日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

A154 PROPERDIN DEFICIENCY DOES NOT IMPACT THE MOUSE RESPONSE TO DSS-INDUCED COLITIS DESPITE DIFFERENCES IN COLONIC MICROBIOME

尽管结肠微生物群存在差异,但A154 PROPERDIN缺陷并不影响小鼠对DSS诱导的结肠炎的反应。

Viau, Charles; Nouar, Alyssa; Xia, Jianguo; Stadnyk, A W; Douglas, G; Comeau, A; Jain, U; Schwaeble, W; Stover, C; Bieko, R; Langille, M

Properdin deficiency protects from 5-fluorouracil-induced small intestinal mucositis in a complement activation-independent, interleukin-10-dependent mechanism

备解素缺乏可通过一种不依赖于补体激活、依赖于白细胞介素-10的机制,保护机体免受5-氟尿嘧啶诱导的小肠黏膜炎的侵害。

Jain, U; Midgen, C A; Woodruff, T M; Schwaeble, W J; Stover, C M; Stadnyk, A W

Further studies of the down-regulation by Factor I of the C3b feedback cycle using endotoxin as a soluble activator and red cells as a source of CR1 on sera of different complotype.

进一步研究因子 I 对 C3b 反馈循环的下调作用,以内毒素为可溶性激活剂,以红细胞为 CR1 的来源,研究不同复合型血清中的 CR1 的下调作用

Lachmann P J, Lay E, Seilly D J, Buchberger A, Schwaeble W, Khadake J

Complotype affects the extent of down-regulation by Factor I of the C3b feedback cycle in vitro.

复合型影响体外 C3b 反馈循环中因子 I 的下调程度

Lay E, Nutland S, Smith J E, Hiles I, Smith R A G, Seilly D J, Buchberger A, Schwaeble W, Lachmann P J

Role of the classical pathway of complement activation in experimentally induced polymicrobial peritonitis

补体激活经典途径在实验诱导的多微生物腹膜炎中的作用

Celik, I; Stover, C; Botto, M; Thiel, S; Tzima, S; Künkel, D; Walport, M; Lorenz, W; Schwaeble, W

Expression of intercellular adhesion molecule 1 (ICAM-1, CD54) in colonic epithelial cells

结肠上皮细胞中细胞间黏附分子1(ICAM-1,CD54)的表达

Dippold, W; Wittig, B; Schwaeble, W; Mayet, W; Meyer zum Büschenfelde, K H