日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Mir-155 is overexpressed in systemic sclerosis fibroblasts and is required for NLRP3 inflammasome-mediated collagen synthesis during fibrosis

Mir-155在系统性硬化症成纤维细胞中过度表达,并且是纤维化过程中NLRP3炎症小体介导的胶原合成所必需的。

Artlett, Carol M; Sassi-Gaha, Sihem; Hope, Jennifer L; Feghali-Bostwick, Carol A; Katsikis, Peter D

The Tsk2/+ mouse fibrotic phenotype is due to a gain-of-function mutation in the PIIINP segment of the Col3a1 gene

Tsk2/+ 小鼠纤维化表型是由于 Col3a1 基因的 PIIINP 片段发生功能获得突变所致

Kristen B Long, Zhenghui Li, Chelsea M Burgwin, Susanna G Choe, Viktor Martyanov, Sihem Sassi-Gaha, Josh P Earl, Rory A Eutsey, Azad Ahmed, Garth D Ehrlich, Carol M Artlett, Michael L Whitfield, Elizabeth P Blankenhorn

Chimeric cells of maternal origin do not appear to be pathogenic in the juvenile idiopathic inflammatory myopathies or muscular dystrophy

母源嵌合细胞似乎不会引起青少年特发性炎症性肌病或肌营养不良症。

Artlett, Carol M; Sassi-Gaha, Sihem; Ramos, Ronald C; Miller, Frederick W; Rider, Lisa G

Targeting the unfolded protein response, XBP1, and the NLRP3 inflammasome in fibrosis and cancer

靶向未折叠蛋白反应、XBP1 和 NLRP3 炎症小体在纤维化和癌症中的作用

Overley-Adamson, Beth; Artlett, Carol M; Stephens, Connie; Sassi-Gaha, Sihem; Weis, Ransome D; Thacker, James D

NLRP3 inflammasome is a target for development of broad-spectrum anti-infective drugs

NLRP3炎症小体是开发广谱抗感染药物的靶点

Thacker, James D; Balin, Brian J; Appelt, Denah M; Sassi-Gaha, Sihem; Purohit, Mitali; Rest, Richard F; Artlett, Carol M