日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Human induced pluripotent stem cells for in vitro modeling of impaired mucociliary clearance in cystic fibrosis lung disease.

利用人类诱导多能干细胞进行体外模型研究囊性纤维化肺病中黏液纤毛清除功能障碍。

Klassen Mark-Christian, Balázs Anita, Zöllner Janina, Cleve Nicole, Czichon Laurien, von Schledorn Laura, Hegermann Jan, Nawroth Janna C, Roth Doris, Mielenz Mia, Hedtfeld Silke, Stanke Frauke, Rubil Tihomir, Ius Fabio, Jonigk Danny, Hanrahan John W, Ruhparwar Arjang, Olmer Ruth, Mall Marcus A, Merkert Sylvia, Martin Ulrich

The potentiator ivacaftor is essential for pharmacological restoration of F508del-CFTR function and mucociliary clearance in cystic fibrosis

增强剂ivacaftor对于囊性纤维化中F508del-CFTR功能和粘液纤毛清除的药理学恢复至关重要。

Balázs, Anita; Rubil, Tihomir; Wong, Christine K; Berger, Jasmin; Drescher, Marika; Seidel, Kathrin; Stahl, Mirjam; Graeber, Simon Y; Mall, Marcus A

Clinical Characteristics, Laboratory Parameters, and Molecular Epidemiology of Neuroinvasive Flavivirus Infections in a Hotspot Region of Eastern Croatia

克罗地亚东部热点地区神经侵袭性黄病毒感染的临床特征、实验室参数和分子流行病学

Sabadi, Dario; Bodulić, Kristian; Savić, Vladimir; Vlahović Vlašić, Nika; Bogdanić, Maja; Perić, Ljiljana; Tabain, Irena; Lišnjić, Dubravka; Duvnjak, Mario; Židovec-Lepej, Snježana; Grubišić, Barbara; Rubil, Ilija; Barbić, Ljubo; Švitek, Luka; Stevanović, Vladimir; Smajić, Petra; Berišić, Bernarda; Zlosa, Mihaela; Rončević, Ivana; Vilibić-Čavlek, Tatjana

Personalized CFTR Modulator Therapy for G85E and N1303K Homozygous Patients with Cystic Fibrosis

针对 G85E 和 N1303K 纯合囊性纤维化患者的个性化 CFTR 调节剂治疗

Simon Y Graeber, Anita Balázs, Niklas Ziegahn, Tihomir Rubil, Constanze Vitzthum, Linus Piehler, Marika Drescher, Kathrin Seidel, Alexander Rohrbach, Jobst Röhmel, Stephanie Thee, Julia Duerr, Marcus A Mall, Mirjam Stahl

Age-Related Differences in Structure and Function of Nasal Epithelial Cultures From Healthy Children and Elderly People

健康儿童和老年人鼻黏膜上皮培养物的结构和功能与年龄相关的差异

Anita Balázs, Pamela Millar-Büchner, Michael Mülleder, Vadim Farztdinov, Lukasz Szyrwiel, Annalisa Addante, Aditi Kuppe, Tihomir Rubil, Marika Drescher, Kathrin Seidel, Sebastian Stricker, Roland Eils, Irina Lehmann, Birgit Sawitzki, Jobst Röhmel, Markus Ralser, Marcus A Mall

Anti-PCSK9 antibodies inhibit pro-atherogenic mechanisms in APOE*3Leiden.CETP mice

抗 PCSK9 抗体可抑制 APOE*3Leiden.CETP 小鼠的促动脉粥样硬化机制

Susanne Schuster, Sandra Rubil, Matthias Endres, Hans M G Princen, Jes-Niels Boeckel, Karsten Winter, Christian Werner, Ulrich Laufs

Activation of gene transcription via CIM0216, a synthetic ligand of transient receptor potential melastatin-3 (TRPM3) channels

通过 CIM0216(瞬时受体电位黑素-3 (TRPM3) 通道的合成配体)激活基因转录

Sandra Rubil, Gerald Thiel

CREB, AP-1, ternary complex factors and MAP kinases connect transient receptor potential melastatin-3 (TRPM3) channel stimulation with increased c-Fos expression

CREB、AP-1、三元复合因子和 MAP 激酶将瞬时受体电位黑素-3 (TRPM3) 通道刺激与 c-Fos 表达增加联系起来

Sandra Rubil, Oliver G Rössler, Gerald Thiel

Activation and inhibition of transient receptor potential TRPM3-induced gene transcription

瞬时受体电位 TRPM3 诱导基因转录的激活和抑制

Andrea Lesch, Sandra Rubil, Gerald Thiel