Ratelade相关文献与解析，生知库 | 链接生命科学的每一步

Reducing Hypermuscularization of the Transitional Segment Between Arterioles and Capillaries Protects Against Spontaneous Intracerebral Hemorrhage

减少小动脉和毛细血管之间过渡段的肌肉过度增生可预防自发性脑出血

doi:10.1161/CIRCULATIONAHA.119.040963

Ratelade, Julien; Klug, Nicholas R; Lombardi, Damiano; Angelim, Monara Kaelle Servulo Cruz; Dabertrand, Fabrice; Domenga-Denier, Valérie; Al-Shahi Salman, Rustam; Smith, Colin; Gerbeau, Jean-Frédéric; Nelson, Mark T; Joutel, Anne

神经科学

发表日期：2020

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Blood brain barrier leakage is not a consistent feature of white matter lesions in CADASIL

血脑屏障渗漏并不是 CADASIL 白质病变的一致特征

期刊:Acta Neuropathologica Communications

影响因子:6.2

doi:10.1186/s40478-019-0844-x

Rikesh M Rajani, Julien Ratelade, Valérie Domenga-Denier, Yoshiki Hase, Hannu Kalimo, Raj N Kalaria, Anne Joutel

Perturbations of the cerebrovascular matrisome: A convergent mechanism in small vessel disease of the brain?

脑血管基质组的扰动：脑小血管疾病的趋同机制？

期刊:Journal of Cerebral Blood Flow and Metabolism

影响因子:4.5

doi:10.1038/jcbfm.2015.62

Joutel, Anne; Haddad, Iman; Ratelade, Julien; Nelson, Mark T

神经科学

发表日期：2016

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Unique neuromyelitis optica pathology produced in naïve rats by intracerebral administration of NMO-IgG

通过脑内注射NMO-IgG，在未感染的大鼠中诱发了独特的视神经脊髓炎病理。

期刊:Acta Neuropathologica

影响因子:9.3

doi:10.1007/s00401-013-1204-8

Asavapanumas, Nithi; Ratelade, Julien; Verkman, A S

Aggregation state determines the localization and function of M1- and M23-aquaporin-4 in astrocytes

聚集状态决定了星形胶质细胞中M1-和M23-水通道蛋白4的定位和功能。

期刊:Journal of Cell Biology

影响因子:6.4

doi:10.1083/jcb.201308118

Smith, Alex J; Jin, Byung-Ju; Ratelade, Julien; Verkman, Alan S

Greatly improved survival and neuroprotection in aquaporin-4-knockout mice following global cerebral ischemia

全脑缺血后，水通道蛋白4敲除小鼠的存活率和神经保护作用显著提高

期刊:FASEB Journal

影响因子:4.2

doi:10.1096/fj.13-231274

Katada, Ryuichi; Akdemir, Gokhan; Asavapanumas, Nithi; Ratelade, Julien; Zhang, Hua; Verkman, A S

神经科学

发表日期：2014

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Human immunoglobulin G reduces the pathogenicity of aquaporin-4 autoantibodies in neuromyelitis optica

人免疫球蛋白G可降低视神经脊髓炎中水通道蛋白4自身抗体的致病性

期刊:Experimental Neurology

影响因子:4.2

doi:10.1016/j.expneurol.2014.03.004

Ratelade, Julien; Smith, Alex J; Verkman, A S

Transient hyperckemia in the setting of neuromyelitis optica (NMO)

视神经脊髓炎（NMO）背景下的短暂性高肌酸激酶血症

期刊:Muscle & Nerve

影响因子:3.1

doi:10.1002/mus.24298

Malik, Rabia; Lewis, Aaron; Cree, Bruce A C; Ratelade, Julien; Rossi, Andrea; Verkman, Alan S; Bollen, Andrew W; Ralph, Jeffrey W

Inhibitor(s) of the classical complement pathway in mouse serum limit the utility of mice as experimental models of neuromyelitis optica

小鼠血清中经典补体途径抑制剂限制了小鼠作为视神经脊髓炎实验模型的应用。

期刊:Molecular Immunology

影响因子:3

doi:10.1016/j.molimm.2014.06.003

Ratelade, Julien; Verkman, A S

Genomic Size Is Critical to Guarantee the Genomic Stability of Non-Replicative HSV1 Vectors

基因组大小对于保证非复制型HSV1载体的基因组稳定性至关重要

Reducing Hypermuscularization of the Transitional Segment Between Arterioles and Capillaries Protects Against Spontaneous Intracerebral Hemorrhage

减少小动脉和毛细血管之间过渡段的肌肉过度增生可预防自发性脑出血

Blood brain barrier leakage is not a consistent feature of white matter lesions in CADASIL

血脑屏障渗漏并不是 CADASIL 白质病变的一致特征

Perturbations of the cerebrovascular matrisome: A convergent mechanism in small vessel disease of the brain?

脑血管基质组的扰动：脑小血管疾病的趋同机制？

Unique neuromyelitis optica pathology produced in naïve rats by intracerebral administration of NMO-IgG

通过脑内注射NMO-IgG，在未感染的大鼠中诱发了独特的视神经脊髓炎病理。

Aggregation state determines the localization and function of M1- and M23-aquaporin-4 in astrocytes

聚集状态决定了星形胶质细胞中M1-和M23-水通道蛋白4的定位和功能。

Greatly improved survival and neuroprotection in aquaporin-4-knockout mice following global cerebral ischemia

全脑缺血后，水通道蛋白4敲除小鼠的存活率和神经保护作用显著提高

Human immunoglobulin G reduces the pathogenicity of aquaporin-4 autoantibodies in neuromyelitis optica

人免疫球蛋白G可降低视神经脊髓炎中水通道蛋白4自身抗体的致病性

Transient hyperckemia in the setting of neuromyelitis optica (NMO)

视神经脊髓炎（NMO）背景下的短暂性高肌酸激酶血症

Inhibitor(s) of the classical complement pathway in mouse serum limit the utility of mice as experimental models of neuromyelitis optica

小鼠血清中经典补体途径抑制剂限制了小鼠作为视神经脊髓炎实验模型的应用。