日期 影响因子
日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:
–

Dupilumab in Patients With Chronic Spontaneous Urticaria: Phase 3 LIBERTY-CSU CUPID Randomized Clinical Trials

Dupilumab治疗慢性自发性荨麻疹患者：LIBERTY-CSU CUPID 3期随机临床试验

期刊:Jama Dermatology
影响因子:11
doi:10.1001/jamadermatol.2025.6023
Casale, Thomas B; Saini, Sarbjit S; Ben-Shoshan, Moshe; Giménez-Arnau, Ana M; Bernstein, Jonathan A; Hayama, Koremasa; Amin, Nikhil; Robinson, Lacey B; Bauer, Deborah; Dakin, Paula; Laws, Elizabeth; Radin, Allen; Makhija, Melanie
麻疹
发表日期：2026
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Signaling pathways regulating VDAC1 overexpression associated with apoptosis, pyroptosis, and ferroptosis

调控VDAC1过表达的信号通路与细胞凋亡、细胞焦亡和铁死亡相关

期刊:Cell Communication and Signaling
影响因子:8.9
doi:10.1186/s12964-025-02647-5
Karunanithi Nivedita, Aditya; Santhanam, Manikandan; Babu, Venkatadri; Shteinfer-Kuzmine, Anna; Garcia Venzor, Alfredo; Toiber, Debra; Shoshan-Barmatz, Varda
凋亡
表观遗传
信号转导
铁死亡
VDAC1
焦亡
细胞生物学
发表日期：2026
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Targeting VDAC1 to protect against mitochondria-linked cell death pathways: apoptosis, pyroptosis, ferroptosis, and associated diseases

靶向VDAC1可预防线粒体相关细胞死亡途径（包括细胞凋亡、细胞焦亡、铁死亡及其相关疾病）。

期刊:Apoptosis
影响因子:8.1
doi:10.1007/s10495-025-02217-7
Shteinfer-Kuzmine, A; Nivedita, A Karunanithi; Santhanam, M; Trishna, S; Swerdlow, R W; Pan, J; Shoshan-Barmatz, V
凋亡
表观遗传
铁死亡
VDAC1
线粒体
焦亡
细胞生物学
发表日期：2026
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Single stereocenter inversion of a cyclic tetrapeptide enables the detoxification of lead-exposed mice

环状四肽的单个立体中心反转可使铅暴露小鼠解毒。

期刊:Chemical Science
影响因子:7.4
doi:10.1039/d5sc09201a
Mohammed, Tagwa A; Sauser, Luca; Pedron, Erica; Hodel, Yaël A; Kalvoda, Tadeáš; Prisco, Francesco; Rulíšek, Lubomír; Holland, Jason P; Shoshan, Michal S
发表日期：2026
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Estimation of metabolic water production in human and rat brain and spinal cord

人脑和鼠脑脊髓代谢水生成量的估算

期刊:Fluids and Barriers of the Cns
影响因子:6.2
doi:10.1186/s12987-026-00769-7
Hvass, Cecilie R; Ben-Shoshan, Shai D; Carlsen, Jonathan F; Aldana, Blanca I; Quistorff, Bjørn; Hansen, Adam E; MacAulay, Nanna
神经科学
代谢
发表日期：2026
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Survivin/BIRC5-derived peptide disrupts survivin dimerization and cell division and induces multifaceted anti-cancer effects

Survivin/BIRC5衍生肽可破坏Survivin二聚化和细胞分裂，并诱导多方面的抗癌作用。

期刊:Molecular Therapy Oncology
影响因子:5.3
doi:10.1016/j.omton.2025.201123
Santhanam, Manikandan; Babu, Venkatadri; Shteinfer-Kuzmine, Anna; Pandey, Swaroop Kumar; Gheber, Larisa; Raday, Gilead; Shoshan-Barmatz, Varda
细胞生物学
肿瘤
肿瘤免疫
Survivin
发表日期：2026
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Erratum: Survivin/BIRC5-derived peptide disrupts survivin dimerization and cell division and induces multifaceted anti-cancer effects

更正：Survivin/BIRC5衍生肽可破坏survivin二聚化和细胞分裂，并诱导多方面的抗癌作用

期刊:Molecular Therapy Oncology
影响因子:5.3
doi:10.1016/j.omton.2026.201142
Santhanam, Manikandan; Babu, Venkatadri; Shteinfer-Kuzmine, Anna; Pandey, Swaroop Kumar; Gheber, Larisa; Raday, Gilead; Shoshan-Barmatz, Varda
细胞生物学
肿瘤
肿瘤免疫
Survivin
发表日期：2026
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Empowering exhausted T cells of Glioblastoma patients by Neurotransmitters and Neuropeptides: decreasing immune checkpoint inhibitors, and increasing CD3zeta, proliferation and Glioblastoma arrest

通过神经递质和神经肽激活胶质母细胞瘤患者耗竭的T细胞：降低免疫检查点抑制剂水平，增加CD3ζ，促进增殖并抑制胶质母细胞瘤生长。

期刊:Cancer Immunology Immunotherapy
影响因子:5.1
doi:10.1007/s00262-025-04226-6
Levite, Mia; Ilouz, Nili; Galun, Eithan; Shoshan, Yigal
T细胞
神经科学
CD3
T细胞
细胞生物学
免疫/内分泌
发表日期：2026
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p53 Interacts with VDAC1, Modulating Its Expression Level and Oligomeric State to Activate Apoptosis.

p53 与 VDAC1 相互作用，调节其表达水平和寡聚状态，从而激活细胞凋亡。

期刊:Biomolecules
影响因子:4.8
doi:10.3390/biom16010141
Gigi Elinor, Karunanithi Nivedita Aditya, Ben-Hail Danya, Santhanam Manikandan, Shteinfer-Kuzmine Anna, Shoshan-Barmatz Varda
细胞生物学
P53
VDAC1
Apoptosis
凋亡
发表日期：2026
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Challenges in Differentiating Chronic Inducible Urticaria from Chronic Spontaneous Urticaria

区分慢性诱发性荨麻疹和慢性自发性荨麻疹的挑战

期刊:Journal of Inflammation Research
影响因子:4.1
doi:10.2147/JIR.S546302
Bendayan, Ethan; Ton That, Alexandre; Zhu, Catherine Keying; Netchiporouk, Elena; Ben-Shoshan, Moshe
麻疹
发表日期：2026
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