Rhaleb相关文献与解析，生知库 | 链接生命科学的每一步

Endothelial CHOP as a central mechanism in renovascular hypertension-induced vascular endothelial dysfunction and cardiac fibrosis.

内皮CHOP是肾血管性高血压诱发血管内皮功能障碍和心脏纤维化的核心机制

期刊:Cellular and Molecular Life Sciences

影响因子:6.2

doi:10.1007/s00018-025-05741-6

Srinivas B, Alluri K, Fortuno P, Rizzi M, Suhail H, Rhaleb N, Matrougui K

Ac-SDKP and eplerenone confer additive cardioprotection against angiotensin II-induced cardiac injury in C57BL/6J mice

Ac-SDKP 和依普利酮对 C57BL/6J 小鼠血管紧张素 II 诱导的心脏损伤具有叠加的心脏保护作用

期刊:Journal of Molecular and Cellular Cardiology Plus

影响因子:2.2

doi:10.1016/j.jmccpl.2025.100485

Hamid, Suhail; Sarkar, Sarah; Peng, Hongmei; Khalid, Matrougui; Ortiz, Pablo A; Xu, Jiang; Liao, Tang-Dong; Knight, Robert A; Rhaleb, Nour-Eddine

心血管

发表日期：2025

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Deletion of CHOP in beta cell protects mice from cardiovascular complications in type 2 diabetes: evidence from a pre-clinical mouse model.

β细胞中CHOP的缺失可保护小鼠免受2型糖尿病心血管并发症的侵害：来自临床前小鼠模型的证据。

期刊:Journal of Molecular and Cellular Cardiology Plus

影响因子:2.2

doi:10.1016/j.jmccpl.2025.100492

Srinivas Balaji, Alluri Kiran, Rhaleb Nour-Eddine, Matrougui Khalid

Relevance of Prescribing Serum Immunofixation Electrophoresis in the Diagnosis of Monoclonal Gammopathies

血清免疫固定电泳在单克隆丙种球蛋白病诊断中的意义

期刊:Cureus Journal of Medical Science

影响因子:1.3

doi:10.7759/cureus.86339

Fadili, Hajar; Ouazzani, Hamza; Rhaleb, Ikrame; Bendi, Ines; Snoussi, Malak; Zrara, Abdelhamid; Bamou, Youssef; El Bakkouri, Jalila

免疫/内分泌

发表日期：2025

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Ac-SDKP attenuates ER stress-stimulated collagen production in cardiac fibroblasts by inhibiting CHOP-mediated NF-κB expression

Ac-SDKP 通过抑制 CHOP 介导的 NF-κB 表达来减弱内质网应激刺激的心脏成纤维细胞胶原蛋白生成

期刊:Frontiers in Pharmacology

影响因子:4.4

doi:10.3389/fphar.2024.1352222

Hamid Suhail, Hongmei Peng, Khalid Matrougui, Nour-Eddine Rhaleb

Role of plasmacytoid dendritic cells in vascular dysfunction in mice with renovascular hypertension

浆细胞样树突状细胞在肾血管性高血压小鼠血管功能障碍中的作用

期刊:Heliyon

影响因子:3.4

doi:10.1016/j.heliyon.2024.e31799

Balaji Srinivas, Kiran Alluri, Nour-Eddine Rhaleb, Souad Belmadani, Khalid Matrougui

Unveiling the vulnerability of C57BL/6J female mice to HFpEF and its related complications

揭示 C57BL/6J 雌性小鼠对 HFpEF 及其相关并发症的脆弱性

期刊:Journal of Molecular and Cellular Cardiology Plus

影响因子:

doi:10.1016/j.jmccpl.2024.100062

B Srinivas, K Alluri, H Peng, P A Ortiz, J Xu, H N Sabbah, N E Rhaleb, K Matrougui

发表日期：2024

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ALMS1 KO rat: a new model of metabolic syndrome with spontaneous hypertension

ALMS1 KO大鼠：一种新的伴有自发性高血压的代谢综合征模型

期刊:

影响因子:

doi:10.1101/2024.09.22.614364

Ankita B Jaykumar, Sumit R Monu, Mariela Mendez, Nour-Eddine Rhaleb, Pablo A Ortiz

Alms1 KO Rat: A New Model of Cardiometabolic Syndrome With Spontaneous Hypertension

Alms1 KO 大鼠：一种新的伴有自发性高血压的心血管代谢综合征模型

Novel insights into beta cell ER stress CHOP and its role in HFpEF development.

对β细胞内质网应激CHOP及其在HFpEF发展中的作用的新见解

Endothelial CHOP as a central mechanism in renovascular hypertension-induced vascular endothelial dysfunction and cardiac fibrosis.

内皮CHOP是肾血管性高血压诱发血管内皮功能障碍和心脏纤维化的核心机制

Ac-SDKP and eplerenone confer additive cardioprotection against angiotensin II-induced cardiac injury in C57BL/6J mice

Ac-SDKP 和依普利酮对 C57BL/6J 小鼠血管紧张素 II 诱导的心脏损伤具有叠加的心脏保护作用

Deletion of CHOP in beta cell protects mice from cardiovascular complications in type 2 diabetes: evidence from a pre-clinical mouse model.

β细胞中CHOP的缺失可保护小鼠免受2型糖尿病心血管并发症的侵害：来自临床前小鼠模型的证据。

Relevance of Prescribing Serum Immunofixation Electrophoresis in the Diagnosis of Monoclonal Gammopathies

血清免疫固定电泳在单克隆丙种球蛋白病诊断中的意义

Ac-SDKP attenuates ER stress-stimulated collagen production in cardiac fibroblasts by inhibiting CHOP-mediated NF-κB expression

Ac-SDKP 通过抑制 CHOP 介导的 NF-κB 表达来减弱内质网应激刺激的心脏成纤维细胞胶原蛋白生成

Role of plasmacytoid dendritic cells in vascular dysfunction in mice with renovascular hypertension

浆细胞样树突状细胞在肾血管性高血压小鼠血管功能障碍中的作用

Unveiling the vulnerability of C57BL/6J female mice to HFpEF and its related complications

揭示 C57BL/6J 雌性小鼠对 HFpEF 及其相关并发症的脆弱性

ALMS1 KO rat: a new model of metabolic syndrome with spontaneous hypertension

ALMS1 KO大鼠：一种新的伴有自发性高血压的代谢综合征模型