Ruiz i Altaba Ariel相关文献与解析，生知库

Metastases and Colon Cancer Tumor Growth Display Divergent Responses to Modulation of Canonical WNT Signaling

转移瘤和结肠癌肿瘤生长对经典WNT信号通路调控表现出不同的反应

期刊:PLoS One

影响因子:2.6

doi:10.1371/journal.pone.0150697

Seth, Chandan; Ruiz i Altaba, Ariel

Context-dependent signal integration by the GLI code: the oncogenic load, pathways, modifiers and implications for cancer therapy

GLI 编码的上下文依赖性信号整合：致癌负荷、通路、修饰因子及其对癌症治疗的影响

期刊:Seminars in Cell & Developmental Biology

影响因子:6

doi:10.1016/j.semcdb.2014.05.003

Aberger, Fritz; Ruiz i Altaba, Ariel

Loss of WNT-TCF addiction and enhancement of HH-GLI1 signalling define the metastatic transition of human colon carcinomas

WNT-TCF依赖性的丧失和HH-GLI1信号传导的增强定义了人类结肠癌的转移转变。

期刊:EMBO Molecular Medicine

影响因子:8.3

doi:10.1002/emmm.201000098

Varnat, Frédéric; Siegl-Cachedenier, Irene; Malerba, Monica; Gervaz, Pascal; Ruiz i Altaba, Ariel

NANOG regulates glioma stem cells and is essential in vivo acting in a cross-functional network with GLI1 and p53.

NANOG 调控神经胶质瘤干细胞，在体内发挥着至关重要的作用，它与 GLI1 和 p53 形成跨功能网络

期刊:EMBO Journal

影响因子:8.3

doi:10.1038/emboj.2010.137

Zbinden Marie, Duquet Arnaud, Lorente-Trigos Aiala, Ngwabyt Sandra-Nadia, Borges Isabel, Ruiz i Altaba Ariel

Context-dependent regulation of the GLI code in cancer by HEDGEHOG and non-HEDGEHOG signals

HEDGEHOG 和非 HEDGEHOG 信号对癌症中 GLI 编码的上下文依赖性调控

期刊:Journal of Molecular Cell Biology

影响因子:5.9

doi:10.1093/jmcb/mjp052

Stecca, Barbara; Ruiz i Altaba, Ariel

Human colon cancer epithelial cells harbour active HEDGEHOG-GLI signalling that is essential for tumour growth, recurrence, metastasis and stem cell survival and expansion

人类结肠癌上皮细胞中存在活跃的HEDGEHOG-GLI信号通路，该通路对肿瘤生长、复发、转移以及干细胞的存活和扩增至关重要。

期刊:EMBO Molecular Medicine

影响因子:8.3

doi:10.1002/emmm.200900039

Varnat, Frédéric; Duquet, Arnaud; Malerba, Monica; Zbinden, Marie; Mas, Christophe; Gervaz, Pascal; Ruiz i Altaba, Ariel

A GLI1-p53 inhibitory loop controls neural stem cell and tumour cell numbers

GLI1-p53抑制环路控制神经干细胞和肿瘤细胞的数量

期刊:EMBO Journal

影响因子:8.3

doi:10.1038/emboj.2009.16

Stecca, Barbara; Ruiz i Altaba, Ariel

Entity versus property: tracking the nature, genesis and role of stem cells in cancer. Conference on Stem cells and cancer

实体与属性：追踪干细胞在癌症中的性质、起源和作用。干细胞与癌症研讨会

期刊:EMBO Reports

影响因子:6.2

doi:10.1038/embor.2009.169

Ruiz i Altaba, Ariel; Brand, Andrea H

Chimeric NANOG repressors inhibit glioblastoma growth in vivo in a context-dependent manner.

嵌合NANOG抑制因子以依赖于环境的方式在体内抑制胶质母细胞瘤的生长

In vivo epigenetic reprogramming of primary human colon cancer cells enhances metastases

体内表观遗传重编程可增强原发性人结肠癌细胞的转移。

Metastases and Colon Cancer Tumor Growth Display Divergent Responses to Modulation of Canonical WNT Signaling

转移瘤和结肠癌肿瘤生长对经典WNT信号通路调控表现出不同的反应

Context-dependent signal integration by the GLI code: the oncogenic load, pathways, modifiers and implications for cancer therapy

GLI 编码的上下文依赖性信号整合：致癌负荷、通路、修饰因子及其对癌症治疗的影响

Loss of WNT-TCF addiction and enhancement of HH-GLI1 signalling define the metastatic transition of human colon carcinomas

WNT-TCF依赖性的丧失和HH-GLI1信号传导的增强定义了人类结肠癌的转移转变。

NANOG regulates glioma stem cells and is essential in vivo acting in a cross-functional network with GLI1 and p53.

NANOG 调控神经胶质瘤干细胞，在体内发挥着至关重要的作用，它与 GLI1 和 p53 形成跨功能网络

Context-dependent regulation of the GLI code in cancer by HEDGEHOG and non-HEDGEHOG signals

HEDGEHOG 和非 HEDGEHOG 信号对癌症中 GLI 编码的上下文依赖性调控

Human colon cancer epithelial cells harbour active HEDGEHOG-GLI signalling that is essential for tumour growth, recurrence, metastasis and stem cell survival and expansion

人类结肠癌上皮细胞中存在活跃的HEDGEHOG-GLI信号通路，该通路对肿瘤生长、复发、转移以及干细胞的存活和扩增至关重要。

A GLI1-p53 inhibitory loop controls neural stem cell and tumour cell numbers

GLI1-p53抑制环路控制神经干细胞和肿瘤细胞的数量

Entity versus property: tracking the nature, genesis and role of stem cells in cancer. Conference on Stem cells and cancer

实体与属性：追踪干细胞在癌症中的性质、起源和作用。干细胞与癌症研讨会