Shirai Remina相关文献与解析，生知库 | 链接生命科学的每一步

Modulating Golgi Stress Signaling Ameliorates Cell Morphological Phenotypes Induced by CHMP2B with Frontotemporal Dementia-Associated p.Asp148Tyr.

调节高尔基应激信号可改善由 CHMP2B 诱导的与额颞叶痴呆相关的 p.Asp148Tyr 的细胞形态表型

doi:10.3390/cimb46020090

Fukatsu Shoya, Okawa Maho, Okabe Miyu, Cho Mizuka, Isogai Mikinori, Yokoi Takanori, Shirai Remina, Oizumi Hiroaki, Yamamoto Masahiro, Ohbuchi Katsuya, Miyamoto Yuki, Yamauchi Junji

FTD/ALS Type 7-Associated Thr104Asn Mutation of CHMP2B Blunts Neuronal Process Elongation, and Is Recovered by Knockdown of Arf4, the Golgi Stress Regulator.

FTD/ALS 7 型相关 CHMP2B Thr104Asn 突变会抑制神经元突起的延伸，而敲低高尔基体应激调节因子 Arf4 可以恢复这种抑制作用

期刊:Neurology International

影响因子:3

doi:10.3390/neurolint15030063

Shirai Remina, Cho Mizuka, Isogai Mikinori, Fukatsu Shoya, Okabe Miyu, Okawa Maho, Miyamoto Yuki, Torii Tomohiro, Yamauchi Junji

RhoG-Binding Domain of Elmo1 Ameliorates Excessive Process Elongation Induced by Autism Spectrum Disorder-Associated Sema5A.

Elmo1 的 RhoG 结合域可改善自闭症谱系障碍相关 Sema5A 引起的过度突起延伸

期刊:Pathophysiology

影响因子:2.6

doi:10.3390/pathophysiology30040040

Okabe Miyu, Miyamoto Yuki, Ikoma Yuta, Takahashi Mikito, Shirai Remina, Kukimoto-Niino Mutsuko, Shirouzu Mikako, Yamauchi Junji

其它

发表日期：2023

文献求助收藏

New Insights into Risk Genes and Their Candidates in Multiple Sclerosis

多发性硬化症风险基因及其候选基因的新见解

期刊:Neurology International

影响因子:3

doi:10.3390/neurolint15010003

Shirai, Remina; Yamauchi, Junji

β-Endorphin Mediates the Development and Instability of Atherosclerotic Plaques

β-内啡肽介导动脉粥样硬化斑块的形成和不稳定性

期刊:International Journal of Endocrinology

影响因子:2.3

doi:10.1155/2020/4139093

Okano, Taisuke; Sato, Kengo; Shirai, Remina; Seki, Tomomi; Shibata, Koichiro; Yamashita, Tomoyuki; Koide, Ayaka; Tezuka, Hitomi; Mori, Yusaku; Hirano, Tsutomu; Watanabe, Takuya

Anti-Atherogenic Effects of Vaspin on Human Aortic Smooth Muscle Cell/Macrophage Responses and Hyperlipidemic Mouse Plaque Phenotype

Vaspin对人主动脉平滑肌细胞/巨噬细胞反应和高脂血症小鼠斑块表型的抗动脉粥样硬化作用

期刊:International Journal of Molecular Sciences

影响因子:4.9

doi:10.3390/ijms19061732

Sato, Kengo; Shirai, Remina; Yamaguchi, Maho; Yamashita, Tomoyuki; Shibata, Koichiro; Okano, Taisuke; Mori, Yusaku; Matsuyama, Taka-Aki; Ishibashi-Ueda, Hatsue; Hirano, Tsutomu; Watanabe, Takuya

Adropin Contributes to Anti-Atherosclerosis by Suppressing Monocyte-Endothelial Cell Adhesion and Smooth Muscle Cell Proliferation.

Adropin 通过抑制单核细胞-内皮细胞粘附和平滑肌细胞增殖，发挥抗动脉粥样硬化作用

期刊:International Journal of Molecular Sciences

影响因子:4.9

doi:10.3390/ijms19051293

Sato Kengo, Yamashita Tomoyuki, Shirai Remina, Shibata Koichiro, Okano Taisuke, Yamaguchi Maho, Mori Yusaku, Hirano Tsutomu, Watanabe Takuya

细胞生物学

发表日期：2018

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Vasoprotective effects of urocortin 1 against atherosclerosis in vitro and in vivo.

尿皮质素 1 在体外和体内对动脉粥样硬化的血管保护作用

期刊:PLoS One

影响因子:2.6

doi:10.1371/journal.pone.0110866

Hasegawa Akinori, Sato Kengo, Shirai Remina, Watanabe Rena, Yamamoto Keigo, Watanabe Kaho, Nohtomi Kyoko, Hirano Tsutomu, Watanabe Takuya

Emerging Evidence of Golgi Stress Signaling for Neuropathies

神经病变中高尔基体应激信号传导的新证据

Modulating Golgi Stress Signaling Ameliorates Cell Morphological Phenotypes Induced by CHMP2B with Frontotemporal Dementia-Associated p.Asp148Tyr.

调节高尔基应激信号可改善由 CHMP2B 诱导的与额颞叶痴呆相关的 p.Asp148Tyr 的细胞形态表型

FTD/ALS Type 7-Associated Thr104Asn Mutation of CHMP2B Blunts Neuronal Process Elongation, and Is Recovered by Knockdown of Arf4, the Golgi Stress Regulator.

FTD/ALS 7 型相关 CHMP2B Thr104Asn 突变会抑制神经元突起的延伸，而敲低高尔基体应激调节因子 Arf4 可以恢复这种抑制作用

RhoG-Binding Domain of Elmo1 Ameliorates Excessive Process Elongation Induced by Autism Spectrum Disorder-Associated Sema5A.

Elmo1 的 RhoG 结合域可改善自闭症谱系障碍相关 Sema5A 引起的过度突起延伸

New Insights into Risk Genes and Their Candidates in Multiple Sclerosis

多发性硬化症风险基因及其候选基因的新见解

β-Endorphin Mediates the Development and Instability of Atherosclerotic Plaques

β-内啡肽介导动脉粥样硬化斑块的形成和不稳定性

Anti-Atherogenic Effects of Vaspin on Human Aortic Smooth Muscle Cell/Macrophage Responses and Hyperlipidemic Mouse Plaque Phenotype

Vaspin对人主动脉平滑肌细胞/巨噬细胞反应和高脂血症小鼠斑块表型的抗动脉粥样硬化作用

Adropin Contributes to Anti-Atherosclerosis by Suppressing Monocyte-Endothelial Cell Adhesion and Smooth Muscle Cell Proliferation.

Adropin 通过抑制单核细胞-内皮细胞粘附和平滑肌细胞增殖，发挥抗动脉粥样硬化作用

Vasoprotective effects of urocortin 1 against atherosclerosis in vitro and in vivo.

尿皮质素 1 在体外和体内对动脉粥样硬化的血管保护作用