Sallin Michelle A相关文献与解析，生知库

Erratum for Hoft et al., "The Rate of CD4 T Cell Entry into the Lungs during Mycobacterium tuberculosis Infection Is Determined by Partial and Opposing Effects of Multiple Chemokine Receptors"

更正：Hoft 等人，“结核分枝杆菌感染期间 CD4 T 细胞进入肺部的速率由多种趋化因子受体的部分和相反作用决定”

期刊:Infection and Immunity

影响因子:2.8

doi:10.1128/IAI.00491-19

Hoft, Stella G; Sallin, Michelle A; Kauffman, Keith D; Sakai, Shunsuke; Ganusov, Vitaly V; Barber, Daniel L

The Rate of CD4 T Cell Entry into the Lungs during Mycobacterium tuberculosis Infection Is Determined by Partial and Opposing Effects of Multiple Chemokine Receptors

结核分枝杆菌感染期间CD4 T细胞进入肺部的速率取决于多种趋化因子受体的部分和相反作用。

期刊:Infection and Immunity

影响因子:2.8

doi:10.1128/IAI.00841-18

Hoft, Stella G; Sallin, Michelle A; Kauffman, Keith D; Sakai, Shunsuke; Ganusov, Vitaly V; Barber, Daniel L

Th1 Differentiation Drives the Accumulation of Intravascular, Non-protective CD4 T Cells during Tuberculosis

Th1分化驱动结核病期间血管内非保护性CD4 T细胞的积累

期刊:Cell Reports

影响因子:6.9

doi:10.1016/j.celrep.2017.03.007

Sallin, Michelle A; Sakai, Shunsuke; Kauffman, Keith D; Young, Howard A; Zhu, Jinfang; Barber, Daniel L

CD4 T Cell-Derived IFN-Î³ Plays a Minimal Role in Control of Pulmonary Mycobacterium tuberculosis Infection and Must Be Actively Repressed by PD-1 to Prevent Lethal Disease.

CD4 T 细胞衍生的 IFN-γ 在控制肺结核分枝杆菌感染中起的作用微乎其微，必须通过 PD-1 主动抑制才能防止致命疾病

期刊:PLoS Pathogens

影响因子:4.9

doi:10.1371/journal.ppat.1005667

Sakai Shunsuke, Kauffman Keith D, Sallin Michelle A, Sharpe Arlene H, Young Howard A, Ganusov Vitaly V, Barber Daniel L

Immune system development and age-dependent maintenance in Klotho-hypomorphic mice.

Klotho低表达小鼠的免疫系统发育和年龄依赖性维持。

Erratum for Hoft et al., "The Rate of CD4 T Cell Entry into the Lungs during Mycobacterium tuberculosis Infection Is Determined by Partial and Opposing Effects of Multiple Chemokine Receptors"

更正：Hoft 等人，“结核分枝杆菌感染期间 CD4 T 细胞进入肺部的速率由多种趋化因子受体的部分和相反作用决定”

The Rate of CD4 T Cell Entry into the Lungs during Mycobacterium tuberculosis Infection Is Determined by Partial and Opposing Effects of Multiple Chemokine Receptors

结核分枝杆菌感染期间CD4 T细胞进入肺部的速率取决于多种趋化因子受体的部分和相反作用。

Th1 Differentiation Drives the Accumulation of Intravascular, Non-protective CD4 T Cells during Tuberculosis

Th1分化驱动结核病期间血管内非保护性CD4 T细胞的积累

CD4 T Cell-Derived IFN-Î³ Plays a Minimal Role in Control of Pulmonary Mycobacterium tuberculosis Infection and Must Be Actively Repressed by PD-1 to Prevent Lethal Disease.

CD4 T 细胞衍生的 IFN-γ 在控制肺结核分枝杆菌感染中起的作用微乎其微，必须通过 PD-1 主动抑制才能防止致命疾病