Targeting p66Shc to restore K(ATP) channel and renal microvascular responses in a preclinical model of diabetic nephropathy
在糖尿病肾病临床前模型中，靶向 p66Shc 可恢复 K(ATP) 通道和肾脏微血管反应。
期刊:Frontiers in Physiology
影响因子:3.4
doi:10.3389/fphys.2025.1620591
Miller, Bradley; Imig, John; Goorani, Samaneh; Schupbach, Perrin; Hays, Franklin; Benbrook, Doris M; Sorokin, Andrey
