HCV NS5A dimer interface residues regulate HCV replication by controlling its self-interaction, hyperphosphorylation, subcellular localization and interaction with cyclophilin A
HCV NS5A二聚体界面残基通过控制其自身相互作用、过度磷酸化、亚细胞定位以及与环孢菌素A的相互作用来调节HCV复制。
期刊:PLoS Pathogens
影响因子:4.9
doi:10.1371/journal.ppat.1007177
Shanmugam, Saravanabalaji; Nichols, Alyssa K; Saravanabalaji, Dhanaranjani; Welsch, Christoph; Yi, MinKyung
