Shen Hengyang相关文献与解析，生知库 | 链接生命科学的每一步

Wang, Zhenling; Wang, Yong; Chen, Yang; Shen, Hengyang; Lu, Yunfei; Tong, Ying; Xu, Lei; Huang, Changzhi; Zhang, Hongqiang; Shao, Yu; Li, Wenjie; Wang, Shuai; Fu, Zan

信号转导

细胞生物学

肿瘤

肿瘤免疫

发表日期：2026

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The E3 Ubiquitin Ligase ARIH1 Facilitates Colorectal Cancer Progression by Promoting Oxidative Phosphorylation via the Mitochondrial Translocation of K63-Linked Ubiquitinated PHB1

E3泛素连接酶ARIH1通过促进K63连接的泛素化PHB1的线粒体转位，进而促进氧化磷酸化，从而促进结直肠癌的进展。

期刊:Advanced Science

影响因子:14.1

doi:10.1002/advs.202501017

Tong, Ying; Wang, Zhenling; Wang, Yong; Chen, Yang; Zhang, Hongqiang; Lu, Yunfei; Xu, Lei; Shen, Hengyang; Huang, Changzhi; Zhao, Min; Li, Wenjie; Wang, Shuai; Shao, Yu; Fu, Zan

Single-cell and spatial transcriptome profiling reveal CTHRC1+âfibroblasts promote EMT through WNT5A signaling in colorectal cancer.

单细胞和空间转录组分析揭示 CTHRC1+成纤维细胞通过 WNT5A 信号传导促进结直肠癌中的 EMT

期刊:Journal of Translational Medicine

影响因子:7.5

doi:10.1186/s12967-025-06236-5

Lu Yunfei, Chen Yang, Wang Zhenling, Shen Hengyang, Xu Lei, Huang Changzhi, Tong Ying, Shao Yu, Zhang Hongqiang, Fu Zan

LncRNA CASC19 promotes the growth and glycolysis of colorectal cancer cells and tumor metastasis in mice

lncRNA CASC19促进小鼠结直肠癌细胞的生长和糖酵解以及肿瘤转移。

期刊:BMC Cancer

影响因子:3.4

doi:10.1186/s12885-025-14170-4

Zhang, Xiao; Zhao, Tingyu; Wu, Cheng; Shen, Hengyang; Yi, Jianing; Liu, Lingxiang

Recruitment of USP10 by GCS1 to deubiquitinate GRP78 promotes the progression of colorectal cancer via alleviating endoplasmic reticulum stress

GCS1 募集 USP10 去泛素化 GRP78，通过缓解内质网应激促进结直肠癌的进展。

期刊:Journal of Experimental & Clinical Cancer Research

影响因子:12.8

doi:10.1186/s13046-024-03176-8

Chen, Yang; Shen, Hengyang; Wang, Zhenling; Huang, Changzhi; Zhang, Hongqiang; Shao, Yu; Tong, Ying; Xu, Lei; Lu, Yunfei; Fu, Zan

Machine learning-based glycolysis-associated molecular classification reveals differences in prognosis, TME, and immunotherapy for colorectal cancer patients

基于机器学习的糖酵解相关分子分型揭示了结直肠癌患者预后、肿瘤微环境和免疫治疗方面的差异

期刊:Frontiers in Immunology

影响因子:5.9

doi:10.3389/fimmu.2023.1181985

Wang, Zhenling; Shao, Yu; Zhang, Hongqiang; Lu, Yunfei; Chen, Yang; Shen, Hengyang; Huang, Changzhi; Wu, Jingyu; Fu, Zan

UBE2M as a bridge spanning neddylation and cell cycle regulation in colorectal adenocarcinoma

UBE2M 在结直肠腺癌中作为连接 neddylation 和细胞周期调控的桥梁

The E3 Ubiquitin Ligase ARIH1 Facilitates Colorectal Cancer Progression by Promoting Oxidative Phosphorylation via the Mitochondrial Translocation of K63-Linked Ubiquitinated PHB1

E3泛素连接酶ARIH1通过促进K63连接的泛素化PHB1的线粒体转位，进而促进氧化磷酸化，从而促进结直肠癌的进展。

Single-cell and spatial transcriptome profiling reveal CTHRC1+âfibroblasts promote EMT through WNT5A signaling in colorectal cancer.

单细胞和空间转录组分析揭示 CTHRC1+成纤维细胞通过 WNT5A 信号传导促进结直肠癌中的 EMT

LncRNA CASC19 promotes the growth and glycolysis of colorectal cancer cells and tumor metastasis in mice

lncRNA CASC19促进小鼠结直肠癌细胞的生长和糖酵解以及肿瘤转移。

Recruitment of USP10 by GCS1 to deubiquitinate GRP78 promotes the progression of colorectal cancer via alleviating endoplasmic reticulum stress

GCS1 募集 USP10 去泛素化 GRP78，通过缓解内质网应激促进结直肠癌的进展。

Machine learning-based glycolysis-associated molecular classification reveals differences in prognosis, TME, and immunotherapy for colorectal cancer patients

基于机器学习的糖酵解相关分子分型揭示了结直肠癌患者预后、肿瘤微环境和免疫治疗方面的差异

UBE2M as a bridge spanning neddylation and cell cycle regulation in colorectal adenocarcinoma

UBE2M 在结直肠腺癌中作为连接 neddylation 和细胞周期调控的桥梁

The E3 Ubiquitin Ligase ARIH1 Facilitates Colorectal Cancer Progression by Promoting Oxidative Phosphorylation via the Mitochondrial Translocation of K63-Linked Ubiquitinated PHB1

E3泛素连接酶ARIH1通过促进K63连接的泛素化PHB1的线粒体转位，进而促进氧化磷酸化，从而促进结直肠癌的进展。

Single-cell and spatial transcriptome profiling reveal CTHRC1+âfibroblasts promote EMT through WNT5A signaling in colorectal cancer.

单细胞和空间转录组分析揭示 CTHRC1+成纤维细胞通过 WNT5A 信号传导促进结直肠癌中的 EMT

LncRNA CASC19 promotes the growth and glycolysis of colorectal cancer cells and tumor metastasis in mice

lncRNA CASC19促进小鼠结直肠癌细胞的生长和糖酵解以及肿瘤转移。

Recruitment of USP10 by GCS1 to deubiquitinate GRP78 promotes the progression of colorectal cancer via alleviating endoplasmic reticulum stress

GCS1 募集 USP10 去泛素化 GRP78，通过缓解内质网应激促进结直肠癌的进展。

Machine learning-based glycolysis-associated molecular classification reveals differences in prognosis, TME, and immunotherapy for colorectal cancer patients

基于机器学习的糖酵解相关分子分型揭示了结直肠癌患者预后、肿瘤微环境和免疫治疗方面的差异

Single-cell and spatial transcriptome profiling reveal CTHRC1+âfibroblasts promote EMT through WNT5A signaling in colorectal cancer.