miR-223-3p improves macrophage polarization balance and mitochondrial metabolism to ameliorate uveitis by orchestrating P65(Lys310) acetylation through RBPJ/HDAC1 axis
miR-223-3p通过RBPJ/HDAC1轴调控P65(Lys310)乙酰化,从而改善巨噬细胞极化平衡和线粒体代谢,进而缓解葡萄膜炎。
期刊:Cellular & Molecular Biology Letters
影响因子:10.2
doi:10.1186/s11658-026-00874-3
Peng, Yuan; Liu, Bin; Xu, Shuqin; Qu, Ruyi; Zhang, Ruixue; Wang, Congling; Liu, Yunfeng; Wei, Huixia; Tian, Qingmei; Zhang, Miao; Bi, Hongsheng; Yin, Xuewei; Guo, Dadong
巨噬细胞
HDAC1
表观遗传
巨噬细胞
信号转导
代谢
线粒体
乙酰化
细胞生物学
免疫/内分泌