日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Conditional BCL-2 Expression in Fibroblasts Promotes Persistent Pulmonary Fibrosis which is Reversible by Therapeutic BCL-2 Inhibition.

成纤维细胞中条件性 BCL-2 表达促进持续性肺纤维化,而治疗性 BCL-2 抑制可逆转这一过程。

Redente Elizabeth F, Song Tengyao, Javkhlan Nomin, Edelman Benjamin L, Foster Daniel G, Wilson Jasmine A, Chakraborty Sangeeta, Cooley Joseph C, Gaurav Rohit, Saguthi Satria, Seibold Max A, Blumhagen Rachel Z, Schwartz David A, Yang Ivana V, Matsuda Jennifer, Bridges James P, Zemans Rachel L, Tuder Rubin M, Riches David W H

Addressing Sex as a Biological Variable in Preclinical Models of Lung Disease: An Official American Thoracic Society Research Statement

在肺部疾病临床前模型中将性别作为生物学变量进行研究:美国胸科协会官方研究声明

Lingappan, Krithika; Sundar, Isaac Kirubakaran; Prakash, Y S; Redente, Elizabeth F; Sin, Don D; Han, MeiLan K; Lahm, Tim; Krymskaya, Vera P; Kotton, Darrell N; Kulkarni, Hrishikesh S; Jain, Raksha; Königshoff, Melanie; Eghbali, Mansoureh; Anguera, Montserrat; Arnold, Arthur; Silveyra, Patricia; DeMeo, Dawn L

Progressive lung fibrosis: reprogramming a genetically vulnerable bronchoalveolar epithelium

进行性肺纤维化:重编程遗传易感支气管肺泡上皮

Bridges, James P; Vladar, Eszter K; Kurche, Jonathan S; Krivoi, Andrei; Stancil, Ian T; Dobrinskikh, Evgenia; Hu, Yan; Sasse, Sarah K; Lee, Joyce S; Blumhagen, Rachel Z; Yang, Ivana V; Gerber, Anthony N; Peljto, Anna L; Evans, Christopher M; Redente, Elizabeth F; Riches, David Wh; Schwartz, David A

Pathogenic expansion: fibroblast proliferation fuels fibrosis

致病性扩张:成纤维细胞增殖促进纤维化

Schott, Cody A; Redente, Elizabeth F

Pulmonary Fibrosis-Focusing on the Future: Aspen Lung Conference 2024 Summary

肺纤维化——着眼未来:2024年阿斯彭肺病大会总结

Freeberg, Margaret A T; Redente, Elizabeth F; Perelas, Apostolos; Riches, David W H; Schwartz, David A; Sime, Patricia J

Reimagining Fibrosis Research, Outcomes, and Therapeutics Through the Lens of Resolution

从分辨率视角重新构想纤维化研究、结果和治疗方法

Fortier, Sean M; Redente, Elizabeth F; Peters-Golden, Marc

Distinct lung functional, histological and cell senescence signatures in the single and repetitive bleomycin mouse models of idiopathic pulmonary fibrosis.

特发性肺纤维化小鼠模型中单次和重复博来霉素治疗后,肺功能、组织学和细胞衰老特征均有显著差异。

Bousamaki Jamal, Rørbeck Emma A, Petersen Asbjørn Graver, Korntner Stefanie H, Pors Susanne E, Redente Elizabeth F, Oró Denise, Salinas Casper Gravesen, Olsen Rebecca Wendelboe, Simonsen Ulf, Feigh Michael, Hansen Henrik H

Alveolar macrophages: guardians of the alveolar lipid galaxy

肺泡巨噬细胞:肺泡脂质星系的守护者

Little, Isaiah; Bersie, Stephanie; Redente, Elizabeth F; McCubbrey, Alexandra L; Tarling, Elizabeth J

Biomarkers Unveil Insights into Pathology of Transitional Epithelial States in Pulmonary Fibrosis

生物标志物揭示肺纤维化中过渡上皮状态的病理学见解

Ting, Christopher; Konopka, Kristine; Benedeck, Rachel E; Riches, David W H; Redente, Elizabeth F; Oldham, Justin M; Zemans, Rachel L

Gpnmb and Spp1 mark a conserved macrophage injury response masking fibrosis-specific programming in the lung

Gpnmb 和 Spp1 标志着一种保守的巨噬细胞损伤反应,这种反应掩盖了肺部纤维化特异性程序。

King, Emily M; Zhao, Yifan; Moore, Camille M; Steinhart, Benjamin; Anderson, Kelsey C; Vestal, Brian; Moore, Peter K; McManus, Shannon A; Evans, Christopher M; Mould, Kara J; Redente, Elizabeth F; McCubbrey, Alexandra L; Janssen, William J