Roth Robert A相关文献与解析，生知库 | 链接生命科学的每一步

Key Characteristics of Human Hepatotoxicants as a Basis for Identification and Characterization of the Causes of Liver Toxicity

人类肝毒性物质的关键特征及其在肝毒性病因鉴定和表征中的作用

doi:10.1002/hep.31999

Rusyn, Ivan; Arzuaga, Xabier; Cattley, Russell C; Corton, J Christopher; Ferguson, Stephen S; Godoy, Patricio; Guyton, Kathryn Z; Kaplowitz, Neil; Khetani, Salman R; Roberts, Ruth A; Roth, Robert A; Smith, Martyn T

Beyond Metabolism: Role of the Immune System in Hepatic Toxicity

代谢之外：免疫系统在肝毒性中的作用

期刊:International Journal of Toxicology

影响因子:1

doi:10.1177/1091581819898399

Hastings, Kenneth L; Green, Martin D; Gao, Bin; Ganey, Patricia E; Roth, Robert A; Burleson, Gary R

What have we learned from animal models of idiosyncratic, drug-induced liver injury?

我们从特异性药物性肝损伤的动物模型中学到了什么？

期刊:

影响因子:

doi:10.1080/17425255.2020.1760246

Roth, Robert A; Ganey, Patricia E

Calcium Contributes to the Cytotoxic Interaction Between Diclofenac and Cytokines

钙离子促进双氯芬酸与细胞因子之间的细胞毒性相互作用

期刊:Toxicological Sciences

影响因子:4.1

doi:10.1093/toxsci/kfv249

Maiuri, Ashley R; Breier, Anna B; Turkus, Jonathan D; Ganey, Patricia E; Roth, Robert A

Platelets and protease-activated receptor-4 contribute to acetaminophen-induced liver injury in mice

血小板和蛋白酶激活受体4参与了对乙酰氨基酚诱导的小鼠肝损伤。

期刊:Blood

影响因子:23.1

doi:10.1182/blood-2014-09-598656

Miyakawa, Kazuhisa; Joshi, Nikita; Sullivan, Bradley P; Albee, Ryan; Brandenberger, Christina; Jaeschke, Hartmut; McGill, Mitchell R; Scott, Michael A; Ganey, Patricia E; Luyendyk, James P; Roth, Robert A

Cytotoxic Synergy Between Cytokines and NSAIDs Associated With Idiosyncratic Hepatotoxicity Is Driven by Mitogen-Activated Protein Kinases.

细胞因子与非甾体抗炎药之间的细胞毒性协同作用与特异性肝毒性相关，这种协同作用是由丝裂原活化蛋白激酶驱动的

期刊:Toxicological Sciences

影响因子:4.1

doi:10.1093/toxsci/kfv091

Maiuri Ashley R, Breier Anna B, Gora Lukas F J, Parkins Robert V, Ganey Patricia E, Roth Robert A

Roles of the hemostatic system and neutrophils in liver injury from co-exposure to amiodarone and lipopolysaccharide

胺碘酮和脂多糖共同作用导致肝损伤，其中止血系统和中性粒细胞的作用

期刊:Toxicological Sciences

影响因子:4.1

doi:10.1093/toxsci/kft170

Lu, Jingtao; Roth, Robert A; Malle, Ernst; Ganey, Patricia E

Pretreatment with TCDD exacerbates liver injury from Concanavalin A: critical role for NK cells

TCDD预处理会加剧刀豆蛋白A引起的肝损伤：NK细胞发挥关键作用

期刊:Toxicological Sciences

影响因子:4.1

doi:10.1093/toxsci/kft174

Fullerton, Aaron M; Roth, Robert A; Ganey, Patricia E

Amiodarone exposure during modest inflammation induces idiosyncrasy-like liver injury in rats: role of tumor necrosis factor-alpha

轻度炎症期间胺碘酮暴露可诱发大鼠特异质瘤样肝损伤：肿瘤坏死因子-α的作用

期刊:Toxicological Sciences

影响因子:4.1

doi:10.1093/toxsci/kfr266

Lu, Jingtao; Jones, A Daniel; Harkema, Jack R; Roth, Robert A; Ganey, Patricia E

Pharmacokinetic and toxicodynamic concepts in idiosyncratic, drug-induced liver injury

特异性药物性肝损伤的药代动力学和毒效学概念

Key Characteristics of Human Hepatotoxicants as a Basis for Identification and Characterization of the Causes of Liver Toxicity

人类肝毒性物质的关键特征及其在肝毒性病因鉴定和表征中的作用

Beyond Metabolism: Role of the Immune System in Hepatic Toxicity

代谢之外：免疫系统在肝毒性中的作用

What have we learned from animal models of idiosyncratic, drug-induced liver injury?

我们从特异性药物性肝损伤的动物模型中学到了什么？

Calcium Contributes to the Cytotoxic Interaction Between Diclofenac and Cytokines

钙离子促进双氯芬酸与细胞因子之间的细胞毒性相互作用

Platelets and protease-activated receptor-4 contribute to acetaminophen-induced liver injury in mice

血小板和蛋白酶激活受体4参与了对乙酰氨基酚诱导的小鼠肝损伤。

Cytotoxic Synergy Between Cytokines and NSAIDs Associated With Idiosyncratic Hepatotoxicity Is Driven by Mitogen-Activated Protein Kinases.

细胞因子与非甾体抗炎药之间的细胞毒性协同作用与特异性肝毒性相关，这种协同作用是由丝裂原活化蛋白激酶驱动的

Roles of the hemostatic system and neutrophils in liver injury from co-exposure to amiodarone and lipopolysaccharide

胺碘酮和脂多糖共同作用导致肝损伤，其中止血系统和中性粒细胞的作用

Pretreatment with TCDD exacerbates liver injury from Concanavalin A: critical role for NK cells

TCDD预处理会加剧刀豆蛋白A引起的肝损伤：NK细胞发挥关键作用

Amiodarone exposure during modest inflammation induces idiosyncrasy-like liver injury in rats: role of tumor necrosis factor-alpha

轻度炎症期间胺碘酮暴露可诱发大鼠特异质瘤样肝损伤：肿瘤坏死因子-α的作用