日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Stat5 induces androgen receptor (AR) gene transcription in prostate cancer and offers a druggable pathway to target AR signaling

Stat5 诱导前列腺癌中的雄激素受体 (AR) 基因转录,并提供一种可用于靶向 AR 信号的药物通路

Cristina Maranto, Lavannya Sabharwal, Vindhya Udhane, Samuel P Pitzen, Braedan McCluskey, Songyan Qi, Christine O'Connor, Savita Devi, Scott Johnson, Kenneth Jacobsohn, Anjishnu Banerjee, Kenneth A Iczkowski, Liang Wang, Scott M Dehm, Marja T Nevalainen

CRISPR-Cas9 cytidine and adenosine base editing of splice-sites mediates highly-efficient disruption of proteins in primary and immortalized cells

CRISPR-Cas9 对剪接位点的胞嘧啶和腺苷碱基编辑可高效破坏原代细胞和永生化细胞中的蛋白质。

Mitchell G Kluesner # ,Walker S Lahr # ,Cara-Lin Lonetree ,Branden A Smeester ,Xiaohong Qiu ,Nicholas J Slipek ,Patricia N Claudio Vázquez ,Samuel P Pitzen ,Emily J Pomeroy ,Madison J Vignes ,Samantha C Lee ,Samuel P Bingea ,Aneesha A Andrew ,Beau R Webber ,Branden S Moriarity

Opposing transcriptional programs of KLF5 and AR emerge during therapy for advanced prostate cancer

晚期前列腺癌治疗期间出现 KLF5 和 AR 的拮抗转录程序

Meixia Che #, Aashi Chaturvedi #, Sarah A Munro #, Samuel P Pitzen, Alex Ling, Weijie Zhang, Josh Mentzer, Sheng-Yu Ku, Loredana Puca, Yanyun Zhu, Andries M Bergman, Tesa M Severson, Colleen Forster, Yuzhen Liu, Jacob Hildebrand, Mark Daniel, Ting-You Wang, Luke A Selth, Theresa Hickey, Amina Zoubei