Relaix相关文献与解析，生知库 | 链接生命科学的每一步

Non-canonical nuclear function of glutaminase cooperates with Wnt signaling to drive EMT during neural crest development

谷氨酰胺酶的非经典核功能与Wnt信号通路协同作用，驱动神经嵴发育过程中的上皮间质转化（EMT）。

Nioosha Nekooie Marnany ,Alwyn Dady ,Isabelle Duband-Goulet ,Frédéric Relaix ,Roberto Motterlini ,Roberta Foresti ,Sylvie Dufour ,Jean-Loup Duband

发育与干细胞

Dual transcriptional activities of PAX3 and PAX7 spatially encode spinal cell fates through distinct gene networks

PAX3 和 PAX7 的双重转录活性通过不同的基因网络在空间上编码脊髓细胞的命运。

期刊:PLoS Biology

影响因子:7.8

doi:10.1371/journal.pbio.3003448.

Robin Rondon,Théaud Hezez,Julien Richard Albert,Shinichiro Hayashi,Bernadette Drayton-Libotte,Gloria Gonzalez Curto,Frédéric Auradé,Elie Balloul,Claire Dugast-Darzacq,Frédéric Relaix,Pascale Gilardi-Hebenstreit,Vanessa Ribes

Forskolin treatment enhances muscle regeneration and shows therapeutic potential with limitations in Duchenne muscular dystrophy

毛喉素治疗可促进肌肉再生，并显示出治疗潜力，但对杜氏肌营养不良症的疗效有限。

期刊:Skeletal Muscle

影响因子:5.3

doi:10.1186/s13395-025-00381-7

Andreea Iuliana Cojocaru # ，Kaouthar Kefi # ，Jean-Daniel Masson # ，Laurent Tiret ，Frederic Relaix ，Valentina Taglietti

发表日期：2025

文献求助收藏

Pharmacologic Inhibition of Glutamate Dehydrogenase 1 Improves Functional Recovery of Neuromuscular Junctions and Muscle Function in Duchenne Muscular Dystrophy

谷氨酸脱氢酶1的药理学抑制可改善杜氏肌营养不良症患者的神经肌肉接头功能恢复和肌肉功能

期刊:American Journal of Pathology

影响因子:4.7

doi:10.1016/j.ajpath.2025.05.003

Andreia Pereira-Nunes ，Ummi Ammarah ，Min Shang ，Iris Charatsidou ，Himal Sharma ，Bruna Pereira Sorroche ，Max Nobis ，Thibaut Burg ，Stijn Verschoren ，Frédéric Relaix ，Alessio Rotini ，Ludo Van Den Bosch ，Marcello Delfini ，Emanuele Berardi ，Massimiliano Mazzone

The rise of rat models for Duchenne muscular dystrophy and therapeutic evaluations

杜氏肌营养不良症大鼠模型及其治疗评估的兴起

期刊:Skeletal Muscle

影响因子:4.4

doi:10.1186/s13395-025-00395-1

Relaix, Frederic; Lafuste, Peggy; Taglietti, Valentina; Tiret, Laurent

Rat

发表日期：2025

文献求助收藏

Extensive striated muscle damage in a rat model of Duchenne muscular dystrophy with Dmd exons 10-17 duplication.

在患有杜氏肌营养不良症的大鼠模型中，Dmd 外显子 10-17 重复导致广泛的横纹肌损伤

期刊:Skeletal Muscle

影响因子:4.4

doi:10.1186/s13395-025-00386-2

Masson Jean-Daniel, Taglietti Valentina, Ruby FranÃ§ois, Ono Hiroya, Mouri Nadir, Jorge Alan, Guillaud Laurent, Tiret Laurent, Relaix Frederic

DAXX-dependent H3.3 deposition maintains myoblast cell identity independently of other histone chaperone complexes.

DAXX 依赖的 H3.3 沉积维持成肌细胞的特性，而与其他组蛋白伴侣复合物无关

期刊:iScience

影响因子:4.1

doi:10.1016/j.isci.2025.113119

细胞生物学

发表日期：2025

文献求助收藏

Transposon expression and repression in skeletal muscle

骨骼肌中转座子的表达和抑制

期刊:Mobile DNA

影响因子:3.1

doi:10.1186/s13100-025-00352-1

Borok, Matthew J; Zaidan, Louai; Relaix, Frederic

发表日期：2025

文献求助收藏

Correction: The rise of rat models for Duchenne muscular dystrophy and therapeutic evaluations

更正：杜氏肌营养不良症大鼠模型及其治疗评估的兴起

The contribution of skeletal muscle interstitial cells to myogenesis

骨骼肌间质细胞对肌生成的贡献

Non-canonical nuclear function of glutaminase cooperates with Wnt signaling to drive EMT during neural crest development

谷氨酰胺酶的非经典核功能与Wnt信号通路协同作用，驱动神经嵴发育过程中的上皮间质转化（EMT）。

Dual transcriptional activities of PAX3 and PAX7 spatially encode spinal cell fates through distinct gene networks

PAX3 和 PAX7 的双重转录活性通过不同的基因网络在空间上编码脊髓细胞的命运。

Forskolin treatment enhances muscle regeneration and shows therapeutic potential with limitations in Duchenne muscular dystrophy

毛喉素治疗可促进肌肉再生，并显示出治疗潜力，但对杜氏肌营养不良症的疗效有限。

Pharmacologic Inhibition of Glutamate Dehydrogenase 1 Improves Functional Recovery of Neuromuscular Junctions and Muscle Function in Duchenne Muscular Dystrophy

谷氨酸脱氢酶1的药理学抑制可改善杜氏肌营养不良症患者的神经肌肉接头功能恢复和肌肉功能

The rise of rat models for Duchenne muscular dystrophy and therapeutic evaluations

杜氏肌营养不良症大鼠模型及其治疗评估的兴起

Extensive striated muscle damage in a rat model of Duchenne muscular dystrophy with Dmd exons 10-17 duplication.

在患有杜氏肌营养不良症的大鼠模型中，Dmd 外显子 10-17 重复导致广泛的横纹肌损伤

DAXX-dependent H3.3 deposition maintains myoblast cell identity independently of other histone chaperone complexes.

DAXX 依赖的 H3.3 沉积维持成肌细胞的特性，而与其他组蛋白伴侣复合物无关

Transposon expression and repression in skeletal muscle

骨骼肌中转座子的表达和抑制