Ru Chih C相关文献与解析，生知库 | 链接生命科学的每一步

Chun Jong Ho, Kimura Kotohiko, Rajput Monika, Hsu Ming-Hua, Liang Yu-Chuan, Shanbhag Akanksha Ramadas, Chiang Pei-Ju, Jackson Tiffany L B, Huang Ru Chih C

HIF1A

SP1

发表日期：2024

文献求助收藏

Interaction and Collaboration of SP1, HIF-1, and MYC in Regulating the Expression of Cancer-Related Genes to Further Enhance Anticancer Drug Development

SP1、HIF-1 和 MYC 的相互作用与协作在调控癌症相关基因表达中的作用，可进一步促进抗癌药物的研发

期刊:Current Issues in Molecular Biology

影响因子:3

doi:10.3390/cimb45110580

Kimura, Kotohiko; Jackson, Tiffany L B; Huang, Ru Chih C

Tetra-O-methyl-nordihydroguaiaretic acid inhibits energy metabolism and synergistically induces anticancer effects with temozolomide on LN229 glioblastoma tumors implanted in mice while preventing obesity in normal mice that consume high-fat diets

四-O-甲基-去甲二氢愈创木酸可抑制能量代谢，并与替莫唑胺协同诱导对小鼠植入的 LN229 胶质母细胞瘤肿瘤的抗癌作用，同时防止食用高脂肪饮食的正常小鼠肥胖

期刊:PLoS One

影响因子:2.9

doi:10.1371/journal.pone.0285536

Kotohiko Kimura, Jong Ho Chun, Yu-Ling Lin, Yu-Chuan Liang, Tiffany L B Jackson, Ru Chih C Huang

Tetra-O-Methyl Nordihydroguaiaretic Acid Broadly Suppresses Cancer Metabolism and Synergistically Induces Strong Anticancer Activity in Combination with Etoposide, Rapamycin and UCN-01

四氧甲基去甲二氢愈创木酸广泛抑制癌症代谢并与依托泊苷、雷帕霉素和 UCN-01 联合产生强大的抗癌活性

期刊:PLoS One

影响因子:2.9

doi:10.1371/journal.pone.0148685

Kotohiko Kimura, Ru Chih C Huang

In vivo amelioration of endogenous antitumor autoantibodies via low-dose P4N through the LTA4H/activin A/BAFF pathway

低剂量 P4N 通过 LTA4H/激活素 A/BAFF 通路在体内改善内源性抗肿瘤自身抗体

期刊:Proceedings of the National Academy of Sciences of the United States of America

影响因子:

doi:10.1073/pnas.1604752113

Yu-Ling Lin, Nu-Man Tsai, Cheng-Hao Hsieh, Shu-Yi Ho, Jung Chang, Hsin-Yi Wu, Ming-Hua Hsu, Chia-Ching Chang, Kuang-Wen Liao, Tiffany L B Jackson, David E Mold, Ru Chih C Huang

Evidence to Support the Collaboration of SP1, MYC, and HIF1A and Their Association with microRNAs.

支持 SP1、MYC 和 HIF1A 的协作及其与 microRNA 关联的证据

Interaction and Collaboration of SP1, HIF-1, and MYC in Regulating the Expression of Cancer-Related Genes to Further Enhance Anticancer Drug Development

SP1、HIF-1 和 MYC 的相互作用与协作在调控癌症相关基因表达中的作用，可进一步促进抗癌药物的研发

Tetra-O-methyl-nordihydroguaiaretic acid inhibits energy metabolism and synergistically induces anticancer effects with temozolomide on LN229 glioblastoma tumors implanted in mice while preventing obesity in normal mice that consume high-fat diets

四-O-甲基-去甲二氢愈创木酸可抑制能量代谢，并与替莫唑胺协同诱导对小鼠植入的 LN229 胶质母细胞瘤肿瘤的抗癌作用，同时防止食用高脂肪饮食的正常小鼠肥胖

Tetra-O-Methyl Nordihydroguaiaretic Acid Broadly Suppresses Cancer Metabolism and Synergistically Induces Strong Anticancer Activity in Combination with Etoposide, Rapamycin and UCN-01

四氧甲基去甲二氢愈创木酸广泛抑制癌症代谢并与依托泊苷、雷帕霉素和 UCN-01 联合产生强大的抗癌活性

In vivo amelioration of endogenous antitumor autoantibodies via low-dose P4N through the LTA4H/activin A/BAFF pathway

低剂量 P4N 通过 LTA4H/激活素 A/BAFF 通路在体内改善内源性抗肿瘤自身抗体