Ren Haigang相关文献与解析，生知库 | 链接生命科学的每一步

Loss of NgBR causes neuronal damage through decreasing KAT7-mediated RFX1 acetylation and FGF1 expression.

NgBR 的缺失会导致神经元损伤，其机制是通过降低 KAT7 介导的 RFX1 乙酰化和 FGF1 表达

期刊:Cellular and Molecular Life Sciences

影响因子:6.2

doi:10.1007/s00018-025-05660-6

Hu Yuwei, Ma Yanni, Liu Lele, Hong Yan, Wang Guanghui, Tang Beisha, Guo Jifeng, Yang Peng, Cao Ying, Ren Haigang

Association of Glial Activation and α-Synuclein Pathology in Parkinson's Disease

帕金森病中神经胶质细胞活化与α-突触核蛋白病理的关联

期刊:Neuroscience Bulletin

影响因子:

doi:10.1007/s12264-022-00957-z

Wang, Rui; Ren, Haigang; Kaznacheyeva, Elena; Lu, Xiaojun; Wang, Guanghui

Bioinformatics and network-based screening and discovery of potential molecular targets and small molecular drugs for breast cancer

生物信息学和基于网络的乳腺癌潜在分子靶点和小分子药物的筛选与发现

期刊:Frontiers in Pharmacology

影响因子:4.8

doi:10.3389/fphar.2022.942126

Alam, Md Shahin; Sultana, Adiba; Sun, Hongyang; Wu, Jin; Guo, Fanfan; Li, Qing; Ren, Haigang; Hao, Zongbing; Zhang, Yi; Wang, Guanghui

Editorial: Glial cells and immune cells in neuroinflammatory and neurodegenerative diseases

社论：神经炎症和神经退行性疾病中的神经胶质细胞和免疫细胞

期刊:Frontiers in Aging Neuroscience

影响因子:4.5

doi:10.3389/fnagi.2022.1120649

Wang, Rui; Ren, Haigang; Hu, Li-Fang; Song, Ning; Long, Ling; You, Zili; Wang, Guanghui

The Cross-Links of Endoplasmic Reticulum Stress, Autophagy, and Neurodegeneration in Parkinson's Disease

帕金森病中内质网应激、自噬和神经退行性变之间的关联

期刊:Frontiers in Aging Neuroscience

影响因子:4.5

doi:10.3389/fnagi.2021.691881

Ren, Haigang; Zhai, Wanqing; Lu, Xiaojun; Wang, Guanghui

Role of the C9ORF72 Gene in the Pathogenesis of Amyotrophic Lateral Sclerosis and Frontotemporal Dementia

C9ORF72基因在肌萎缩侧索硬化症和额颞叶痴呆发病机制中的作用

期刊:Neuroscience Bulletin

影响因子:5.8

doi:10.1007/s12264-020-00567-7

Hao, Zongbing; Wang, Rui; Ren, Haigang; Wang, Guanghui

发表日期：2020

文献求助收藏

Imbalance of Lysine Acetylation Contributes to the Pathogenesis of Parkinson's Disease

赖氨酸乙酰化失衡是帕金森病发病机制的促成因素

期刊:International Journal of Molecular Sciences

影响因子:4.9

doi:10.3390/ijms21197182

Wang, Rui; Sun, Hongyang; Wang, Guanghui; Ren, Haigang

P7C3 Inhibits LPS-Induced Microglial Activation to Protect Dopaminergic Neurons Against Inflammatory Factor-Induced Cell Death in vitro and in vivo

P7C3抑制LPS诱导的小胶质细胞活化，从而在体外和体内保护多巴胺能神经元免受炎症因子诱导的细胞死亡。

期刊:Frontiers in Cellular Neuroscience

影响因子:4

doi:10.3389/fncel.2018.00400

Gu, Chao; Hu, Qingsong; Wu, Jiayuan; Mu, Chenchen; Ren, Haigang; Liu, Chun-Feng; Wang, Guanghui

P7C3 inhibits GSK3β activation to protect dopaminergic neurons against neurotoxin-induced cell death in vitro and in vivo

P7C3通过抑制GSK3β活化，在体外和体内保护多巴胺能神经元免受神经毒素诱导的细胞死亡。

期刊:Cell Death & Disease

影响因子:9.6

doi:10.1038/cddis.2017.250

Gu, Chao; Zhang, Yan; Hu, Qingsong; Wu, Jiayuan; Ren, Haigang; Liu, Chun-Feng; Wang, Guanghui

Hax-1 is rapidly degraded by the proteasome dependent on its PEST sequence

Hax-1 依赖于其 PEST 序列而被蛋白酶体快速降解。

Loss of NgBR causes neuronal damage through decreasing KAT7-mediated RFX1 acetylation and FGF1 expression.

NgBR 的缺失会导致神经元损伤，其机制是通过降低 KAT7 介导的 RFX1 乙酰化和 FGF1 表达

Association of Glial Activation and α-Synuclein Pathology in Parkinson's Disease

帕金森病中神经胶质细胞活化与α-突触核蛋白病理的关联

Bioinformatics and network-based screening and discovery of potential molecular targets and small molecular drugs for breast cancer

生物信息学和基于网络的乳腺癌潜在分子靶点和小分子药物的筛选与发现

Editorial: Glial cells and immune cells in neuroinflammatory and neurodegenerative diseases

社论：神经炎症和神经退行性疾病中的神经胶质细胞和免疫细胞

The Cross-Links of Endoplasmic Reticulum Stress, Autophagy, and Neurodegeneration in Parkinson's Disease

帕金森病中内质网应激、自噬和神经退行性变之间的关联

Role of the C9ORF72 Gene in the Pathogenesis of Amyotrophic Lateral Sclerosis and Frontotemporal Dementia

C9ORF72基因在肌萎缩侧索硬化症和额颞叶痴呆发病机制中的作用

Imbalance of Lysine Acetylation Contributes to the Pathogenesis of Parkinson's Disease

赖氨酸乙酰化失衡是帕金森病发病机制的促成因素

P7C3 Inhibits LPS-Induced Microglial Activation to Protect Dopaminergic Neurons Against Inflammatory Factor-Induced Cell Death in vitro and in vivo

P7C3抑制LPS诱导的小胶质细胞活化，从而在体外和体内保护多巴胺能神经元免受炎症因子诱导的细胞死亡。

P7C3 inhibits GSK3β activation to protect dopaminergic neurons against neurotoxin-induced cell death in vitro and in vivo

P7C3通过抑制GSK3β活化，在体外和体内保护多巴胺能神经元免受神经毒素诱导的细胞死亡。