日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Decreased pyruvate dehydrogenase activity in Tafazzin-deficient cells is caused by dysregulation of pyruvate dehydrogenase phosphatase 1 (PDP1)

Tafazzin 缺乏的细胞中丙酮酸脱氢酶活性降低是由于丙酮酸脱氢酶磷酸酶 1 (PDP1) 失调引起的

Zhuqing Liang, Tyler Ralph-Epps, Michael W Schmidtke, Vikalp Kumar, Miriam L Greenberg

Author Correction: Upregulation of the AMPK-FOXO1-PDK4 pathway is a primary mechanism of pyruvate dehydrogenase activity reduction in tafazzin-deficient cells

作者更正:AMPK-FOXO1-PDK4通路的激活是tafazzin缺陷细胞中丙酮酸脱氢酶活性降低的主要机制。

Liang, Zhuqing; Ralph-Epps, Tyler; Schmidtke, Michael W; Lazcano, Pablo; Denis, Simone W; Balážová, Mária; Teixeira da Rosa, Nevton Jr; Chakkour, Mohamed; Hazime, Sanaa; Ren, Mindong; Schlame, Michael; Houtkooper, Riekelt H; Greenberg, Miriam L

Upregulation of the AMPK-FOXO1-PDK4 pathway is a primary mechanism of pyruvate dehydrogenase activity reduction in tafazzin-deficient cells

AMPK-FOXO1-PDK4 通路的上调是 tafazzin 缺乏细胞中丙酮酸脱氢酶活性降低的主要机制

Zhuqing Liang, Tyler Ralph-Epps, Michael W Schmidtke, Pablo Lazcano, Simone W Denis, Mária Balážová, Nevton Teixeira da Rosa Jr, Mohamed Chakkour, Sanaa Hazime, Mindong Ren, Michael Schlame, Riekelt H Houtkooper, Miriam L Greenberg

Upregulation of the AMPK-FOXO1-PDK4 pathway is a primary mechanism of pyruvate dehydrogenase activity reduction and leads to increased glucose uptake in tafazzin-deficient cells

AMPK-FOXO1-PDK4 通路的上调是丙酮酸脱氢酶活性降低的主要机制,并导致缺乏 tafazzin 的细胞对葡萄糖的吸收增加

Zhuqing Liang, Tyler Ralph-Epps, Michael W Schmidtke, Pablo Lazcano, Simone W Denis, Mária Balážová, Mohamed Chakkour, Sanaa Hazime, Mindong Ren, Michael Schlame, Riekelt H Houtkooper, Miriam L Greenberg

Studying Lipid-Related Pathophysiology Using the Yeast Model

利用酵母模型研究脂质相关病理生理学

Ralph-Epps, Tyler; Onu, Chisom J; Vo, Linh; Schmidtke, Michael W; Le, Anh; Greenberg, Miriam L