Qin Luye相关文献与解析，生知库 | 链接生命科学的每一步

A sexually dimorphic signature of activity-dependent BDNF signaling on the intrinsic excitability of pyramidal neurons in the prefrontal cortex

前额叶皮层锥体神经元固有兴奋性中，活动依赖性 BDNF 信号传导的性别二态性特征

期刊:Frontiers in Cellular Neuroscience

影响因子:4

doi:10.3389/fncel.2024.1496930

Ma, Kaijie; Zhang, Daoqi; McDaniel, Kylee; Webb, Maria; Newton, Samuel S; Lee, Francis S; Qin, Luye

Chemogenetic Inhibition of Prefrontal Cortex Ameliorates Autism-Like Social Deficits and Absence-Like Seizures in a Gene-Trap Ash1l Haploinsufficiency Mouse Model.

化学遗传学抑制前额皮质可改善基因捕获 Ash1l 单倍体不足小鼠模型中的自闭症样社交缺陷和失神性癫痫发作

期刊:Genes

影响因子:2.8

doi:10.3390/genes15121619

Ma Kaijie, McDaniel Kylee, Zhang Daoqi, Webb Maria, Qin Luye

Diminished activity-dependent BDNF signaling differentially causes autism-like behavioral deficits in male and female mice

活动依赖性 BDNF 信号传导减弱对雄性和雌性小鼠造成不同的自闭症样行为缺陷。

期刊:Frontiers in Psychiatry

影响因子:3.2

doi:10.3389/fpsyt.2023.1182472

Ma, Kaijie; Taylor, Connie; Williamson, Mark; Newton, Samuel S; Qin, Luye

Dopamine Differentially Regulates Response Dynamics of Prefrontal Cortical Principal Neurons and Interneurons to Optogenetic Stimulation of Inputs from Ventral Tegmental Area

多巴胺差异性地调节前额皮质主要神经元和中间神经元对来自腹侧被盖区输入的遗传光刺激的反应动力学

期刊:Cerebral Cortex

影响因子:2.9

doi:10.1093/cercor/bhaa027

Zhong, Ping; Qin, Luye; Yan, Zhen

神经科学

发表日期：2020

文献求助收藏

Chemogenetic Activation of Prefrontal Cortex in Shank3-Deficient Mice Ameliorates Social Deficits, NMDAR Hypofunction, and Sgk2 Downregulation

化学遗传学方法激活Shank3缺陷小鼠的前额叶皮层可改善其社交缺陷、NMDAR功能低下和Sgk2下调。

期刊:iScience

影响因子:4.1

doi:10.1016/j.isci.2019.06.014

Qin, Luye; Ma, Kaijie; Yan, Zhen

发表日期：2019

文献求助收藏

Histone deacetylase inhibitor MS-275 restores social and synaptic function in a Shank3-deficient mouse model of autism.

组蛋白去乙酰化酶抑制剂 MS-275 可恢复 Shank3 缺陷型自闭症小鼠模型的社交和突触功能

期刊:Neuropsychopharmacology

影响因子:7.1

doi:10.1038/s41386-018-0073-1

Ma Kaijie, Qin Luye, Matas Emmanuel, Duffney Lara J, Liu Aiyi, Yan Zhen

The ADHD-linked human dopamine D4 receptor variant D4.7 induces over-suppression of NMDA receptor function in prefrontal cortex.

与 ADHD 相关的人类多巴胺 D4 受体变体 D4.7 会过度抑制前额皮质中的 NMDA 受体功能

期刊:Neurobiology of Disease

影响因子:5.6

doi:10.1016/j.nbd.2016.07.024

Qin Luye, Liu Wenhua, Ma Kaijie, Wei Jing, Zhong Ping, Cho Kei, Yan Zhen

Autism-like Deficits in Shank3-Deficient Mice Are Rescued by Targeting Actin Regulators

通过靶向肌动蛋白调节因子可以挽救Shank3缺陷小鼠的自闭症样缺陷

期刊:Cell Reports

影响因子:6.9

doi:10.1016/j.celrep.2015.04.064

Duffney, Lara J; Zhong, Ping; Wei, Jing; Matas, Emmanuel; Cheng, Jia; Qin, Luye; Ma, Kaijie; Dietz, David M; Kajiwara, Yuji; Buxbaum, Joseph D; Yan, Zhen

An adaptive role for BDNF Val66Met polymorphism in motor recovery in chronic stroke

BDNF Val66Met多态性在慢性卒中运动功能恢复中的适应性作用

期刊:Journal of Neuroscience

影响因子:4

doi:10.1523/JNEUROSCI.4140-13.2014

Qin, Luye; Jing, Deqiang; Parauda, Sarah; Carmel, Jason; Ratan, Rajiv R; Lee, Francis S; Cho, Sunghee

BDNF

发表日期：2014

文献求助收藏

Kmt2c/Mll3 Haploinsufficiency Causes Autism-like Behavioral Deficits in Mice

Kmt2c/Mll3单倍体不足导致小鼠出现类似自闭症的行为缺陷

A sexually dimorphic signature of activity-dependent BDNF signaling on the intrinsic excitability of pyramidal neurons in the prefrontal cortex

前额叶皮层锥体神经元固有兴奋性中，活动依赖性 BDNF 信号传导的性别二态性特征

Chemogenetic Inhibition of Prefrontal Cortex Ameliorates Autism-Like Social Deficits and Absence-Like Seizures in a Gene-Trap Ash1l Haploinsufficiency Mouse Model.

化学遗传学抑制前额皮质可改善基因捕获 Ash1l 单倍体不足小鼠模型中的自闭症样社交缺陷和失神性癫痫发作

Diminished activity-dependent BDNF signaling differentially causes autism-like behavioral deficits in male and female mice

活动依赖性 BDNF 信号传导减弱对雄性和雌性小鼠造成不同的自闭症样行为缺陷。

Dopamine Differentially Regulates Response Dynamics of Prefrontal Cortical Principal Neurons and Interneurons to Optogenetic Stimulation of Inputs from Ventral Tegmental Area

多巴胺差异性地调节前额皮质主要神经元和中间神经元对来自腹侧被盖区输入的遗传光刺激的反应动力学

Chemogenetic Activation of Prefrontal Cortex in Shank3-Deficient Mice Ameliorates Social Deficits, NMDAR Hypofunction, and Sgk2 Downregulation

化学遗传学方法激活Shank3缺陷小鼠的前额叶皮层可改善其社交缺陷、NMDAR功能低下和Sgk2下调。

Histone deacetylase inhibitor MS-275 restores social and synaptic function in a Shank3-deficient mouse model of autism.

组蛋白去乙酰化酶抑制剂 MS-275 可恢复 Shank3 缺陷型自闭症小鼠模型的社交和突触功能

The ADHD-linked human dopamine D4 receptor variant D4.7 induces over-suppression of NMDA receptor function in prefrontal cortex.

与 ADHD 相关的人类多巴胺 D4 受体变体 D4.7 会过度抑制前额皮质中的 NMDA 受体功能

Autism-like Deficits in Shank3-Deficient Mice Are Rescued by Targeting Actin Regulators

通过靶向肌动蛋白调节因子可以挽救Shank3缺陷小鼠的自闭症样缺陷

An adaptive role for BDNF Val66Met polymorphism in motor recovery in chronic stroke

BDNF Val66Met多态性在慢性卒中运动功能恢复中的适应性作用