Rahat Michal A相关文献与解析，生知库 | 链接生命科学的每一步

The Anti-EMMPRIN Monoclonal Antibody hMR18-mAb Induces Tumor Dormancy and Inhibits the EMT Process in Human Carcinoma Cell Lines Co-Cultured with Macrophages

抗EMMPRIN单克隆抗体hMR18-mAb可诱导肿瘤休眠并抑制与巨噬细胞共培养的人类癌细胞系中的EMT过程。

期刊:Biomedicines

影响因子:3.9

doi:10.3390/biomedicines13122950

Simanovich, Elina; Oyelami, Felix; Brockmeyer, Phillipp; Rahat, Michal A

Editorial: Angiogenesis and tumor metastasis

社论：血管生成与肿瘤转移

期刊:Frontiers in Oncology

影响因子:3.3

doi:10.3389/fonc.2022.1129736

Zhang, Qiang-Zhe; Zhu, Yi-Pan; Rahat, Michal A; Kzhyshkowska, Julia

Editorial: Targeting Angiogenesis to Treat Autoimmune Diseases and Cancer

社论：靶向血管生成治疗自身免疫性疾病和癌症

期刊:Frontiers in Immunology

影响因子:5.9

doi:10.3389/fimmu.2020.01005

Rahat, Michal A; Kzhyshkowska, Julia; Iragavarapu-Charyulu, Vijaya

Angiogenesis

发表日期：2020

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Effects of Tocilizumab, an Anti-Interleukin-6 Receptor Antibody, on Serum Lipid and Adipokine Levels in Patients with Rheumatoid Arthritis.

托珠单抗（一种抗白细胞介素-6受体抗体）对类风湿性关节炎患者血清脂质和脂肪因子水平的影响

期刊:International Journal of Molecular Sciences

影响因子:4.9

doi:10.3390/ijms20184633

Hoffman Elinoar, Rahat Michal A, Feld Joy, Elias Muna, Rosner Itzhak, Kaly Lisa, Lavie Idit, Gazitt Tal, Zisman Devy

Function of miR-146a-5p in Tumor Cells As a Regulatory Switch between Cell Death and Angiogenesis: Macrophage Therapy Revisited

miR-146a-5p在肿瘤细胞中的功能：作为细胞死亡和血管生成之间的调控开关：巨噬细胞疗法再探

期刊:Frontiers in Immunology

影响因子:5.7

doi:10.3389/fimmu.2017.01931

Elina Simanovich ，Vera Brod ，Maya M Rahat ，Michal A Rahat

Parallel Aspects of the Microenvironment in Cancer and Autoimmune Disease

癌症和自身免疫性疾病微环境的相似之处

期刊:Mediators of Inflammation

影响因子:4.2

doi:10.1155/2016/4375120

Rahat, Michal A; Shakya, Jivan

Macrophages and Neutrophils: Regulation of the Inflammatory Microenvironment in Autoimmunity and Cancer

巨噬细胞和中性粒细胞：自身免疫和癌症中炎症微环境的调控

期刊:Mediators of Inflammation

影响因子:4.2

doi:10.1155/2016/5894347

Rahat, Michal A; Coffelt, Seth B; Granot, Zvi; Muthana, Munitta; Amedei, Amedeo

Tumor cell-macrophage interactions increase angiogenesis through secretion of EMMPRIN

肿瘤细胞-巨噬细胞相互作用通过分泌 EMMPRIN 增加血管生成

期刊:Frontiers in Physiology

影响因子:3.2

doi:10.3389/fphys.2013.00178

Bat-Chen Amit-Cohen, Maya M Rahat, Michal A Rahat

Molecular mechanisms regulating macrophage response to hypoxia

调控巨噬细胞对缺氧反应的分子机制

期刊:Frontiers in Immunology

影响因子:5.9

doi:10.3389/fimmu.2011.00045

Rahat, Michal A; Bitterman, Haim; Lahat, Nitza

Serum EMMPRIN/CD147 promotes the lung pre-metastatic niche in a D2A1 mammary carcinoma mouse model

血清 EMMPRIN/CD147 在 D2A1 乳腺癌小鼠模型中促进肺转移前微环境的形成

The Anti-EMMPRIN Monoclonal Antibody hMR18-mAb Induces Tumor Dormancy and Inhibits the EMT Process in Human Carcinoma Cell Lines Co-Cultured with Macrophages

抗EMMPRIN单克隆抗体hMR18-mAb可诱导肿瘤休眠并抑制与巨噬细胞共培养的人类癌细胞系中的EMT过程。

Editorial: Angiogenesis and tumor metastasis

社论：血管生成与肿瘤转移

Editorial: Targeting Angiogenesis to Treat Autoimmune Diseases and Cancer

社论：靶向血管生成治疗自身免疫性疾病和癌症

Effects of Tocilizumab, an Anti-Interleukin-6 Receptor Antibody, on Serum Lipid and Adipokine Levels in Patients with Rheumatoid Arthritis.

托珠单抗（一种抗白细胞介素-6受体抗体）对类风湿性关节炎患者血清脂质和脂肪因子水平的影响

Function of miR-146a-5p in Tumor Cells As a Regulatory Switch between Cell Death and Angiogenesis: Macrophage Therapy Revisited

miR-146a-5p在肿瘤细胞中的功能：作为细胞死亡和血管生成之间的调控开关：巨噬细胞疗法再探

Parallel Aspects of the Microenvironment in Cancer and Autoimmune Disease

癌症和自身免疫性疾病微环境的相似之处

Macrophages and Neutrophils: Regulation of the Inflammatory Microenvironment in Autoimmunity and Cancer

巨噬细胞和中性粒细胞：自身免疫和癌症中炎症微环境的调控

Tumor cell-macrophage interactions increase angiogenesis through secretion of EMMPRIN

肿瘤细胞-巨噬细胞相互作用通过分泌 EMMPRIN 增加血管生成

Molecular mechanisms regulating macrophage response to hypoxia

调控巨噬细胞对缺氧反应的分子机制