Improvements in impaired GABA and GAD65/67 production in the spinal dorsal horn contribute to exercise-induced hypoalgesia in a mouse model of neuropathic pain

脊髓背角中受损的 GABA 和 GAD65/67 生成得到改善，导致神经性疼痛小鼠模型中出现运动诱发的痛觉减退