日期 影响因子
日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:
–

Splenic CD169(+)Tim4(+) Marginal Metallophilic Macrophages Are Essential for Wound Healing After Myocardial Infarction

脾脏CD169(+)Tim4(+)边缘金属嗜性巨噬细胞对心肌梗死后的伤口愈合至关重要

期刊:Circulation
影响因子:38.6
doi:10.1161/CIRCULATIONAHA.124.071772
Ismahil, Mohamed Ameen; Zhou, Guihua; Rajasekar, Shreya; Gao, Min; Bansal, Shyam S; Patel, Bindiya; Limdi, Nita; Xie, Min; Antipenko, Sergey; Rokosh, Gregg; Hamid, Tariq; Prabhu, Sumanth D
巨噬细胞
CD16
巨噬细胞
心血管
细胞生物学
心肌梗塞
免疫/内分泌
发表日期：2025
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CD206(+)IL-4Rα(+) Macrophages Are Drivers of Adverse Cardiac Remodeling in Ischemic Cardiomyopathy

CD206(+)IL-4Rα(+)巨噬细胞是缺血性心肌病中不良心脏重塑的驱动因素

期刊:Circulation
影响因子:38.6
doi:10.1161/CIRCULATIONAHA.124.072411
Wang, Qiongxin; Ismahil, Mohamed Ameen; Zhu, Yujie; Rokosh, Gregg; Hamid, Tariq; Zhou, Guihua; Pogwizd, Steven M; Prabhu, Sumanth D
巨噬细胞
心肌病
巨噬细胞
CD206
心血管
CD20
细胞生物学
CD2
IL-4
免疫/内分泌
发表日期：2025
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Correction: The heme oxygenase 1 inducer (CoPP) protects human cardiac stem cells against apoptosis through activation of the extracellular signal-related kinase (ERK)/NRF2 signaling pathway and cytokine release

更正：血红素加氧酶1诱导剂（CoPP）通过激活细胞外信号调节激酶（ERK）/NRF2信号通路和细胞因子释放来保护人类心脏干细胞免于凋亡。

期刊:Journal of Biological Chemistry
影响因子:3.9
doi:10.1074/jbc.AAC119.008182
Cai, Chuanxi; Teng, Lei; Vu, Duc; He, Jia-Qiang; Guo, Yiru; Li, Qianghong; Tang, Xian-Liang; Rokosh, Gregg; Bhatnagar, Aruni; Bolli, Roberto
发育与干细胞
凋亡
表观遗传
信号转导
MAPK/ERK
NRF2
干细胞
细胞生物学
免疫/内分泌
发表日期：2019
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Elevating CXCR7 Improves Angiogenic Function of EPCs via Akt/GSK-3β/Fyn-Mediated Nrf2 Activation in Diabetic Limb Ischemia

CXCR7 升高可通过 Akt/GSK-3β/Fyn 介导的 Nrf2 激活改善糖尿病肢体缺血中 EPC 的血管生成功能

期刊:Circulation Research
影响因子:16.2
doi:10.1161/CIRCRESAHA.117.310619
Dai, Xiaozhen; Yan, Xiaoqing; Zeng, Jun; Chen, Jing; Wang, Yuehui; Chen, Jun; Li, Yan; Barati, Michelle T; Wintergerst, Kupper A; Pan, Kejian; Nystoriak, Matthew A; Conklin, Daniel J; Rokosh, Gregg; Epstein, Paul N; Li, Xiaokun; Tan, Yi
糖尿病
血管生成
代谢
发表日期：2017
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Leukocyte iNOS is required for inflammation and pathological remodeling in ischemic heart failure

