日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

F2,6BP restores mitochondrial genome integrity in Huntington's disease.

F2,6BP 可恢复亨廷顿病患者的线粒体基因组完整性。

Chakraborty Anirban, Mandal Santi M, Mankevich Mikita, Mani Rajam S, Shahabi Shandy, Kodavati Manohar, Sreenivasmurthy Sravan Gopalkrishnashetty, Tapryal Nisha, Hamilton Dale J, Krishnan Balaji, Hegde Muralidhar L, Weinfeld Michael, Ghosh Gourisankar, Hazra Tapas

Chromogranin A deficiency attenuates tauopathy by altering epinephrine-alpha-adrenergic receptor signaling in PS19 mice.

嗜铬粒蛋白 A 缺乏通过改变 PS19 小鼠的肾上腺素-α-肾上腺素能受体信号传导来减轻 tau 蛋白病

Jati Suborno, Munoz-Mayorga Daniel, Shahabi Shandy, Tang Kechun, Tao Yuren, Dickson Dennis W, Litvan Irene, Ghosh Gourisankar, Mahata Sushil K, Chen Xu

Teamwork of clustered low-affinity κB sites and accessory factors regulates transcriptional strength of NF-κB RelA dimers.

聚集的低亲和力 αB 位点和辅助因子的协同作用调节 NF-αB RelA 二聚体的转录强度。

Shahabi Shandy, Biswas Tapan, Shen Yuting, Sanahmadi Rose, Zou Yaya, Ghosh Gourisankar

Cooperativity among clustered κB sites within promoters and enhancers dictates transcriptional specificity of NF-κB RelA along with specific cofactors

启动子和增强子内聚集的κB位点之间的协同作用以及特定的辅助因子决定了NF-κB RelA的转录特异性。

Shahabi, Shandy; Biswas, Tapan; Shen, Yuting; Sanahmadi, Rose; Zou, Yaya; Ghosh, Gourisankar

Tumor suppressor NME1/NM23-H1 modulates DNA binding of NF-κB RelA.

肿瘤抑制因子 NME1/NM23-H1 调节 NF-κB RelA 的 DNA 结合

Shahabi Shandy, Maurya Mano, Subramaniam Shankar, Ghosh Gourisankar