Shin Bongjin相关文献与解析，生知库 | 链接生命科学的每一步

T-Cell Death Associated Gene 51 Is a Novel Negative Regulator of PPARγ That Inhibits PPARγ-RXRα Heterodimer Formation in Adipogenesis

T细胞死亡相关基因51是PPARγ的一种新型负调控因子，可抑制脂肪生成过程中PPARγ-RXRα异二聚体的形成

doi:10.14348/molcells.2020.0143

Kim, Sumi; Lee, Nari; Park, Eui-Soon; Yun, Hyeongseok; Ha, Tae-Uk; Jeon, Hyoeun; Yu, Jiyeon; Choi, Seunga; Shin, Bongjin; Yu, Jungeun; Rhee, Sang Dal; Choi, Yongwon; Rho, Jaerang

TDAG51 is a crucial regulator of maternal care and depressive-like behavior after parturition.

TDAG51 是产后母性照护和抑郁样行为的关键调节因子

期刊:PLoS Genetics

影响因子:3.7

doi:10.1371/journal.pgen.1008214

Yun Hyeongseok, Park Eui-Soon, Choi Seunga, Shin Bongjin, Yu Jungeun, Yu Jiyeon, Amarasekara Dulshara Sachini, Kim Sumi, Lee Nari, Choi Jong-Soon, Choi Yongwon, Rho Jaerang

Protease-Activated Receptor 1 Deletion Causes Enhanced Osteoclastogenesis in Response to Inflammatory Signals through a Notch2-Dependent Mechanism

蛋白酶激活受体1缺失通过Notch2依赖性机制导致炎症信号诱导的破骨细胞生成增强。

期刊:Journal of Immunology

影响因子:3.4

doi:10.4049/jimmunol.1801032

Jastrzebski, Sandra; Kalinowski, Judith; Mun, Sehwan; Shin, Bongjin; Adapala, Naga Suresh; Jacome-Galarza, Christian E; Mirza, Faryal; Aguila, H Leonardo; Drissi, Hicham; Sanjay, Archana; Canalis, Ernesto; Lee, Sun-Kyeong; Lorenzo, Joseph A

Notch

发表日期：2019

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Contradictory Role of CD97 in Basal and Tumor Necrosis Factor-Induced Osteoclastogenesis In Vivo

CD97在体内基础和肿瘤坏死因子诱导的破骨细胞生成中发挥着矛盾的作用

期刊:Arthritis & Rheumatology

影响因子:10.9

doi:10.1002/art.39538

Yeon Won, Hee; Hwan Mun, Se; Shin, Bongjin; Lee, Sun-Kyeong

Secretion of a truncated osteopetrosis-associated transmembrane protein 1 (OSTM1) mutant inhibits osteoclastogenesis through down-regulation of the B lymphocyte-induced maturation protein 1 (BLIMP1)-nuclear factor of activated T cells c1 (NFATc1) axis

截短的骨硬化症相关跨膜蛋白1 (OSTM1) 突变体的分泌通过下调B淋巴细胞诱导成熟蛋白1 (BLIMP1)-活化T细胞核因子c1 (NFATc1) 轴抑制破骨细胞生成。

期刊:Journal of Biological Chemistry

影响因子:3.9

doi:10.1074/jbc.M114.589614

Shin, Bongjin; Yu, Jungeun; Park, Eui-Soon; Choi, Seunga; Yu, Jiyeon; Hwang, Jung Me; Yun, Hyeongseok; Chung, Young-Ho; Hong, Kwan Soo; Choi, Jong-Soon; Takami, Masamichi; Rho, Jaerang

The miR-29-3p family suppresses inflammatory osteolysis

miR-29-3p家族抑制炎症性骨溶解

T-Cell Death Associated Gene 51 Is a Novel Negative Regulator of PPARγ That Inhibits PPARγ-RXRα Heterodimer Formation in Adipogenesis

T细胞死亡相关基因51是PPARγ的一种新型负调控因子，可抑制脂肪生成过程中PPARγ-RXRα异二聚体的形成

TDAG51 is a crucial regulator of maternal care and depressive-like behavior after parturition.

TDAG51 是产后母性照护和抑郁样行为的关键调节因子

Protease-Activated Receptor 1 Deletion Causes Enhanced Osteoclastogenesis in Response to Inflammatory Signals through a Notch2-Dependent Mechanism

蛋白酶激活受体1缺失通过Notch2依赖性机制导致炎症信号诱导的破骨细胞生成增强。

Contradictory Role of CD97 in Basal and Tumor Necrosis Factor-Induced Osteoclastogenesis In Vivo

CD97在体内基础和肿瘤坏死因子诱导的破骨细胞生成中发挥着矛盾的作用

Secretion of a truncated osteopetrosis-associated transmembrane protein 1 (OSTM1) mutant inhibits osteoclastogenesis through down-regulation of the B lymphocyte-induced maturation protein 1 (BLIMP1)-nuclear factor of activated T cells c1 (NFATc1) axis

截短的骨硬化症相关跨膜蛋白1 (OSTM1) 突变体的分泌通过下调B淋巴细胞诱导成熟蛋白1 (BLIMP1)-活化T细胞核因子c1 (NFATc1) 轴抑制破骨细胞生成。