Reto Asmis相关文献与解析，生知库 | 链接生命科学的每一步

日期影响因子

Glutaredoxin 1 controls monocyte reprogramming during nutrient stress and protects mice against obesity and atherosclerosis in a sex-specific manner

谷氧还蛋白 1 在营养应激期间控制单核细胞重编程，并以性别特异性的方式保护小鼠免于肥胖和动脉粥样硬化

期刊:Nature Communications

影响因子:14.7

doi:10.1038/s41467-022-28433-2

Yong Joo Ahn #, Luxi Wang #, Sina Tavakoli, Huynh Nga Nguyen, John D Short, Reto Asmis

动脉粥样硬化

发表日期：2022

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Dietary 23-hydroxy ursolic acid protects against atherosclerosis and obesity by preventing dyslipidemia-induced monocyte priming and dysfunction

膳食中的 23-羟基熊果酸可通过预防血脂异常引起的单核细胞启动和功能障碍来预防动脉粥样硬化和肥胖

期刊:Atherosclerosis

影响因子:4.9

doi:10.1016/j.atherosclerosis.2018.06.882

Huynh Nga Nguyen, Yong Joo Ahn, Edward Antonio Medina, Reto Asmis

细胞生物学

动脉粥样硬化

发表日期：2018

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Acute maternal oxidant exposure causes susceptibility of the fetal brain to inflammation and oxidative stress

急性母体氧化剂暴露导致胎儿大脑易受炎症和氧化应激

期刊:Journal of Neuroinflammation

影响因子:9.3

doi:10.1186/s12974-017-0965-8

Feroz Akhtar, Christopher A Rouse, Gabriel Catano, Marcus Montalvo, Sarah L Ullevig, Reto Asmis, Kusum Kharbanda, Shivani K Maffi

发表日期：2017

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Dyslipidemic Diet-Induced Monocyte "Priming" and Dysfunction in Non-Human Primates Is Triggered by Elevated Plasma Cholesterol and Accompanied by Altered Histone Acetylation

非人类灵长类动物中，血脂异常饮食诱发的单核细胞“启动”和功能障碍是由血浆胆固醇升高引发的，并伴有组蛋白乙酰化改变

期刊:Frontiers in Immunology

影响因子:5.7

doi:10.3389/fimmu.2017.00958

John D Short, Sina Tavakoli, Huynh Nga Nguyen, Ana Carrera, Chelbee Farnen, Laura A Cox, Reto Asmis

细胞生物学

发表日期：2017

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Protein S-Glutathionylation Mediates Macrophage Responses to Metabolic Cues from the Extracellular Environment

蛋白质 S-谷胱甘肽化介导巨噬细胞对细胞外环境代谢线索的反应

期刊:Antioxidants & Redox Signaling

影响因子:5.9

doi:10.1089/ars.2015.6531

Sarah L Ullevig, Hong Seok Kim, John D Short, Sina Tavakoli, Susan T Weintraub, Kevin Downs, Reto Asmis

发表日期：2016

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Macrophage TGF- β 1 and the Proapoptotic Extracellular Matrix Protein BIGH3 Induce Renal Cell Apoptosis in Prediabetic and Diabetic Conditions

巨噬细胞 TGF- β 1 和促凋亡细胞外基质蛋白 BIGH3 在糖尿病前期和糖尿病状态下诱导肾细胞凋亡

doi:10.4236/ijcm.2016.77055

Robert J Moritz, Richard G LeBaron, Clyde F Phelix, Rajesha Rupaimoole, Hong Seok Kim, Andrew Tsin, Reto Asmis

细胞生物学

发表日期：2016

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BIGH3 protein and macrophages in retinal endothelial cell apoptosis

BIGH3蛋白与巨噬细胞在视网膜内皮细胞凋亡中的作用

期刊:Apoptosis

影响因子:6.1

doi:10.1007/s10495-014-1052-6

Albert A Mondragon, Brandi S Betts-Obregon, Robert J Moritz, Kalpana Parvathaneni, Mary M Navarro, Hong Seok Kim, Chi Fung Lee, Richard G LeBaron, Reto Asmis, Andrew T Tsin

细胞生物学

发表日期：2015

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Differences in forward angular light scattering distributions between M1 and M2 macrophages

M1 和 M2 巨噬细胞前向角光散射分布的差异

期刊:Journal of Biomedical Optics

doi:10.1117/1.JBO.20.11.115002

David L Halaney, Aydin Zahedivash, Jennifer E Phipps, Tianyi Wang, Jordan Dwelle, Claude Jourdan Le Saux, Reto Asmis, Thomas E Milner, Marc D Feldman

细胞生物学

发表日期：2015

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Ursolic acid protects monocytes against metabolic stress-induced priming and dysfunction by preventing the induction of Nox4

熊果酸通过阻止 Nox4 的诱导，保护单核细胞免受代谢应激引起的启动和功能障碍

期刊:Redox Biology

影响因子:10.7

doi:10.1016/j.redox.2014.01.003

Sarah L Ullevig, Hong Seok Kim, Huynh Nga Nguyen, William S Hambright, Andrew J Robles, Sina Tavakoli, Reto Asmis

发表日期：2014

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Bioenergetic profiles diverge during macrophage polarization: implications for the interpretation of 18F-FDG PET imaging of atherosclerosis

巨噬细胞极化过程中生物能量特征出现分歧：对解释动脉粥样硬化 18F-FDG PET 成像的意义

期刊:Journal of Nuclear Medicine

影响因子:9.1

doi:10.2967/jnumed.112.119099

Sina Tavakoli, Debora Zamora, Sarah Ullevig, Reto Asmis

细胞生物学

动脉粥样硬化

发表日期：2013

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