Glutaredoxin 2a overexpression in macrophages promotes mitochondrial dysfunction but has little or no effect on atherogenesis in LDL-receptor null mice
巨噬细胞中谷氧还蛋白 2a 的过表达会促进线粒体功能障碍，但对 LDL 受体缺陷小鼠的动脉粥样硬化形成几乎没有影响
期刊:Atherosclerosis
影响因子:4.9
doi:10.1016/j.atherosclerosis.2015.04.805
D A Zamora, K P Downs, S L Ullevig, S Tavakoli, H S Kim, M Qiao, D R Greaves, R Asmis
