日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

An imbalance between CD80 and CD86 levels and CD4 regulatory T cell number and transendocytosis function exists in the liver in autoimmune hepatitis

自身免疫性肝炎患者肝脏中存在CD80和CD86水平以及CD4调节性T细胞数量和跨内吞功能之间的失衡。

Halliday, Neil; Kennedy, Alan; Williams, Cayman; Soskic, Blagoje; Hinze, Claudia; Pinzani, Massimo; Thorburn, Douglas; Sansom, David M

Immune dysregulation from a novel CTLA-4 haploinsufficiency variant.

由新型 CTLA-4 单倍体不足变异引起的免疫失调。

Brodsky Nina N, Kennedy Alan, Glaser Daniel, Jeffries Lauren, Ji Weizhen, Natarajan Eesha, Shin Junghee J, Sansom David M, Lucas Carrie L, Lakhani Saquib A

CD28 and TCR differentially impact naïve and memory T cell responses

CD28 和 TCR 对初始 T 细胞和记忆 T 细胞的反应具有不同的影响。

Williams, Cayman; Giovacchini, Dalisay; Kennedy, Alan; Halliday, Neil; Waters, Erin; Robinson, Maximillian; Hinze, Claudia; Sansom, David M

Noninvasive Stratification of Colon Cancer by Multiplex PET Imaging

通过多重 PET 成像对结肠癌进行无创分层

Gaurav Malviya, Tamsin R M Lannagan, Emma Johnson, Agata Mackintosh, Robert Bielik, Adam Peters, Dmitry Soloviev, Gavin Brown, Rene Jackstadt, Colin Nixon, Kathryn Gilroy, Andrew Campbell, Owen J Sansom, David Y Lewis

Rigid, bivalent CTLA-4 binding to CD80 is required to disrupt the cis CD80/PD-L1 interaction.

CTLA-4 与 CD80 的刚性二价结合是破坏顺式 CD80/PD-L1 相互作用所必需的。

Robinson Maximillian A, Kennedy Alan, Orozco Carolina T, Chen Hung-Chang, Waters Erin, Giovacchini Dalisay, Yeung Kay, Filer Lily, Hinze Claudia, Lloyd Christopher, Dovedi Simon J, Sansom David M

Lack of phosphatidylinositol 3-kinase VPS34 in regulatory T cells leads to a fatal lymphoproliferative disorder without affecting their development.

调节性 T 细胞中缺乏磷脂酰肌醇 3-激酶 VPS34 会导致致命的淋巴增生性疾病,但并不影响其发育

Courreges Christina J F, Davenport Elizabeth C M, Bilanges Benoit, Rebollo-Gomez Elena, Hukelmann Jens, Schoenfelder Priya, Edgar James R, Sansom David, Scudamore Cheryl L, Roychoudhuri Rahul, Garden Oliver A, Vanhaesebroeck Bart, Okkenhaug Klaus

Both intratumoral regulatory T cell depletion and CTLA-4 antagonism are required for maximum efficacy of anti-CTLA-4 antibodies

肿瘤内调节性T细胞耗竭和CTLA-4拮抗作用对于抗CTLA-4抗体的最大疗效都是必需的。

Lax, Brianna M; Palmeri, Joseph R; Lutz, Emi A; Sheen, Allison; Stinson, Jordan A; Duhamel, Lauren; Santollani, Luciano; Kennedy, Alan; Rothschilds, Adrienne M; Spranger, Stefani; Sansom, David M; Wittrup, K Dane

An IL-2 mutein increases IL-10 and CTLA-4-dependent suppression of dendritic cells by regulatory T cells

IL-2突变体增强了调节性T细胞对树突状细胞的IL-10和CTLA-4依赖性抑制作用

Jamison, Braxton L; Lawrance, Matthew; Wang, Chun Jing; DeBerg, Hannah A; Sansom, David M; Gavin, Marc A; Walker, Lucy S K; Campbell, Daniel J

In Vitro Analysis of CTLA-4-Mediated Transendocytosis by Regulatory T Cells

CTLA-4介导的调节性T细胞跨内吞作用的体外分析

Waters, Erin; Williams, Cayman; Kennedy, Alan; Sansom, David M

RAL GTPases mediate EGFR-driven intestinal stem cell proliferation and tumourigenesis

RAL GTP酶介导EGFR驱动的肠道干细胞增殖和肿瘤发生

Máté Nászai ,Karen Bellec ,Yachuan Yu ,Alvaro Román-Fernández ,Emma Sandilands ,Joel Johansson ,Andrew D Campbell ,Jim C Norman ,Owen J Sansom ,David M Bryant ,Julia B Cordero