disease-associated oligodendrocyte signatures are spatiotemporally dysregulated in spinocerebellar ataxia type 3

疾病相关的少突胶质细胞特征在脊髓小脑共济失调 3 型中时空失调

期刊:Frontiers in Neuroscience

影响因子:3.2

doi:10.3389/fnins.2023.1118429

Kristen H Schuster, Danielle M DiFranco, Alexandra F Putka, Juan P Mato, Sabrina I Jarrah, Nicholas R Stec, Vikram O Sundararajan, Hayley S McLoughlin

Pathogenetic Mechanisms Underlying Spinocerebellar Ataxia Type 3 Are Altered in Primary Oligodendrocyte Culture.

原代少突胶质细胞培养改变了脊髓小脑性共济失调3型的发病机制

期刊:Cells

影响因子:5.2

doi:10.3390/cells11162615

Schuster Kristen H, Putka Alexandra F, McLoughlin Hayley S

细胞生物学

发表日期：2022

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An unusual cause of acute anemia in an immunosuppressed patient

免疫抑制患者急性贫血的一种罕见病因

期刊:Gastroenterology Report

影响因子:4.2

doi:10.1093/gastro/gov005

Modaresi Esfeh, Jamak; Jackson, Whitney; Ansari-Gilani, Kianoush; Putka, Brian

Cerebellar lipid dysregulation in SCA3: A comparative study in patients and mice

SCA3患者小脑脂质代谢紊乱：患者与小鼠的比较研究

ASOs are an effective treatment for disease-associated oligodendrocyte signatures in premanifest and symptomatic SCA3 mice.

ASO 可有效治疗 SCA3 前驱期和症状期小鼠的疾病相关少突胶质细胞特征

Revisiting Glutamate Excitotoxicity in Amyotrophic Lateral Sclerosis and Age-Related Neurodegeneration

重新审视肌萎缩侧索硬化症和年龄相关性神经退行性疾病中的谷氨酸兴奋性毒性

Myelinating Glia: Potential Therapeutic Targets in Polyglutamine Spinocerebellar Ataxias

髓鞘形成胶质细胞：多聚谷氨酰胺脊髓小脑性共济失调的潜在治疗靶点

Disease-associated oligodendrocyte signatures are spatiotemporally dysregulated in spinocerebellar ataxia type 3

疾病相关的少突胶质细胞特征在脊髓小脑共济失调 3 型中时空失调

Pathogenetic Mechanisms Underlying Spinocerebellar Ataxia Type 3 Are Altered in Primary Oligodendrocyte Culture.

原代少突胶质细胞培养改变了脊髓小脑性共济失调3型的发病机制

An unusual cause of acute anemia in an immunosuppressed patient

免疫抑制患者急性贫血的一种罕见病因