Exosomes derived from BMSCs ameliorate cyclophosphamide-induced testosterone deficiency by enhancing the autophagy of Leydig cells via the AMPK-mTOR signaling pathway

源自 BMSCs 的外泌体通过 AMPK-mTOR 信号通路增强 Leydig 细胞的自噬，从而改善环磷酰胺诱导的睾酮缺乏症