日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Evolution of multiple myeloma from a genomic perspective

从基因组学角度看多发性骨髓瘤的演变

Maura, Francesco; Samur, Mehmet; Munshi, Nikhil

Longitudinal Profiling of Tumor and Immune Compartments Uncovers Patterns of Dysregulation and Associations with Response in Multiple Myeloma

肿瘤和免疫区室的纵向分析揭示了多发性骨髓瘤中失调模式及其与治疗反应的关联

Ohlstrom, Denis J; Pilcher, William C Jr; Michaud, Marina E; Acharya, Chaitanya; Satpathy, Sarthak; Gonzalez-Kozlova, Edgar; Jayasinghe, Reyka G; Ferguson, Katherine; Mumme, Hope L; Nanda, Shivani; Song, Yizhe; Mantrala, Sowmitri; Karagkouni, Dimitra; Schulman, Jessica; Pabustan, Nick; Vieira Dos Santos, Junia; Sherbenou, Daniel W; Keats, Jonathan J; Gout, Alexander M; Foltz, Steven; Lagana, Alessandro; Kourelis, Taxiarchis; Vij, Ravi; Dhodapkar, Madhav V; Avigan, David; Cho, Hearn Jay; Baughn, Linda B; Nooka, Ajay K; Lonial, Sagar; Kumar, Shaji; Samur, Mehmet K; Vlachos, Ioannis S; Ding, Li; Gnjatic, Sacha; Mulligan, George; Bhasin, Manoj K

ID2 Suppresses Multiple Myeloma Cell Proliferation by Repressing the Activity of the Transcription Factor TCF3.

ID2 通过抑制转录因子 TCF3 的活性来抑制多发性骨髓瘤细胞增殖。

Fulciniti Mariateresa, Yao Yao, Perini Tommaso, Fong Ng Jessica, Schavgoulidze Anaïs, Deng Shuhui, Cui Jian, Encinas Mayoral Jessica, Ladisa Francesco, Young Ryan M, Epstein Charles B, White Cassandra M, Ott Christopher J, Gulla Annamaria, Matulis Shannon M, Sperling Adam S, Morelli Eugenio, Boise Lawrence H, Binder Moritz, Szalat Raphael, Samur Mehmet K, Anderson Kenneth C, Munshi Nikhil C

CRISPR-Cas13d functional transcriptomics reveals widespread isoform-selective cancer dependencies on lncRNAs

CRISPR-Cas13d功能转录组学揭示了癌症对lncRNA的广泛异构体选择性依赖性

Morelli, Eugenio; Aktas-Samur, Anil; Maisano, Domenico; Gao, Claire; Favasuli, Vanessa; Papaioannou, Dimitrios; De Nola, Giovanni; Henninger, Jon E; Liu, Na; Turi, Marcello; Folino, Pietro; Vreux, Laure; Cumerlato, Michela; Chen, Liang; Aifantis, Iannis; Fulciniti, Mariateresa; Anderson, Kenneth C; Lytton-Jean, Abigail K R; Gullà, Annamaria; Young, Richard A; Samur, Mehmet K; Munshi, Nikhil C

The mitochondrial protease ClpP is a metabolic vulnerability and an immunogenic trigger against multiple myeloma

线粒体蛋白酶ClpP是代谢脆弱点,也是多发性骨髓瘤的免疫原性触发因素。

Perini, Tommaso; Zordan, Paola; Del Pizzo, Rossella; Resnati, Massimo; Viviani, Lisa; Stefanoni, Davide; Cassina, Laura; Orfanelli, Ugo; Trudu, Matteo; Oliva, Laura; Lacidogna, Daniel; Samur, Mehmet K; Materozzi, Maria; Drago, Denise; Andolfo, Annapaola; Patrone, Marco; Degano, Massimo; Boletta, Alessandra; Milan, Enrico; Ciceri, Fabio; Munshi, Nikhil C; Bellone, Matteo; Cenci, Simone

Development of hyperdiploidy starts at an early age and takes a decade to complete

超二倍体发育始于幼年时期,需要十年时间才能完成。

Samur, Mehmet K; Aktas Samur, Anil; Shah, Parth; Park, Joseph S; Fulciniti, Mariateresa; Shammas, Masood; Corre, Jill; Anderson, Kenneth C; Parmigiani, Giovanni; Avet-Loiseau, Hervé; Munshi, Nikhil C

Deciphering neutrophil dynamics in the focal lesion tumor microenvironment to overcome immunosuppression in multiple myeloma

解析局灶性病变肿瘤微环境中中性粒细胞的动态变化,以克服多发性骨髓瘤的免疫抑制

Rivera, Joshua; Yan, Qi; Daneshmandi, Saeed; Lannes, Romain; Katsuta, Eriko; Choi, JeeEun; Singh, Prashant; Belal, Ahmed; Alberico, Ronald; Lund, Ian; Schaefer, Megan; Hassan, Hamza; Parker, Sarah; Anderson, Kenneth C; Munshi, Nikhil C; Samur, Mehmet; McCarthy, Philip L; Hillengass, Jens; Mohammadpour, Hemn

Loss of BCL7A permits IRF4 transcriptional activity and cellular growth in multiple myeloma.

BCL7A 的缺失使得 IRF4 转录活性增强,并促进多发性骨髓瘤细胞的生长。

Chakraborty Chandraditya, Talluri Srikanth, Binder Moritz, Morelli Eugenio, Mayoral Jessica Encinas, Derebail Sanika, Samur Anil Aktas, Epstein Charles, Anderson Kenneth C, Shammas Masood, Samur Mehmet K, Fulciniti Mariateresa, Munshi Nikhil C

Immune profiling of smoldering multiple myeloma patients treated in a phase lb study of PVX-410 vaccine targeting XBP1/CD138/CS1 antigens, and citarinostat, a histone deacetylase inhibitor (HDACi) with and without lenalidomide

本研究对接受PVX-410疫苗(靶向XBP1/CD138/CS1抗原)和西他诺司他(一种组蛋白去乙酰化酶抑制剂,HDACi)联合或不联合来那度胺治疗的冒烟型多发性骨髓瘤患者进行了Ib期临床试验,并分析了接受西他诺司他治疗的冒烟型多发性骨髓瘤患者的免疫特征。

Cirstea, Diana; Shome, Rajib; Samur, Mehmet; Talluri, Srikanth; Connolly, Joseph J; Wright, Alexandra Jean; Duvallet, Emilie; Joyce, Amanda N R; Lively, Kathleen; Basinsky, Gina; Yee, Andrew J; Chase, Cristiana Costa; Malek, Ehsan; Niesvizky, Ruben; Richardson, Paul G; Raje, Noopur S

Targeting Acetyl-CoA Carboxylase Suppresses De Novo Lipogenesis and Tumor Cell Growth in Multiple Myeloma.

靶向乙酰辅酶A羧化酶可抑制多发性骨髓瘤中的从头脂肪生成和肿瘤细胞生长

Morelli Eugenio, Ribeiro Caroline Fidalgo, Rodrigues Silvia D, Gao Claire, Socciarelli Fabio, Maisano Domenico, Favasuli Vanessa, Liu Na, Todoerti Katia, Chakraborty Chandraditya, Yao Yao, Fulciniti Mariateresa, Samur Mehmet, Aktas-Samur Anil, Amodio Nicola, Turi Marcello, Barello Francesca, Penailillo Johany, Giallongo Cesarina, Romano Alessandra, Gulla Annamaria, Anderson Kenneth C, Inghirami Giorgio, Munshi Nikhil C, Loda Massimo