白细胞iNOS是缺血性心力衰竭中炎症和病理重塑所必需的

期刊:Basic Research in Cardiology
影响因子:8
doi:10.1007/s00395-017-0609-2
Kingery, Justin R; Hamid, Tariq; Lewis, Robert K; Ismahil, Mohamed Ameen; Bansal, Shyam S; Rokosh, Gregg; Townes, Tim M; Ildstad, Suzanne T; Jones, Steven P; Prabhu, Sumanth D
炎症
炎症/感染
心血管
iNOS
细胞生物学
心力衰竭
发表日期：2017
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Long-Term Outcome of Administration of c-kit(POS) Cardiac Progenitor Cells After Acute Myocardial Infarction: Transplanted Cells Do not Become Cardiomyocytes, but Structural and Functional Improvement and Proliferation of Endogenous Cells Persist for at Least One Year

急性心肌梗死后移植c-kit(POS)心脏祖细胞的长期疗效：移植的细胞不会分化为心肌细胞，但内源性细胞的结构和功能改善以及增殖至少持续一年。

期刊:Circulation Research
影响因子:16.2
doi:10.1161/CIRCRESAHA.115.307647
Tang, Xian-Liang; Li, Qianhong; Rokosh, Gregg; Sanganalmath, Santosh K; Chen, Ning; Ou, Qinghui; Stowers, Heather; Hunt, Greg; Bolli, Roberto
细胞生物学
心肌梗塞
心血管
发表日期：2016
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Inhibition of mammalian target of rapamycin protects against reperfusion injury in diabetic heart through STAT3 signaling

抑制哺乳动物雷帕霉素靶蛋白可通过STAT3信号通路保护糖尿病心脏免受再灌注损伤

期刊:Basic Research in Cardiology
影响因子:8
doi:10.1007/s00395-015-0486-5
Das, Anindita; Salloum, Fadi N; Filippone, Scott M; Durrant, David E; Rokosh, Gregg; Bolli, Roberto; Kukreja, Rakesh C
JAK/STAT
信号转导
代谢
STAT3
糖尿病
发表日期：2015
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Cardiomyocyte-restricted overexpression of extracellular superoxide dismutase increases nitric oxide bioavailability and reduces infarct size after ischemia/reperfusion

心肌细胞特异性过表达细胞外超氧化物歧化酶可提高一氧化氮的生物利用度，并减少缺血/再灌注后的梗死面积。

期刊:Basic Research in Cardiology
影响因子:8
doi:10.1007/s00395-012-0305-1
Obal, Detlef; Dai, Shujing; Keith, Rachel; Dimova, Neviana; Kingery, Justin; Zheng, Yu-Ting; Zweier, Jay; Velayutham, Murugesan; Prabhu, Sumanth D; Li, Qianghong; Conklin, Daniel; Yang, Dan; Bhatnagar, Aruni; Bolli, Roberto; Rokosh, Gregg
细胞生物学
发表日期：2012
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Hepatocyte growth factor/Met gene transfer in cardiac stem cells--potential for cardiac repair

肝细胞生长因子/Met基因转移至心脏干细胞——心脏修复的潜力

期刊:Basic Research in Cardiology
影响因子:8
doi:10.1007/s00395-010-0102-7
Madonna, Rosalinda; Rokosh, Gregg; De Caterina, Raffaele; Bolli, Roberto
细胞生物学
发育与干细胞
干细胞
发表日期：2010
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Chronic AMD3100 antagonism of SDF-1alpha-CXCR4 exacerbates cardiac dysfunction and remodeling after myocardial infarction

慢性 AMD3100 对 SDF-1α-CXCR4 的拮抗作用会加剧心肌梗死后的心脏功能障碍和重塑。

期刊:Journal of Molecular and Cellular Cardiology
影响因子:4.7
doi:10.1016/j.yjmcc.2010.07.010
Dai, Shujing; Yuan, Fangping; Mu, Jingyao; Li, Chengxin; Chen, Ning; Guo, Shangzhi; Kingery, Justin; Prabhu, Sumanth D; Bolli, Roberto; Rokosh, Gregg
AMD
SDF-1α
CXCR4
心血管
心肌梗塞
发表日期：2010
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