日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

NLRP3 inflammasome-driven hemophagocytic lymphohistiocytosis occurs independent of IL-1β and IL-18 and is targetable by BET inhibitors.

NLRP3炎症小体驱动的噬血细胞性淋巴组织细胞增生症的发生与IL-1β和IL-18无关,并且是BET抑制剂的靶点

Shojaee Farzaneh, Azadian Esmaeel, Wong Min Xian, Ma Xiuquan, Rickard James, Pang Jiyi, Hall Catherine, Kueh Andrew J, Masters Seth L, Rioja Inmaculada, Prinjha Rab K, Doerflinger Marcel, Lawlor Kate E, Rashidi Maryam, Vince James E

A Design of Experiments Approach to Identify Critical Processing Parameters for Manufacture of an Autologous Platelet Gel for Diabetic Foot Ulcer

采用实验设计方法确定自体血小板凝胶治疗糖尿病足溃疡的关键工艺参数

Olszewska, Aleksandra; Egorova, Olga; Gaggia, Gabriella; Mylona, Kalliopi; Pitchford, Simon; Rickard, James; Forbes, Ben

Correction to: RIPK1 prevents TRADD-driven, but TNFR1 independent, apoptosis during development

更正:RIPK1 在发育过程中阻止 TRADD 驱动但 TNFR1 非依赖性的细胞凋亡

Anderton, Holly; Bandala-Sanchez, Esther; Simpson, Daniel S; Rickard, James A; Ng, Ashley P; Di Rago, Ladina; Hall, Cathrine; Vince, James E; Silke, John; Liccardi, Gianmaria; Feltham, Rebecca

Manufacture and Initial Characterisation of RAPID(TM) Biodynamic Haematogel, an Autologous Platelet and Leukocyte-Rich Plasma Gel for Diabetic Foot Ulcers.

RAPID(TM)生物动力血液凝胶(一种用于治疗糖尿病足溃疡的自体血小板和白细胞富集血浆凝胶)的制造和初步表征

Olszewska Aleksandra, Duan Jiajing, Javorovic Jana, Chan K L Andrew, Rickard James, Pitchford Simon, Forbes Ben

Correction: TRAF2 regulates TNF and NF-κB signalling to suppress apoptosis and skin inflammation independently of Sphingosine kinase 1

更正:TRAF2 调节 TNF 和 NF-κB 信号通路,从而抑制细胞凋亡和皮肤炎症,且该过程独立于鞘氨醇激酶 1。

Etemadi, Nima; Chopin, Michael; Anderton, Holly; Tanzer, Maria C; Rickard, James A; Abeysekera, Waruni; Hall, Cathrine; Spall, Sukhdeep K; Wang, Bing; Xiong, Yuquan; Hla, Timothy; Pitson, Stuart M; Bonder, Claudine S; Wong, Wendy Wei-Lynn; Ernst, Matthias; Smyth, Gordon K; Vaux, David L; Nutt, Stephen L; Nachbur, Ueli; Silke, John

In TNF-stimulated cells, RIPK1 promotes cell survival by stabilizing TRAF2 and cIAP1, which limits induction of non-canonical NF-κB and activation of caspase-8

在TNF刺激的细胞中,RIPK1通过稳定TRAF2和cIAP1来促进细胞存活,从而限制非经典NF-κB的诱导和caspase-8的激活。

Gentle, Ian E; Wong, W Wei-Lynn; Evans, Joseph M; Bankovacki, Alexandra; Cook, Wendy D; Khan, Nufail R; Nachbur, Ulrich; Rickard, James; Anderton, Holly; Moulin, Maryline; Lluis, Josep Maria; Moujalled, Donia M; Silke, John; Vaux, David L