日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

The case for targeting latent and lytic Epstein-Barr virus infection in multiple sclerosis

针对多发性硬化症中潜伏性和裂解性 Epstein-Barr 病毒感染的治疗策略

Giovannoni, Gavin; James, Louisa; Adeniran, Adekunle A; Gold, Julian; Young, Lawrence S; Selwood, David L; Baker, David; Dobson, Ruth

Quinoline-Based Neuropilin‑1 Antagonists Exhibit a Pure Antagonist Profile and Block Vascular Endothelial Growth Factor-Induced Pain

基于喹啉的神经纤毛蛋白-1拮抗剂表现出纯拮抗剂特性,并能阻断血管内皮生长因子诱导的疼痛。

Hestehave, Sara; Dragoni, Silvia; Fallon, Philip; Mota, Filipa; Calderon-Rivera, Aida; Gomez, Kimberly; Powell, Jonathan; Patsiarika, Anastasia; Reisinger, Tifelle; Crosby, Stuart; Chan, A W Edith; Steadman, David; Winfield, Natalie; Jarvis, Ashley; Martin, John; Zachary, Ian C; Frankel, Paul; Djordjevic, Snezana; Ruhrberg, Christiana; Khanna, Rajesh; Selwood, David L

Delocalized quinolinium-macrocyclic peptides, an atypical chemotype for CNS penetration

离域喹啉大环肽,一种非典型的中枢神经系统渗透化学类型

Pingitore, Valeria; Pancholi, Jessica; Hornsby, Thomas W; Warne, Justin; Pryce, Gareth; McCormick, Laura J; Hill, Julia; Bhosale, Gauri; Peng, Jing; Newton, Lydia S; Towers, Greg J; Coles, Simon J; Chan, Ah Wing Edith; Duchen, Michael R; Szabadkai, Gyorgy; Baker, David; Selwood, David L

Neuroprotection in an Experimental Model of Multiple Sclerosis via Opening of Big Conductance, Calcium-Activated Potassium Channels

通过开放大电导钙激活钾通道在多发性硬化症实验模型中发挥神经保护作用

Pryce, Gareth; Sisay, Sofia; Giovannoni, Gavin; Selwood, David L; Baker, David

Reversal of behavioural phenotype by the cannabinoid-like compound VSN16R in fragile X syndrome mice

类大麻素化合物VSN16R逆转脆性X综合征小鼠的行为表型

Hurley, Michael J; Deacon, Robert M J; Chan, A W Edith; Baker, David; Selwood, David L; Cogram, Patricia

Oligodendrocytes, BK channels and the preservation of myelin

少突胶质细胞、BK通道和髓鞘的保存

Rupnik, Maddalena; Baker, David; Selwood, David L

Emerging small-molecule treatments for multiple sclerosis: focus on B cells

治疗多发性硬化症的新兴小分子疗法:聚焦B细胞

Gregson, Aaron; Thompson, Kaitlyn; Tsirka, Stella E; Selwood, David L

N(10) -carbonyl-substituted phenothiazines inhibiting lipid peroxidation and associated nitric oxide consumption powerfully protect brain tissue against oxidative stress

N(10)-羰基取代的吩噻嗪类化合物能够抑制脂质过氧化及其相关的氧化氮消耗,从而有效保护脑组织免受氧化应激的损害。

Keynes, Robert G; Karchevskaya, Anastasia; Riddall, Dieter; Griffiths, Charmaine H; Bellamy, Tomas C; Chan, A W Edith; Selwood, David L; Garthwaite, John

Critical Care Network in the State of Qatar

卡塔尔国重症监护网络

Hijjeh, Majed; Al Shaikh, Loua; Alinier, Guillaume; Selwood, David; Malmstrom, Faisal; Hassan, Ibrahim Fawzy

Nonhuman TRIM5 Variants Enhance Recognition of HIV-1-Infected Cells by CD8+ T Cells

非人源TRIM5变体增强CD8+ T细胞对HIV-1感染细胞的识别

Jimenez-Moyano, Esther; Ruiz, Alba; Kløverpris, Henrik N; Rodriguez-Plata, Maria T; Peña, Ruth; Blondeau, Caroline; Selwood, David L; Izquierdo-Useros, Nuria; Moris, Arnaud; Clotet, Bonaventura; Goulder, Philip; Towers, Greg J; Prado, Julia G