Reed-Geaghan相关文献与解析，生知库 | 链接生命科学的每一步

Microglia depletion rapidly and reversibly alters amyloid pathology by modification of plaque compaction and morphologies

小胶质细胞耗竭可通过改变斑块致密性和形态，迅速且可逆地改变淀粉样蛋白病理。

期刊:Neurobiology of Disease

影响因子:5.1

doi:10.1016/j.nbd.2020.104956

Brad T Casali ，Kathryn P MacPherson ，Erin G Reed-Geaghan ，Gary E Landreth

The Trem2 R47H variant confers loss-of-function-like phenotypes in Alzheimer's disease

Trem2 R47H 变异体在阿尔茨海默病中表现出类似功能丧失的表型

期刊:Molecular Neurodegeneration

影响因子:14.9

doi:10.1186/s13024-018-0262-8

Paul J Cheng-Hathaway ，Erin G Reed-Geaghan ，Taylor R Jay ，Brad T Casali ，Shane M Bemiller ，Shweta S Puntambekar ，Victoria E von Saucken ，Roxanne Y Williams ，J Colleen Karlo ，Miguel Moutinho ，Guixiang Xu ，Richard M Ransohoff ，Bruce T Lamb ，Gary E Landreth

阿尔茨海默症

发表日期：2018

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Nuclear receptor agonist-driven modification of inflammation and amyloid pathology enhances and sustains cognitive improvements in a mouse model of Alzheimer's disease

核受体激动剂驱动的炎症和淀粉样蛋白病理改变可增强并维持阿尔茨海默病小鼠模型的认知改善

期刊:Journal of Neuroinflammation

影响因子:9.3

doi:10.1186/s12974-018-1091-y

Brad T Casali ，Erin G Reed-Geaghan ，Gary E Landreth

Merkel Cell-Driven BDNF Signaling Specifies SAI Neuron Molecular and Electrophysiological Phenotypes

默克尔细胞驱动的 BDNF 信号指定 SAI 神经元分子和电生理表型

期刊:Journal of Neuroscience

影响因子:4.4

doi:10.1523/JNEUROSCI.3781-15.2016

Erin G Reed-Geaghan, Margaret C Wright, Lauren A See, Peter C Adelman, Kuan Hsien Lee, H Richard Koerber, Stephen M Maricich

Unipotent, Atoh1+ progenitors maintain the Merkel cell population in embryonic and adult mice

单能性 Atoh1+ 祖细胞维持胚胎和成年小鼠中的默克尔细胞群

期刊:Journal of Cell Biology

影响因子:7.4

doi:10.1083/jcb.201407101

Margaret C Wright, Erin G Reed-Geaghan, Alexa M Bolock, Tomoyuki Fujiyama, Mikio Hoshino, Stephen M Maricich

Ibuprofen attenuates oxidative damage through NOX2 inhibition in Alzheimer's disease

布洛芬通过抑制NOX2来减轻阿尔茨海默病中的氧化损伤

期刊:Neurobiology of Aging

影响因子:3.5

doi:10.1016/j.neurobiolaging.2010.06.014

Wilkinson, Brandy L; Cramer, Paige E; Varvel, Nicholas H; Reed-Geaghan, Erin; Jiang, Qingguang; Szabo, Alison; Herrup, Karl; Lamb, Bruce T; Landreth, Gary E

Peripheral somatosensation: a touch of genetics

周围躯体感觉：遗传学的一点作用

期刊:Current Opinion in Genetics & Development

影响因子:3.6

doi:10.1016/j.gde.2010.12.009

Reed-Geaghan, Erin G; Maricich, Stephen M

发表日期：2011

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Attenuation of neuroinflammation and Alzheimer's disease pathology by liver x receptors

肝脏 x 受体减轻神经炎症和阿尔茨海默病病理

期刊:Proceedings of the National Academy of Sciences of the United States of America

影响因子:

doi:10.1073/pnas.0701096104

Noam Zelcer, Negar Khanlou, Ryan Clare, Qingguang Jiang, Erin G Reed-Geaghan, Gary E Landreth, Harry V Vinters, Peter Tontonoz

神经

神经炎症

Immunology/Inflammation

iNOS

发表日期：2007

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Plaque-associated myeloid cells derive from resident microglia in an Alzheimer's disease model

在阿尔茨海默病模型中，斑块相关的髓系细胞来源于常驻小胶质细胞。

Microglia depletion rapidly and reversibly alters amyloid pathology by modification of plaque compaction and morphologies

小胶质细胞耗竭可通过改变斑块致密性和形态，迅速且可逆地改变淀粉样蛋白病理。

The Trem2 R47H variant confers loss-of-function-like phenotypes in Alzheimer's disease

Trem2 R47H 变异体在阿尔茨海默病中表现出类似功能丧失的表型

Nuclear receptor agonist-driven modification of inflammation and amyloid pathology enhances and sustains cognitive improvements in a mouse model of Alzheimer's disease

核受体激动剂驱动的炎症和淀粉样蛋白病理改变可增强并维持阿尔茨海默病小鼠模型的认知改善

Merkel Cell-Driven BDNF Signaling Specifies SAI Neuron Molecular and Electrophysiological Phenotypes

默克尔细胞驱动的 BDNF 信号指定 SAI 神经元分子和电生理表型

Unipotent, Atoh1+ progenitors maintain the Merkel cell population in embryonic and adult mice

单能性 Atoh1+ 祖细胞维持胚胎和成年小鼠中的默克尔细胞群

Ibuprofen attenuates oxidative damage through NOX2 inhibition in Alzheimer's disease

布洛芬通过抑制NOX2来减轻阿尔茨海默病中的氧化损伤

Peripheral somatosensation: a touch of genetics

周围躯体感觉：遗传学的一点作用

Attenuation of neuroinflammation and Alzheimer's disease pathology by liver x receptors

肝脏 x 受体减轻神经炎症和阿尔茨海默病病理