Quitterer相关文献与解析，生知库 | 链接生命科学的每一步

BBLN triggers CAMK2D pathology in mice under cardiac pressure overload and potentially in unrepaired hearts with tetralogy of Fallot

BBLN 可引发心脏压力超负荷小鼠的 CAMK2D 病理，并可能引发法洛四联症未修复心脏的 CAMK2D 病理

期刊:Nature Cardiovascular Research

影响因子:9.4

doi:10.1038/s44161-023-00351-6

Joshua Abd Alla, Andreas Langer, Stefan Wolf, Xuebin Fu, Mohamed Abdelfattah Rageh, Ursula Quitterer

The RAF Kinase Inhibitor Protein (RKIP): Good as Tumour Suppressor, Bad for the Heart

RAF激酶抑制蛋白（RKIP）：作为肿瘤抑制剂有益，但对心脏有害

期刊:Cells

影响因子:5.2

doi:10.3390/cells11040654

Abd Alla, Joshua; Quitterer, Ursula

Pathological AT1R-B2R Protein Aggregation and Preeclampsia

病理性AT1R-B2R蛋白聚集与先兆子痫

期刊:Cells

影响因子:5.2

doi:10.3390/cells10102609

Quitterer, Ursula; AbdAlla, Said

发表日期：2021

文献求助收藏

Stearoyl-CoA Desaturase (SCD) Induces Cardiac Dysfunction with Cardiac Lipid Overload and Angiotensin II AT1 Receptor Protein Up-Regulation

硬脂酰辅酶 A 去饱和酶 (SCD) 诱发心脏功能障碍，导致心脏脂质超负荷和血管紧张素 II AT1 受体蛋白上调

期刊:International Journal of Molecular Sciences

影响因子:0.7

doi:10.3390/ijms22189883

Joshua Abd Alla, Yahya F Jamous, Ursula Quitterer

Beta-Arrestin1 Prevents Preeclampsia by Downregulation of Mechanosensitive AT1-B2 Receptor Heteromers

Beta-Arrestin1 通过下调机械敏感性 AT1-B2 受体异聚体来预防先兆子痫

期刊:Cell

影响因子:45.5

doi:10.1016/j.cell.2018.10.050

Ursula Quitterer, Xuebin Fu, Armin Pohl, Karam M Bayoumy, Andreas Langer, Said AbdAlla

信号转导

发表日期：2019

文献求助收藏

Increased Reactive Oxygen Species Generation Contributes to the Atherogenic Activity of the B2 Bradykinin Receptor

活性氧生成增加促进 B2 缓激肽受体的致动脉粥样硬化活性

期刊:Frontiers in Medicine

影响因子:

doi:10.3389/fmed.2019.00032

Alexander Perhal, Stefan Wolf, Yahya F Jamous, Andreas Langer, Joshua Abd Alla, Ursula Quitterer

Sensitization of the Angiotensin II AT1 Receptor Contributes to RKIP-Induced Symptoms of Heart Failure

血管紧张素 II AT1 受体的敏化导致 RKIP 诱发心力衰竭症状

期刊:Frontiers in Medicine

影响因子:

doi:10.3389/fmed.2018.00359

Stefan Wolf, Joshua Abd Alla, Ursula Quitterer

Inhibition of G-protein-coupled Receptor Kinase 2 Prevents the Dysfunctional Cardiac Substrate Metabolism in Fatty Acid Synthase Transgenic Mice

蛋白偶联受体激酶 2 的抑制可防止脂肪酸合酶转基因小鼠的心脏底物代谢功能障碍

期刊:Journal of Biological Chemistry

影响因子:4

doi:10.1074/jbc.M115.702688

Joshua Abd Alla, Muriel Graemer, Xuebin Fu, Ursula Quitterer

代谢

发表日期：2016

文献求助收藏

Microarray gene expression profiling reveals antioxidant-like effects of angiotensin II inhibition in atherosclerosis

微阵列基因表达谱分析揭示血管紧张素II抑制剂在动脉粥样硬化中具有抗氧化样作用

期刊:Frontiers in Physiology

影响因子:3.4

doi:10.3389/fphys.2013.00148

Abd Alla, Joshua; El Faramawy, Yasser; Quitterer, Ursula

Analysis of GRK2 aggregation in the pathology of Alzheimer disease in animal models

动物模型中阿尔茨海默病病理学中GRK2聚集的分析

BBLN triggers CAMK2D pathology in mice under cardiac pressure overload and potentially in unrepaired hearts with tetralogy of Fallot

BBLN 可引发心脏压力超负荷小鼠的 CAMK2D 病理，并可能引发法洛四联症未修复心脏的 CAMK2D 病理

The RAF Kinase Inhibitor Protein (RKIP): Good as Tumour Suppressor, Bad for the Heart

RAF激酶抑制蛋白（RKIP）：作为肿瘤抑制剂有益，但对心脏有害

Pathological AT1R-B2R Protein Aggregation and Preeclampsia

病理性AT1R-B2R蛋白聚集与先兆子痫

Stearoyl-CoA Desaturase (SCD) Induces Cardiac Dysfunction with Cardiac Lipid Overload and Angiotensin II AT1 Receptor Protein Up-Regulation

硬脂酰辅酶 A 去饱和酶 (SCD) 诱发心脏功能障碍，导致心脏脂质超负荷和血管紧张素 II AT1 受体蛋白上调

Beta-Arrestin1 Prevents Preeclampsia by Downregulation of Mechanosensitive AT1-B2 Receptor Heteromers

Beta-Arrestin1 通过下调机械敏感性 AT1-B2 受体异聚体来预防先兆子痫

Increased Reactive Oxygen Species Generation Contributes to the Atherogenic Activity of the B2 Bradykinin Receptor

活性氧生成增加促进 B2 缓激肽受体的致动脉粥样硬化活性

Sensitization of the Angiotensin II AT1 Receptor Contributes to RKIP-Induced Symptoms of Heart Failure

血管紧张素 II AT1 受体的敏化导致 RKIP 诱发心力衰竭症状

Inhibition of G-protein-coupled Receptor Kinase 2 Prevents the Dysfunctional Cardiac Substrate Metabolism in Fatty Acid Synthase Transgenic Mice

蛋白偶联受体激酶 2 的抑制可防止脂肪酸合酶转基因小鼠的心脏底物代谢功能障碍

Microarray gene expression profiling reveals antioxidant-like effects of angiotensin II inhibition in atherosclerosis

微阵列基因表达谱分析揭示血管紧张素II抑制剂在动脉粥样硬化中具有抗氧化样作用