日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Mechanisms of whole-genome doubling in cancer evolution

癌症进化中全基因组加倍的机制

McKenney, Connor D; Regot, Sergi

Cell cycle regulation by the ribotoxic stress response

核糖体毒性应激反应对细胞周期的调控

McKenney, Connor D; Regot, Sergi

CDK2 activity crosstalk on the ERK kinase translocation reporter can be resolved computationally

CDK2活性对ERK激酶易位报告基因的串扰可以通过计算方法解析。

Hoffman, Timothy E; Tian, Chengzhe; Nangia, Varuna; Yang, Chen; Regot, Sergi; Gerosa, Luca; Spencer, Sabrina L

CDK4/6 activity is required during G(2) arrest to prevent stress-induced endoreplication.

CDK4/6 活性在 G(2) 阻滞期间是必需的,以防止应激诱导的内复制

McKenney Connor, Lendner Yovel, Guerrero Zuniga Adler, Sinha Niladri, Veresko Benjamin, Aikin Timothy J, Regot Sergi

The ribotoxic stress response drives UV-mediated cell death.

核糖体毒性应激反应驱动紫外线介导的细胞死亡

Sinha Niladri K, McKenney Connor, Yeow Zhong Y, Li Jeffrey J, Nam Ki Hong, Yaron-Barir Tomer M, Johnson Jared L, Huntsman Emily M, Cantley Lewis C, Ordureau Alban, Regot Sergi, Green Rachel

Oncogenic Kras induces spatiotemporally specific tissue deformation through converting pulsatile into sustained ERK activation.

致癌 Kras 通过将脉冲式 ERK 激活转化为持续性 ERK 激活,诱导时空特异性的组织变形

Xin Tianchi, Gallini Sara, Wei Haoyang, Gonzalez David G, Matte-Martone Catherine, Machida Hiroki, Fujiwara Hironobu, Pasolli H Amalia, Suozzi Kathleen C, Gonzalez Lauren E, Regot Sergi, Greco Valentina

Editorial: Understanding Immunobiology Through the Specificity of NF-κB

社论:通过NF-κB的特异性理解免疫生物学

Sung, Myong-Hee; Regot, Sergi

A Real-Time Biosensor for ERK Activity Reveals Signaling Dynamics during C. elegans Cell Fate Specification

一种用于检测ERK活性的实时生物传感器揭示了秀丽隐杆线虫细胞命运决定过程中的信号传导动态。

de la Cova, Claire; Townley, Robert; Regot, Sergi; Greenwald, Iva

A New Way to Target Degradation of the Epidermal Growth Factor Receptor in Cancer Cells♦: Destabilization of the Epidermal Growth Factor Receptor (EGFR) by a Peptide That Inhibits EGFR Binding to Heat Shock Protein 90 and Receptor Dimerization

一种靶向降解癌细胞中表皮生长因子受体的新方法♦:通过抑制表皮生长因子受体 (EGFR) 与热休克蛋白 90 结合及受体二聚化的肽段,实现 EGFR 的去稳定化。

Ppar Research; Regot, Sergi; de Nadal, Eulàlia; Rodríguez-Navarro, Susana; González-Novo, Alberto; Pérez-Fernandez, Jorge; Gadal, Olivier; Seisenbacher, Gerhard; Ammerer, Gustav; Posas, Francesc; West, Matthew B; Chen, Yunyu; Wickham, Stephanie; Heroux, Ann; Cahill, Kyle; Hanigan, Marie H; Mooers, Blaine H M; Tee, Meng Kian; Miller, Walter L; Charvet, Casey D; Saadane, Aicha; Wang, Meiyao; Salomon, Robert G; Brunengraber, Henri; Turko, Illarion V; Pikuleva, Irina A; Qian, Cheng; Qian, Li; Yu, Yizhi; An, Huazhang; Guo, Zhenhong; Han, Yanmei; Chen, Yongjian; Bai, Yi; Wang, Qingqing; Cao, Xuetao

The HOG pathway dictates the short-term translational response after hyperosmotic shock

HOG通路决定高渗休克后的短期转化反应。

Warringer, Jonas; Hult, Malin; Regot, Sergi; Posas, Francesc; Sunnerhagen, Per