日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Multivariate Host-Pathogen Interactions Driving Heterogeneous Viral Shedding and CD8(+) T cell Control of Influenza Virus Infection

多变量宿主-病原体相互作用驱动病毒脱落的异质性以及CD8(+) T细胞对流感病毒感染的控制

Bruce, Nicole; Weaver, Jordan J A; Jeynes-Smith, Cailan; Beigmohammadi, Fatemeh; Hegarty-Cremer, Solène; Smith, Amanda P; Craig, Morgan; Smith, Amber M

Dissecting Phenotype from Genotype with Clinical Isolates of SARS-CoV-2 First Wave Variants.

利用SARS-CoV-2第一波变异株的临床分离株,从基因型和表型中解析出差异

Taylor Mariah K, Williams Evan P, Xue Yi, Jenjaroenpun Piroon, Wongsurawat Thidathip, Smith Amanda P, Smith Amber M, Parvathareddy Jyothi, Kong Ying, Vogel Peter, Cao Xueyuan, Reichard Walter, Spruill-Harrell Briana, Samarasinghe Amali E, Nookaew Intawat, Fitzpatrick Elizabeth A, Smith Micholas Dean, Aranha Michelle, Smith Jeremy C, Jonsson Colleen B

Time-Dependent Increase in Susceptibility and Severity of Secondary Bacterial Infections During SARS-CoV-2.

SARS-CoV-2 感染期间继发性细菌感染的易感性和严重程度随时间增加

Smith Amanda P, Williams Evan P, Plunkett Taylor R, Selvaraj Muneeswaran, Lane Lindey C, Zalduondo Lillian, Xue Yi, Vogel Peter, Channappanavar Rudragouda, Jonsson Colleen B, Smith Amber M

Increased virus dissemination leads to enhanced lung injury but not inflammation during influenza-associated secondary bacterial infection.

病毒扩散加剧会导致流感相关继发性细菌感染期间肺损伤加重,但不会导致炎症

Smith Amanda P, Lane Lindey C, Ramirez Zuniga Ivan, Moquin David M, Vogel Peter, Smith Amber M

Time-Dependent Increase in Susceptibility and Severity of Secondary Bacterial Infection during SARS-CoV-2 Infection.

SARS-CoV-2 感染期间继发细菌感染的易感性和严重程度随时间增加

Smith Amanda P, Williams Evan P, Plunkett Taylor R, Selvaraj Muneeswaran, Lane Lindey C, Zalduondo Lillian, Xue Yi, Vogel Peter, Channappanavar Rudragouda, Jonsson Colleen B, Smith Amber M

Dynamically linking influenza virus infection kinetics, lung injury, inflammation, and disease severity.

动态关联流感病毒感染动力学、肺损伤、炎症和疾病严重程度

Myers Margaret A, Smith Amanda P, Lane Lindey C, Moquin David J, Aogo Rosemary, Woolard Stacie, Thomas Paul, Vogel Peter, Smith Amber M

COVID-19 virtual patient cohort suggests immune mechanisms driving disease outcomes

新冠肺炎虚拟患者队列研究提示免疫机制可能驱动疾病结局

Jenner, Adrianne L; Aogo, Rosemary A; Alfonso, Sofia; Crowe, Vivienne; Deng, Xiaoyan; Smith, Amanda P; Morel, Penelope A; Davis, Courtney L; Smith, Amber M; Craig, Morgan

Time to revisit the endpoint dilution assay and to replace the TCID50 as a measure of a virus sample's infection concentration

是时候重新审视终点稀释法,并用其取代 TCID50 作为病毒样本感染浓度的衡量标准了。

Cresta, Daniel; Warren, Donald C; Quirouette, Christian; Smith, Amanda P; Lane, Lindey C; Smith, Amber M; Beauchemin, Catherine A A

Influenza Virus Infection Model With Density Dependence Supports Biphasic Viral Decay.

具有密度依赖性的流感病毒感染模型支持双相病毒衰减

Smith Amanda P, Moquin David J, Bernhauerova Veronika, Smith Amber M

A Critical, Nonlinear Threshold Dictates Bacterial Invasion and Initial Kinetics During Influenza

关键的非线性阈值决定流感期间的细菌入侵和初始动力学

Amber M Smith, Amanda P Smith