Shinde Arti V相关文献与解析，生知库 | 链接生命科学的每一步

Humeres, Claudio; Shinde, Arti V; Tuleta, Izabela; Hernandez, Silvia C; Hanna, Anis; Huang, Shuaibo; Venugopal, Harikrishnan; Aguilan, Jennifer T; Conway, Simon J; Sidoli, Simone; Frangogiannis, Nikolaos G

成纤维细胞

细胞生物学

发表日期：2024

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Protective Effects of Activated Myofibroblasts in the Pressure-Overloaded Myocardium Are Mediated Through Smad-Dependent Activation of a Matrix-Preserving Program

活化肌成纤维细胞在压力超负荷心肌中的保护作用是通过Smad依赖性基质保护程序的激活介导的。

期刊:Circulation Research

影响因子:16.2

doi:10.1161/CIRCRESAHA.118.314438

Russo, Ilaria; Cavalera, Michele; Huang, Shuaibo; Su, Ya; Hanna, Anis; Chen, Bijun; Shinde, Arti V; Conway, Simon J; Graff, Jonathan; Frangogiannis, Nikolaos G

Distinct roles of myofibroblast-specific Smad2 and Smad3 signaling in repair and remodeling of the infarcted heart.

肌成纤维细胞特异性 Smad2 和 Smad3 信号在梗死心脏的修复和重塑中发挥着不同的作用

期刊:Journal of Molecular and Cellular Cardiology

影响因子:4.7

doi:10.1016/j.yjmcc.2019.05.006

Huang Shuaibo, Chen Bijun, Su Ya, Alex Linda, Humeres Claudio, Shinde Arti V, Conway Simon J, Frangogiannis Nikolaos G

细胞生物学

发表日期：2019

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Opposing Actions of Fibroblast and Cardiomyocyte Smad3 Signaling in the Infarcted Myocardium

梗死心肌中成纤维细胞和心肌细胞Smad3信号的相反作用

期刊:Circulation

影响因子:38.6

doi:10.1161/CIRCULATIONAHA.117.029622

Kong, Ping; Shinde, Arti V; Su, Ya; Russo, Ilaria; Chen, Bijun; Saxena, Amit; Conway, Simon J; Graff, Jonathan M; Frangogiannis, Nikolaos G

Tissue transglutaminase in the pathogenesis of heart failure

组织转谷氨酰胺酶在心力衰竭发病机制中的作用

期刊:Cell Death and Differentiation

影响因子:15.4

doi:10.1038/s41418-017-0028-9

Shinde, Arti V; Frangogiannis, Nikolaos G

Mechanisms of Fibroblast Activation in the Remodeling Myocardium

重塑心肌中成纤维细胞活化的机制

期刊:Current Pathobiology Reports

影响因子:

doi:10.1007/s40139-017-0132-z

Shinde, Arti V; Frangogiannis, Nikolaos G

Myocardial Galectin-3 Expression Is Associated with Remodeling of the Pressure-Overloaded Heart and May Delay the Hypertrophic Response without Affecting Survival, Dysfunction, and Cardiac Fibrosis

心肌半乳糖凝集素-3表达与压力负荷过重心脏的重塑相关，并可能延缓肥大反应，而不影响生存、功能障碍和心肌纤维化。

期刊:American Journal of Pathology

影响因子:3.6

doi:10.1016/j.ajpath.2015.12.017

Frunza, Olga; Russo, Ilaria; Saxena, Amit; Shinde, Arti V; Humeres, Claudio; Hanif, Waqas; Rai, Vikrant; Su, Ya; Frangogiannis, Nikolaos G

发表日期：2016

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The Î±4Î²1 integrin and the EDA domain of fibronectin regulate a profibrotic phenotype in dermal fibroblasts.

α4β1整合素和纤连蛋白的EDA结构域调节真皮成纤维细胞的促纤维化表型

期刊:Matrix Biology

影响因子:4.8

doi:10.1016/j.matbio.2014.11.004

Shinde Arti V, Kelsh Rhiannon, Peters John H, Sekiguchi Kiyotoshi, Van De Water Livingston, McKeown-Longo Paula J

细胞生物学

发表日期：2015

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The role of Interleukin Receptor Associated Kinase (IRAK)-M in regulation of myofibroblast phenotype in vitro, and in an experimental model of non-reperfused myocardial infarction

白细胞介素受体相关激酶（IRAK）-M在体外和非再灌注心肌梗死实验模型中对肌成纤维细胞表型调控的作用

期刊:Journal of Molecular and Cellular Cardiology

影响因子:4.7

doi:10.1016/j.yjmcc.2015.11.001

Saxena, Amit; Shinde, Arti V; Haque, Zaffar; Wu, Yi-Jin; Chen, Wei; Su, Ya; Frangogiannis, Nikolaos G

Fibroblasts in myocardial infarction: a role in inflammation and repair

心肌梗死中的成纤维细胞：在炎症和修复中的作用

期刊:Journal of Molecular and Cellular Cardiology

影响因子:4.7

doi:10.1016/j.yjmcc.2013.11.015

Shinde, Arti V; Frangogiannis, Nikolaos G

Fibroblast Smad7 Induction Protects the Remodeling Pressure-Overloaded Heart

成纤维细胞Smad7诱导可保护重塑期压力负荷过重的心脏

Protective Effects of Activated Myofibroblasts in the Pressure-Overloaded Myocardium Are Mediated Through Smad-Dependent Activation of a Matrix-Preserving Program

活化肌成纤维细胞在压力超负荷心肌中的保护作用是通过Smad依赖性基质保护程序的激活介导的。

Distinct roles of myofibroblast-specific Smad2 and Smad3 signaling in repair and remodeling of the infarcted heart.

肌成纤维细胞特异性 Smad2 和 Smad3 信号在梗死心脏的修复和重塑中发挥着不同的作用

Opposing Actions of Fibroblast and Cardiomyocyte Smad3 Signaling in the Infarcted Myocardium

梗死心肌中成纤维细胞和心肌细胞Smad3信号的相反作用

Tissue transglutaminase in the pathogenesis of heart failure

组织转谷氨酰胺酶在心力衰竭发病机制中的作用

Mechanisms of Fibroblast Activation in the Remodeling Myocardium

重塑心肌中成纤维细胞活化的机制

Myocardial Galectin-3 Expression Is Associated with Remodeling of the Pressure-Overloaded Heart and May Delay the Hypertrophic Response without Affecting Survival, Dysfunction, and Cardiac Fibrosis

心肌半乳糖凝集素-3表达与压力负荷过重心脏的重塑相关，并可能延缓肥大反应，而不影响生存、功能障碍和心肌纤维化。

The Î±4Î²1 integrin and the EDA domain of fibronectin regulate a profibrotic phenotype in dermal fibroblasts.

α4β1整合素和纤连蛋白的EDA结构域调节真皮成纤维细胞的促纤维化表型

The role of Interleukin Receptor Associated Kinase (IRAK)-M in regulation of myofibroblast phenotype in vitro, and in an experimental model of non-reperfused myocardial infarction

白细胞介素受体相关激酶（IRAK）-M在体外和非再灌注心肌梗死实验模型中对肌成纤维细胞表型调控的作用

Fibroblasts in myocardial infarction: a role in inflammation and repair

心肌梗死中的成纤维细胞：在炎症和修复中的作用