日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Stromal Cells in the Pathogenesis of Inflammatory Bowel Disease

炎症性肠病发病机制中的基质细胞

Barnhoorn, M C; Hakuno, S K; Bruckner, R S; Rogler, G; Hawinkels, L J A C; Scharl, M

Severity of local inflammation does not impact development of fibrosis in mouse models of intestinal fibrosis

局部炎症的严重程度并不影响小鼠肠道纤维化模型中纤维化的发展。

Hünerwadel, A; Fagagnini, S; Rogler, G; Lutz, C; Jaeger, S U; Mamie, C; Weder, B; Ruiz, P A; Hausmann, M

Myeloid differentiation primary response gene (MyD) 88 signalling is not essential for intestinal fibrosis development

髓系分化原初反应基因(MyD)88信号通路并非肠道纤维化发展所必需。

Lutz, C; Weder, B; Hünerwadel, A; Fagagnini, S; Lang, B; Beerenwinkel, N; Rossel, J B; Rogler, G; Misselwitz, B; Hausmann, M

Increased lymphocyte apoptosis in mouse models of colitis upon ABT-737 treatment is dependent upon BIM expression

在小鼠结肠炎模型中,ABT-737治疗后淋巴细胞凋亡增加依赖于BIM表达。

Lutz, C; Mozaffari, M; Tosevski, V; Caj, M; Cippà, P; McRae, B L; Graff, C L; Rogler, G; Fried, M; Hausmann, M

Aluminum enhances inflammation and decreases mucosal healing in experimental colitis in mice

铝会加剧小鼠实验性结肠炎的炎症反应并抑制黏膜愈合。

Pineton de Chambrun, G; Body-Malapel, M; Frey-Wagner, I; Djouina, M; Deknuydt, F; Atrott, K; Esquerre, N; Altare, F; Neut, C; Arrieta, M C; Kanneganti, T-D; Rogler, G; Colombel, J-F; Cortot, A; Desreumaux, P; Vignal, C

Impaired removal of Vβ8(+) lymphocytes aggravates colitis in mice deficient for B cell lymphoma-2-interacting mediator of cell death (Bim)

Vβ8(+)淋巴细胞清除受损会加重缺乏B细胞淋巴瘤-2相互作用细胞死亡介质(Bim)的小鼠的结肠炎。

Leucht, K; Caj, M; Fried, M; Rogler, G; Hausmann, M

Recipient NOD2/CARD15 status affects cellular infiltrates in human intestinal graft-versus-host disease

受体NOD2/CARD15状态影响人类肠道移植物抗宿主病中的细胞浸润

Landfried, K; Bataille, F; Rogler, G; Brenmoehl, J; Kosovac, K; Wolff, D; Hilgendorf, I; Hahn, J; Edinger, M; Hoffmann, P; Obermeier, F; Schoelmerich, J; Andreesen, R; Holler, E

(GT)N dinucleotide repeat polymorphism of haem oxygenase-1 promotor region is not associated with inflammatory bowel disease risk or disease course

血红素加氧酶-1启动子区域的(GT)N二核苷酸重复序列多态性与炎症性肠病风险或疾病进程无关。

Hausmann, M; Paul, G; Kellermeier, S; Frey, I; Schölmerich, J; Falk, W; Menzel, K; Fried, M; Herfarth, H; Rogler, G

Regulation of galectin-3 function in mucosal fibroblasts: potential role in mucosal inflammation.

黏膜成纤维细胞中半乳糖凝集素-3功能的调控:在黏膜炎症中的潜在作用

Lippert E, Gunckel M, Brenmoehl J, Bataille F, Falk W, Scholmerich J, Obermeier F, Rogler G

Soluble galectin-3 is a strong, colonic epithelial-cell-derived, lamina propria fibroblast-stimulating factor

可溶性半乳糖凝集素-3是一种强效的、源自结肠上皮细胞的固有层成纤维细胞刺激因子。

Lippert, E; Falk, W; Bataille, F; Kaehne, T; Naumann, M; Goeke, M; Herfarth, H; Schoelmerich, J; Rogler, G