hUCMSC mitochondrial EVs confer neuroprotection after ischemia by Tom1l2-mediated mitochondrial fusion and Crls1-cardiolipin axis reprogramming.

hUCMSC 线粒体 EVs 通过 Tom1l2 介导的线粒体融合和 Crls1-心磷脂轴重编程，在缺血后发挥神经保护作用。