Sleiman Yvonne相关文献与解析，生知库 | 链接生命科学的每一步

Cupelli, Michael; Ginjupalli, Vamsi Krishna Murthy; Reisqs, Jean-Baptiste; Sleiman, Yvonne; Becerra-Flores, Manuel; Accioli, Riccardo; Lazzerini, Pietro Enea; Cardozo, Timothy; Boutjdir, Mohamed

免疫/内分泌

ERG

发表日期：2026

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Electrophysiological abnormalities associated with a CACNA1D variant are rescued by AAV6-Cav1.3-C-terminus gene therapy in patient-iPSC-CMs

在患者诱导多能干细胞来源的心肌细胞中，AAV6-Cav1.3-C端基因疗法可以挽救与CACNA1D变异相关的电生理异常。

期刊:

影响因子:

doi:10.64898/2025.12.15.694396

Reisqs, Jean-Baptiste; Sleiman, Yvonne; Gando, Ivan; Cupelli, Michael; Ginjupalli, Vamsi Krishna Murthy; Delanoe-Ayari, Helene; Coetzee, William; Qu, Yongxia S; Delmar, Mario; Cecchin, Frank; Tan, Reina B; Chahine, Mohamed; Boutjdir, Mohamed

Role of Cav1.3 Channels in Brain-Heart Interactions: An Unexpected Journey

Cav1.3通道在脑-心相互作用中的作用：一段意想不到的旅程

期刊:Biomedicines

影响因子:

doi:10.3390/biomedicines13061376

Reisqs, Jean-Baptiste; Sleiman, Yvonne; Cupelli, Michael; Boutjdir, Mohamed

神经科学

发表日期：2025

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Electrophysiological basis of cardiac arrhythmia in a mouse model of myotonic dystrophy type 1.

1 型强直性肌营养不良小鼠模型心律失常的电生理基础

期刊:Frontiers in Physiology

影响因子:3.4

doi:10.3389/fphys.2023.1257682

Ginjupalli Vamsi Krishna Murthy, Cupelli Michael, Reisqs Jean-Baptiste, Sleiman Yvonne, El-Sherif Nabil, Gourdon Genevieve, Puymirat Jack, Chahine Mohamed, Boutjdir Mohamed

Spironolactone as a Potential New Treatment to Prevent Arrhythmias in Arrhythmogenic Cardiomyopathy Cell Model

螺内酯作为一种潜在的新疗法，可用于预防致心律失常性心肌病细胞模型中的心律失常

期刊:Journal of Personalized Medicine

影响因子:3

doi:10.3390/jpm13020335

Reisqs, Jean-Baptiste; Moreau, Adrien; Sleiman, Yvonne; Charrabi, Azzouz; Delinière, Antoine; Bessière, Francis; Gardey, Kevin; Richard, Sylvain; Chevalier, Philippe

Correction: "Ryanopathies" and RyR2 dysfunctions: can we further decipher them using in vitro human disease models?

更正：“Ryanopathies”和 RyR2 功能障碍：我们能否利用体外人类疾病模型进一步解读它们？

期刊:Cell Death & Disease

影响因子:9.6

doi:10.1038/s41419-022-05468-3

Sleiman, Yvonne; Lacampagne, Alain; Meli, Albano C

发表日期：2022

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The PPARγ pathway determines electrophysiological remodelling and arrhythmia risks in DSC2 arrhythmogenic cardiomyopathy

PPARγ通路决定DSC2致心律失常性心肌病中的电生理重构和心律失常风险。

期刊:Clinical and Translational Medicine

影响因子:6.8

doi:10.1002/ctm2.748

Reisqs, Jean-Baptiste; Moreau, Adrien; Charrabi, Azzouz; Sleiman, Yvonne; Meli, Albano C; Millat, Gilles; Briand, Veronique; Beauverger, Philippe; Richard, Sylvain; Chevalier, Philippe

"Ryanopathies" and RyR2 dysfunctions: can we further decipher them using in vitro human disease models?

“Ryanopathies”和RyR2功能障碍：我们能否利用体外人类疾病模型进一步解读它们？

期刊:Cell Death & Disease

影响因子:9.6

doi:10.1038/s41419-021-04337-9

Sleiman, Yvonne; Lacampagne, Alain; Meli, Albano C

发表日期：2021

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hERG epitope mimic-decoy peptide corrects autoimmune-long QT syndrome in guinea pigs

hERG表位模拟诱饵肽可纠正豚鼠自身免疫性长QT间期综合征

Electrophysiological abnormalities associated with a CACNA1D variant are rescued by AAV6-Cav1.3-C-terminus gene therapy in patient-iPSC-CMs

在患者诱导多能干细胞来源的心肌细胞中，AAV6-Cav1.3-C端基因疗法可以挽救与CACNA1D变异相关的电生理异常。

Role of Cav1.3 Channels in Brain-Heart Interactions: An Unexpected Journey

Cav1.3通道在脑-心相互作用中的作用：一段意想不到的旅程

Electrophysiological basis of cardiac arrhythmia in a mouse model of myotonic dystrophy type 1.

1 型强直性肌营养不良小鼠模型心律失常的电生理基础

Spironolactone as a Potential New Treatment to Prevent Arrhythmias in Arrhythmogenic Cardiomyopathy Cell Model

螺内酯作为一种潜在的新疗法，可用于预防致心律失常性心肌病细胞模型中的心律失常

Correction: "Ryanopathies" and RyR2 dysfunctions: can we further decipher them using in vitro human disease models?

更正：“Ryanopathies”和 RyR2 功能障碍：我们能否利用体外人类疾病模型进一步解读它们？

The PPARγ pathway determines electrophysiological remodelling and arrhythmia risks in DSC2 arrhythmogenic cardiomyopathy

PPARγ通路决定DSC2致心律失常性心肌病中的电生理重构和心律失常风险。

"Ryanopathies" and RyR2 dysfunctions: can we further decipher them using in vitro human disease models?

“Ryanopathies”和RyR2功能障碍：我们能否利用体外人类疾病模型进一步解读它们？