日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Loss of MLKL impairs abdominal aortic aneurysm development by attenuating smooth muscle cell necroptosis.

MLKL 的缺失会减弱平滑肌细胞坏死,从而损害腹主动脉瘤的发展。

Nemade Harshal, Mehrkens Dennis, Lottermoser Hannah Sophia, Yilmaz Zeynep Ece, Mader Julian, Schelemei Patrik, Picard Felix Ruben, Geißen Simon, Schwab Gülsah Fülgen, Hoyer Friedrich Felix, Guthoff Henning, Hof Alexander, Nettersheim Felix Sebastian, Sachinidis Agapios, Gerdes Norbert, Sengle Gerhard, Winkels Holger, Baldus Stefan, Pasparakis Manolis, Mollenhauer Martin, Adam Matti

Regulation of Vascular Tone of Preglomerular Renal Vasculature by Caldesmon

钙调蛋白对肾小球前血管张力的调节

Pryymachuk, Galyna; Velyanov, Viktor V; Lazarov, Nikola R; Nestele, Simon; Schroeter, Mechthild M; Jelinek, Sarah; Piekarek, Nadin; Bust, Maria; Evtimov, Nikolai T; Marinov, Simeon P; Enzmann, Thomas; Borgmann, Hendrik; Sengle, Gerhard; Hescheler, Jürgen; Korotkova, Tatiana; Gagov, Hristo; Todorov, Vladimir; Bratoeva, Kamelya; Tonchev, Anton B; Pfitzer, Gabriele; Grisk, Olaf; Lubomirov, Lubomir T

Correction: Weimer et al. Localisation-Dependent Variations in Articular Cartilage ECM: Implications for Tissue Engineering and Cartilage Repair. Int. J. Mol. Sci. 2025, 26, 9331

更正:Weimer 等人,《关节软骨细胞外基质的定位依赖性变化:对组织工程和软骨修复的意义》,《国际分子科学杂志》,2025 年,26,9331。

Weimer, Laura; Schmidt, Luisa M; Sengle, Gerhard; Krüger, Marcus; Smith, Alan M; Brändlin, Ilona; Zaucke, Frank

Hyaluronan synthase 3 deficiency lowers the incidence of ruptures of abdominal aortic aneurysms by reducing monocyte infiltration

透明质酸合成酶3缺乏症通过减少单核细胞浸润降低腹主动脉瘤破裂的发生率。

Viola Niemann #,Fedor Brack #,Luca Rolauer,Janet Kaczur,Patrick Petzsch,Karl Köhrer,Christine Quast,Norbert Gerdes,Pascal Bouvain,Katharina Voigt,Martina Krüger,Alexander Brückner,Bernd K Fleischmann,Daniela Wenzel,Philipp Barnowski ,Laura-Marie Zimmermann ,Sakine Simsekyilmaz,Timm Filler,Wiebke Ibing,Tobias Feige,Kim J Krott,Markus U Wagenhäuser,Jens W Fischer,Margitta Elvers,Gerhard Sengle ,Ulrich Flögel,Christian Hundhausen,Tatsiana Suvorava,Maria Grandoch

CLEC3A-Derived Antimicrobial Peptides Reduce Staphylococcus aureus Bacterial Counts in an In Vivo Biomaterial-Associated Infection Mouse Model.

CLEC3A衍生的抗菌肽可降低体内生物材料相关感染小鼠模型中的金黄色葡萄球菌数量

Meinberger Denise, Hermes Gabriele, Brachvogel Bent, Sengle Gerhard, Elezagic Dzemal, Roth Annika, Ruthard Johannes, Streichert Thomas, Klatt Andreas R

Localisation-Dependent Variations in Articular Cartilage ECM: Implications for Tissue Engineering and Cartilage Repair

关节软骨细胞外基质的局部依赖性差异:对组织工程和软骨修复的启示

Weimer, Laura; Schmidt, Luisa M; Sengle, Gerhard; Krüger, Marcus; Smith, Alan M; Brändlin, Ilona; Zaucke, Frank

Vitamin D-inducible antimicrobial peptide LL-37 binds SARS-CoV-2 Spike and accessory proteins ORF7a and ORF8

维生素D诱导型抗菌肽LL-37可与SARS-CoV-2刺突蛋白及其辅助蛋白ORF7a和ORF8结合。

Roth, Annika; Lütke, Steffen; Mörgelin, Matthias; Meinberger, Denise; Hermes, Gabriele; Sengle, Gerhard; Koch, Manuel; Drexelius, Marco; Gebauer, Jan; Neundorf, Ines; Elezagic, Dzemal; Paulsson, Mats; Streichert, Thomas; Klatt, Andreas R

An improved porcine model of infrarenal abdominal aortic aneurysm.

改良的猪肾下腹主动脉瘤模型。

Stei Marta, Arkenberg Per, Uebing Theresa, Mazrekaj Agnesa, Mulorz Joscha, Mehrkens Dennis, Ahdab Maysam, Barnowski Philipp, Sengle Gerhard, Adam Matti, Lohner Valerie, Hoerr Verena, Nahardani Ali, Schubert-Quecke Claudia, Mewes Birgitta, Lindemeyer Johannes, Grüll Holger, Busch Albert, Zimmer Sebastian, Winkels Holger, Schelzig Hubert, Kelm Malte, Nickenig Georg, Baldus Stephan, Wagenhäuser Markus U, Mollenhauer Martin

Exploring thoracic aorta ECM alterations in Marfan syndrome: insights into aorta wall structure.

探索马凡综合征中胸主动脉 ECM 的改变:对主动脉壁结构的深入了解

de Souza Rodrigo Barbosa, Cassiano Luara Lucena, Barnowski Philipp, Ventura Sara, Turato Walter Miguel, Nascimento Suelen Cristina Russafa, Mignanelli Giovanna Lodi, Caldeira Waldir, da Fonseca Martins Ana Maria Cristina Rebelo Pinto, de Carvalho Luposeli Flavio, Laurindo Francisco Rafael Martins, Reinhardt Dieter P, Sengle Gerhard, Koh Ivan Hong Jun, Meek Keith M, Lewis Philip N

ADAMTS12 promotes fibrosis by restructuring extracellular matrix to enable activation of injury-responsive fibroblasts

ADAMTS12通过重塑细胞外基质促进纤维化,从而激活损伤反应性成纤维细胞。

Konrad Hoeft,Lars Koch,Susanne Ziegler,Ling Zhang,Steffen Luetke,Maria C Tanzer,Debashish Mohanta,David Schumacher,Felix Schreibing  ,Qingqing Long,Hyojin Kim,Barbara M Klinkhammer,Carla Schikarski,Sidrah Maryam,Mathijs Baens,Juliane Hermann,Sarah Krieg,Fabian Peisker,Laura De Laporte    ,Gideon Jl Schaefer,Sylvia Menzel,Joachim Jankowski,Benjamin D Humphreys,Adam Wahida,Rebekka K Schneider  0 ,Matthias Versele,Peter Boor  ,Matthias Mann,Gerhard Sengle,Sikander Hayat,Rafael Kramann