Sheng Wen S相关文献与解析，生知库 | 链接生命科学的每一步

Programmed death ligand-1 induction restrains the cytotoxic T lymphocyte response against microglia.

程序性死亡配体-1的诱导抑制了针对小胶质细胞的细胞毒性T淋巴细胞反应

期刊:Glia

影响因子:5.1

doi:10.1002/glia.23932

Chauhan Priyanka, Hu Shuxian, Prasad Sujata, Sheng Wen S, Lokensgard James R

细胞生物学

发表日期：2021

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Recall Responses from Brain-Resident Memory CD8(+) T Cells (bT(RM)) Induce Reactive Gliosis

脑内驻留记忆CD8(+) T细胞(bT(RM))的回忆反应诱导反应性胶质增生

期刊:iScience

影响因子:4.1

doi:10.1016/j.isci.2019.10.005

Prasad, Sujata; Hu, Shuxian; Sheng, Wen S; Chauhan, Priyanka; Lokensgard, James R

Reactive glia promote development of CD103(+) CD69(+) CD8(+) T-cells through programmed cell death-ligand 1 (PD-L1)

反应性胶质细胞通过程序性细胞死亡配体 1 (PD-L1) 促进 CD103(+) CD69(+) CD8(+) T 细胞的发育

期刊:Immunity Inflammation and Disease

影响因子:2.7

doi:10.1002/iid3.221

Prasad, Sujata; Hu, Shuxian; Sheng, Wen S; Chauhan, Priyanka; Lokensgard, James R

The PD-1: PD-L1 pathway promotes development of brain-resident memory T cells following acute viral encephalitis

PD-1:PD-L1通路促进急性病毒性脑炎后脑内驻留记忆T细胞的发育

期刊:Journal of Neuroinflammation

影响因子:10.1

doi:10.1186/s12974-017-0860-3

Prasad, Sujata; Hu, Shuxian; Sheng, Wen S; Chauhan, Priyanka; Singh, Amar; Lokensgard, James R

Chronic reactive gliosis following regulatory T cell depletion during acute MCMV encephalitis

急性巨细胞病毒性脑炎期间调节性T细胞耗竭后发生的慢性反应性胶质增生

期刊:Glia

影响因子:5.1

doi:10.1002/glia.22868

Lokensgard, James R; Schachtele, Scott J; Mutnal, Manohar B; Sheng, Wen S; Prasad, Sujata; Hu, Shuxian

Reactive oxygen species from human astrocytes induced functional impairment and oxidative damage

人星形胶质细胞产生的活性氧会诱导功能障碍和氧化损伤。

期刊:Neurochemical Research

影响因子:3.8

doi:10.1007/s11064-013-1123-z

Sheng, Wen S; Hu, Shuxian; Feng, Amy; Rock, R Bryan

Reactive oxygen species drive herpes simplex virus (HSV)-1-induced proinflammatory cytokine production by murine microglia

活性氧驱动单纯疱疹病毒（HSV）-1诱导的小鼠小胶质细胞产生促炎细胞因子。

期刊:Journal of Neuroinflammation

影响因子:10.1

doi:10.1186/1742-2094-8-123

Hu, Shuxian; Sheng, Wen S; Schachtele, Scott J; Lokensgard, James R

Immunomodulatory properties of kappa opioids and synthetic cannabinoids in HIV-1 neuropathogenesis

κ阿片类药物和合成大麻素在HIV-1神经发病机制中的免疫调节特性

期刊:Journal of Neuroimmune Pharmacology

影响因子:3.5

doi:10.1007/s11481-011-9306-3

Hu, Shuxian; Sheng, Wen S; Rock, Robert Bryan

Hemin inhibits NO production by IL-1β-stimulated human astrocytes through induction of heme oxygenase-1 and reduction of p38 MAPK activation

血红素通过诱导血红素加氧酶-1和降低p38 MAPK活化来抑制IL-1β刺激的人星形胶质细胞产生NO。

期刊:Journal of Neuroinflammation

影响因子:10.1

doi:10.1186/1742-2094-7-51

Sheng, Wen S; Hu, Shuxian; Nettles, Adam R; Lokensgard, James R; Vercellotti, Gregory M; Rock, R Bryan

bT(RM) Control of Murine Cytomegalovirus CNS Reactivation

bT(RM) 控制小鼠巨细胞病毒中枢神经系统再激活

Programmed death ligand-1 induction restrains the cytotoxic T lymphocyte response against microglia.

程序性死亡配体-1的诱导抑制了针对小胶质细胞的细胞毒性T淋巴细胞反应

Recall Responses from Brain-Resident Memory CD8(+) T Cells (bT(RM)) Induce Reactive Gliosis

脑内驻留记忆CD8(+) T细胞(bT(RM))的回忆反应诱导反应性胶质增生

Reactive glia promote development of CD103(+) CD69(+) CD8(+) T-cells through programmed cell death-ligand 1 (PD-L1)

反应性胶质细胞通过程序性细胞死亡配体 1 (PD-L1) 促进 CD103(+) CD69(+) CD8(+) T 细胞的发育

The PD-1: PD-L1 pathway promotes development of brain-resident memory T cells following acute viral encephalitis

PD-1:PD-L1通路促进急性病毒性脑炎后脑内驻留记忆T细胞的发育

Chronic reactive gliosis following regulatory T cell depletion during acute MCMV encephalitis

急性巨细胞病毒性脑炎期间调节性T细胞耗竭后发生的慢性反应性胶质增生

Reactive oxygen species from human astrocytes induced functional impairment and oxidative damage

人星形胶质细胞产生的活性氧会诱导功能障碍和氧化损伤。

Reactive oxygen species drive herpes simplex virus (HSV)-1-induced proinflammatory cytokine production by murine microglia

活性氧驱动单纯疱疹病毒（HSV）-1诱导的小鼠小胶质细胞产生促炎细胞因子。

Immunomodulatory properties of kappa opioids and synthetic cannabinoids in HIV-1 neuropathogenesis

κ阿片类药物和合成大麻素在HIV-1神经发病机制中的免疫调节特性

Hemin inhibits NO production by IL-1β-stimulated human astrocytes through induction of heme oxygenase-1 and reduction of p38 MAPK activation

血红素通过诱导血红素加氧酶-1和降低p38 MAPK活化来抑制IL-1β刺激的人星形胶质细胞产生NO。