文献库，生知库 | 链接生命科学的每一步

Santaolalla Sanchez, Francisco Javier; Gutierrez Posso, Juan David; Santaolalla Montoya, Francisco; Zabala, Javier Aitor; Arrizabalaga-Iriondo, Ane; Revuelta, Miren; Sánchez Del Rey, Ana

发表日期：2024

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Therapeutic role of voltage-gated potassium channels in age-related neurodegenerative diseases

电压门控钾通道在年龄相关神经退行性疾病中的治疗作用

期刊:Frontiers in Cellular Neuroscience

影响因子:4

doi:10.3389/fncel.2024.1406709

Urrutia, Janire; Arrizabalaga-Iriondo, Ane; Sanchez-Del-Rey, Ana; Martinez-Ibargüen, Agustín; Gallego, Mónica; Casis, Oscar; Revuelta, Miren

神经科学

发表日期：2024

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Microglia-Mediated Inflammation and Neural Stem Cell Differentiation in Alzheimer's Disease: Possible Therapeutic Role of K(V)1.3 Channel Blockade

小胶质细胞介导的炎症和神经干细胞分化在阿尔茨海默病中的作用：K(V)1.3通道阻滞剂的潜在治疗作用

期刊:Frontiers in Cellular Neuroscience

影响因子:4

doi:10.3389/fncel.2022.868842

Revuelta, Miren; Urrutia, Janire; Villarroel, Alvaro; Casis, Oscar

Immunology/Inflammation

In vitro P38MAPK inhibition in aged astrocytes decreases reactive astrocytes, inflammation and increases nutritive capacity after oxygen-glucose deprivation

体外抑制衰老星形胶质细胞中的P38MAPK可减少反应性星形胶质细胞和炎症，并提高缺氧缺糖后的营养能力。

期刊:Aging-Us

影响因子:3.9

doi:10.18632/aging.202651

Revuelta, Miren; Elicegui, Amaia; Scheuer, Till; Endesfelder, Stefanie; Bührer, Christoph; Moreno-Cugnon, Leire; Matheu, Ander; Schmitz, Thomas

Glial Factors Regulating White Matter Development and Pathologies of the Cerebellum

神经胶质因子调控小脑白质发育和病理

期刊:Neurochemical Research

影响因子:3.8

doi:10.1007/s11064-020-02961-z

Revuelta, Miren; Scheuer, Till; Chew, Li-Jin; Schmitz, Thomas

Neuronal p38α mediates age-associated neural stem cell exhaustion and cognitive decline

神经元p38α介导与年龄相关的神经干细胞耗竭和认知衰退

期刊:Aging Cell

影响因子:7.1

doi:10.1111/acel.13044

Moreno-Cugnon, Leire; Revuelta, Miren; Arrizabalaga, Olatz; Colie, Sandra; Moreno-Valladares, Manuel; Jimenez-Blasco, Daniel; Gil-Bea, Francisco; Llarena, Irantzu; Bolaños, Juan Pedro; Nebreda, Angel R; Matheu, Ander

Autophagy in stem cell aging

干细胞衰老中的自噬

期刊:Aging Cell

影响因子:7.1

doi:10.1111/acel.12655

Revuelta, Miren; Matheu, Ander

Role of Antioxidants in Neonatal Hypoxic-Ischemic Brain Injury: New Therapeutic Approaches.

抗氧化剂在新生儿缺氧缺血性脑损伤中的作用：新的治疗方法

期刊:International Journal of Molecular Sciences

影响因子:4.9

doi:10.3390/ijms18020265

Arteaga Olatz, Ãlvarez Antonia, Revuelta Miren, Santaolalla Francisco, Urtasun Andoni, Hilario Enrique

Antioxidant Treatments Recover the Alteration of Auditory-Evoked Potentials and Reduce Morphological Damage in the Inferior Colliculus after Perinatal Asphyxia in Rat.

抗氧化治疗可恢复围产期窒息大鼠下丘听觉诱发电位的改变并减少其形态损伤

期刊:Brain Pathology

影响因子:6.2

doi:10.1111/bpa.12272

Revuelta Miren, Arteaga Olatz, Montalvo Haizea, Alvarez Antonia, Hilario Enrique, Martinez-IbargÃ¼en Agustin

毒理研究

发表日期：2016

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Editorial: Ion channels in the nervous system

社论：神经系统中的离子通道

Pathogenesis and New Pharmacological Approaches to Noise-Induced Hearing Loss: A Systematic Review

噪声性听力损失的发病机制及新的药物治疗方法：系统综述

Therapeutic role of voltage-gated potassium channels in age-related neurodegenerative diseases

电压门控钾通道在年龄相关神经退行性疾病中的治疗作用

Microglia-Mediated Inflammation and Neural Stem Cell Differentiation in Alzheimer's Disease: Possible Therapeutic Role of K(V)1.3 Channel Blockade

小胶质细胞介导的炎症和神经干细胞分化在阿尔茨海默病中的作用：K(V)1.3通道阻滞剂的潜在治疗作用

In vitro P38MAPK inhibition in aged astrocytes decreases reactive astrocytes, inflammation and increases nutritive capacity after oxygen-glucose deprivation

体外抑制衰老星形胶质细胞中的P38MAPK可减少反应性星形胶质细胞和炎症，并提高缺氧缺糖后的营养能力。

Glial Factors Regulating White Matter Development and Pathologies of the Cerebellum

神经胶质因子调控小脑白质发育和病理

Neuronal p38α mediates age-associated neural stem cell exhaustion and cognitive decline

神经元p38α介导与年龄相关的神经干细胞耗竭和认知衰退

Autophagy in stem cell aging

干细胞衰老中的自噬

Role of Antioxidants in Neonatal Hypoxic-Ischemic Brain Injury: New Therapeutic Approaches.

抗氧化剂在新生儿缺氧缺血性脑损伤中的作用：新的治疗方法

Antioxidant Treatments Recover the Alteration of Auditory-Evoked Potentials and Reduce Morphological Damage in the Inferior Colliculus after Perinatal Asphyxia in Rat.

抗氧化治疗可恢复围产期窒息大鼠下丘听觉诱发电位的改变并减少其形态损伤