Silverstein Roy L相关文献与解析，生知库

Goncalves, Bruno S; Wang, Yaxin; Pillai, Sneha S; Lakhani, Hari V; Chaudhry, Aslam; Shapiro, Joseph I; Silverstein, Roy L; Sodhi, Komal; Chen, Yiliang

信号转导

细胞生物学

心血管

动脉粥样硬化

发表日期：2025

文献求助收藏

Macrophage PIM1 Drives Atherosclerosis by Enhancing Foam Cell Formation Via CD36

巨噬细胞 PIM1 通过 CD36 促进泡沫细胞形成，从而驱动动脉粥样硬化

期刊:

影响因子:

doi:10.1101/2025.10.31.685966

Beg, Mirza Ahmar; Luu, Quoc Quang; Chen, Vaya; Wang, Yaxin; Xin, Gang; Cui, Weiguo; Silverstein, Roy L; Chen, Yiliang

Distinguishing ASH clinical practice guidelines from other forms of ASH clinical advice

区分ASH临床实践指南与其他形式的ASH临床建议

期刊:Blood Advances

影响因子:7.1

doi:10.1182/bloodadvances.2023011102

Cuker, Adam; Kunkle, Robert; Bercovitz, Rachel S; Byrne, Michael; Djulbegovic, Benjamin; Haberichter, Sandra L; Holter-Chakrabarty, Jennifer; Lottenberg, Richard; Pai, Menaka; Rezende, Suely M; Seftel, Matthew D; Silverstein, Roy L; Terrell, Deirdra R; Cheung, Matthew C

发表日期：2024

文献求助收藏

Targeting Cysteine Oxidation in Thrombotic Disorders

靶向半胱氨酸氧化治疗血栓性疾病

期刊:Antioxidants

影响因子:6.6

doi:10.3390/antiox13010083

Yang, Moua; Silverstein, Roy L

心血管

发表日期：2024

文献求助收藏

A CD36 transmembrane domain peptide interrupts CD36 interactions with membrane partners on macrophages and inhibits atherogenic functions

CD36跨膜结构域肽段可干扰CD36与巨噬细胞膜伴侣的相互作用，从而抑制动脉粥样硬化功能。

期刊:Translational Research

影响因子:5.9

doi:10.1016/j.trsl.2022.10.005

Huang, Wenxin; Li, Renhao; Zhang, Jue; Cheng, Yiliang; Ramakrishnan, Devi P; Silverstein, Roy L

Contribution of adipocyte Na/K-ATPase α1/CD36 signaling induced exosome secretion in response to oxidized LDL

脂肪细胞Na/K-ATPase α1/CD36信号通路在氧化低密度脂蛋白（oxLDL）刺激下诱导外泌体分泌。

期刊:Frontiers in Cardiovascular Medicine

影响因子:2.9

doi:10.3389/fcvm.2023.1046495

Pillai, Sneha S; Pereira, Duane G; Zhang, Jue; Huang, Wenxin; Beg, Mirza Ahmar; Knaack, Darcy A; de Souza Goncalves, Bruno; Sahoo, Daisy; Silverstein, Roy L; Shapiro, Joseph I; Sodhi, Komal; Chen, Yiliang

CD36, a signaling receptor and fatty acid transporter that regulates immune cell metabolism and fate

CD36是一种信号受体和脂肪酸转运蛋白，可调节免疫细胞的代谢和命运。

期刊:Journal of Experimental Medicine

影响因子:10.6

doi:10.1084/jem.20211314

Chen, Yiliang; Zhang, Jue; Cui, Weiguo; Silverstein, Roy L

Chronic diseases alter the platelet rheostat to promote hyperreactivity and thrombosis

慢性疾病会改变血小板的调节机制，促进其过度反应和血栓形成。

Just Say NO to CD36 Expression: A Regulatory Pathway With Implications in Many Chronic Diseases

拒绝抑制CD36表达：一条与多种慢性疾病相关的调控通路

FcR γ-chain and platelet hyperactivity

FcR γ链和血小板高活性

Na/K-ATPase Signaling in Adipocytes Promotes Atherosclerosis

脂肪细胞中的Na/K-ATPase信号传导促进动脉粥样硬化

Macrophage PIM1 Drives Atherosclerosis by Enhancing Foam Cell Formation Via CD36

巨噬细胞 PIM1 通过 CD36 促进泡沫细胞形成，从而驱动动脉粥样硬化

Distinguishing ASH clinical practice guidelines from other forms of ASH clinical advice

区分ASH临床实践指南与其他形式的ASH临床建议

Targeting Cysteine Oxidation in Thrombotic Disorders

靶向半胱氨酸氧化治疗血栓性疾病

A CD36 transmembrane domain peptide interrupts CD36 interactions with membrane partners on macrophages and inhibits atherogenic functions

CD36跨膜结构域肽段可干扰CD36与巨噬细胞膜伴侣的相互作用，从而抑制动脉粥样硬化功能。

Contribution of adipocyte Na/K-ATPase α1/CD36 signaling induced exosome secretion in response to oxidized LDL

脂肪细胞Na/K-ATPase α1/CD36信号通路在氧化低密度脂蛋白（oxLDL）刺激下诱导外泌体分泌。

CD36, a signaling receptor and fatty acid transporter that regulates immune cell metabolism and fate

CD36是一种信号受体和脂肪酸转运蛋白，可调节免疫细胞的代谢和命运。