Neuronal cathepsin S increases neuroinflammation and causes cognitive decline via CX3CL1-CX3CR1 axis and JAK2-STAT3 pathway in aging and Alzheimer's disease
神经元组织蛋白酶S通过CX3CL1-CX3CR1轴和JAK2-STAT3通路增加神经炎症,并在衰老和阿尔茨海默病中导致认知功能下降。
期刊:Aging Cell
影响因子:8
doi:10.1111/acel.14393
Pei-Pei Liu,Xiao-Hui Liu,Ming-Jing Ren,Xiao-Tong Liu,Xiao-Qing Shi,Ming-Li Li,Shu-Ang Li,Yang Yang,Dian-Dian Wang,Yue Wu,Fan-Xiang Yin,Yan-Hong Guo,Run-Zhou Yang,Meng Cheng,Yong-Juan Xin,Jian-Sheng Kang,Bing Huang,Kai-Di Ren
神经炎症
神经科学
炎症/感染
信号转导
发育与干细胞
CR1
JAK2
STAT3
CX3CL1
Immunology/Inflammation
炎症