Rellmann相关文献与解析，生知库 | 链接生命科学的每一步

Extracellular ERp57 promotes fibronectin fibril formation during matrix assembly of articular cartilage

细胞外ERp57促进关节软骨基质组装过程中纤连蛋白原纤维的形成

期刊:iScience

影响因子:4.1

doi:10.1016/j.isci.2025.114046

Rellmann, Yvonne; Eidhof, Elco; Hansen, Uwe; Schulte, Sandra; Stücker, Sina; Pap, Thomas; Dreier, Rita

细胞生物学

发表日期：2025

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Estradiol Inhibits ER Stress-Induced Apoptosis in Chondrocytes and Contributes to a Reduced Osteoarthritic Cartilage Degeneration in Female Mice

雌二醇抑制内质网应激诱导的软骨细胞凋亡，并有助于减少雌性小鼠骨关节炎软骨的退化

期刊:Frontiers in Cell and Developmental Biology

影响因子:4.3

doi:10.3389/fcell.2022.913118

Dreier, Rita; Ising, Thomas; Ramroth, Markus; Rellmann, Yvonne

Loss of surface transport is a main cellular pathomechanism of CRB2 variants causing podocytopathies

表面运输丧失是 CRB2 变异导致足细胞病的主要细胞病理机制

期刊:Life Science Alliance

影响因子:3.3

doi:10.26508/lsa.202201649

Annika Möller-Kerutt, Birgit Schönhoff, Yvonne Rellmann, Britta George, Daniela Anne Braun, Hermann Pavenstädt, Thomas Weide

ER Stress in ERp57 Knockout Knee Joint Chondrocytes Induces Osteoarthritic Cartilage Degradation and Osteophyte Formation

ERp57 敲除膝关节软骨细胞中的 ER 应激诱导骨关节炎软骨退化和骨赘形成

期刊:International Journal of Molecular Sciences

影响因子:0.7

doi:10.3390/ijms23010182

Yvonne Rellmann, Elco Eidhof, Uwe Hansen, Lutz Fleischhauer, Jonas Vogel, Hauke Clausen-Schaumann, Attila Aszodi, Rita Dreier

4-Phenylbutyric Acid Reduces Endoplasmic Reticulum Stress in Chondrocytes That Is Caused by Loss of the Protein Disulfide Isomerase ERp57

4-苯基丁酸可降低软骨细胞中由蛋白质二硫键异构酶ERp57缺失引起的内质网应激

期刊:Oxidative Medicine and Cellular Longevity

影响因子:

doi:10.1155/2019/6404035

Rellmann, Yvonne; Gronau, Isabel; Hansen, Uwe; Dreier, Rita

Different Forms of ER Stress in Chondrocytes Result in Short Stature Disorders and Degenerative Cartilage Diseases: New Insights by Cartilage-Specific ERp57 Knockout Mice

软骨细胞中不同形式的内质网应激导致身材矮小症和软骨退行性疾病：软骨特异性ERp57敲除小鼠的新见解

期刊:Oxidative Medicine and Cellular Longevity

影响因子:

doi:10.1155/2018/8421394

Rellmann, Yvonne; Dreier, Rita

ER-resident proteins are key players in cartilage and bone homeostasis

内质网驻留蛋白是软骨和骨骼稳态的关键参与者。

Extracellular ERp57 promotes fibronectin fibril formation during matrix assembly of articular cartilage

细胞外ERp57促进关节软骨基质组装过程中纤连蛋白原纤维的形成

Estradiol Inhibits ER Stress-Induced Apoptosis in Chondrocytes and Contributes to a Reduced Osteoarthritic Cartilage Degeneration in Female Mice

雌二醇抑制内质网应激诱导的软骨细胞凋亡，并有助于减少雌性小鼠骨关节炎软骨的退化

Loss of surface transport is a main cellular pathomechanism of CRB2 variants causing podocytopathies

表面运输丧失是 CRB2 变异导致足细胞病的主要细胞病理机制

ER Stress in ERp57 Knockout Knee Joint Chondrocytes Induces Osteoarthritic Cartilage Degradation and Osteophyte Formation

ERp57 敲除膝关节软骨细胞中的 ER 应激诱导骨关节炎软骨退化和骨赘形成

4-Phenylbutyric Acid Reduces Endoplasmic Reticulum Stress in Chondrocytes That Is Caused by Loss of the Protein Disulfide Isomerase ERp57

4-苯基丁酸可降低软骨细胞中由蛋白质二硫键异构酶ERp57缺失引起的内质网应激

Different Forms of ER Stress in Chondrocytes Result in Short Stature Disorders and Degenerative Cartilage Diseases: New Insights by Cartilage-Specific ERp57 Knockout Mice

软骨细胞中不同形式的内质网应激导致身材矮小症和软骨退行性疾病：软骨特异性ERp57敲除小鼠的新见解