Systemic treatment with ubiquitin carboxy terminal hydrolase L1 TAT protein ameliorates axonal injury and reduces functional deficits after traumatic brain injury in mice.
用泛素羧基末端水解酶 L1 TAT 蛋白进行全身治疗可减轻小鼠创伤性脑损伤后的轴突损伤并减少功能缺陷
期刊:Experimental Neurology
影响因子:4.2
doi:10.1016/j.expneurol.2023.114650
Mi Zhiping, Ma Jie, Zeh Dennis J, Rose Marie E, Henchir Jeremy J, Liu Hao, Ma Xiecheng, Cao Guodong, Dixon C Edward, Graham Steven H
