日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Protein kinase C epsilon-mediated modulation of T-type calcium channels underlies alcohol withdrawal hyperexcitability in the midline thalamus.

蛋白激酶 C ε 介导的 T 型钙通道调节是中线丘脑酒精戒断过度兴奋的基础

Shan Hong Qu, Smith Thuy, Klorig David C, Godwin Dwayne W

Efficacy of FGFR Inhibitors and Combination Therapies for Acquired Resistance in FGFR2-Fusion Cholangiocarcinoma

FGFR 抑制剂和联合疗法对 FGFR2 融合胆管癌获得性耐药的疗效

Melanie A Krook, Alexandria Lenyo, Max Wilberding, Hannah Barker, Mikayla Dantuono, Kelly M Bailey, Hui-Zi Chen, Julie W Reeser, Michele R Wing, Jharna Miya, Eric Samorodnitsky, Amy M Smith, Thuy Dao, Dorrelyn M Martin, Kristen K Ciombor, John Hays, Aharon G Freud, Sameek Roychowdhury

Characterization of a KLK2-FGFR2 fusion gene in two cases of metastatic prostate cancer

两例转移性前列腺癌中 KLK2-FGFR2 融合基因的表征

Melanie A Krook, Hannah Barker, Hui-Zi Chen, Julie W Reeser, Michele R Wing, Dorrelyn Martin, Amy M Smith, Thuy Dao, Russell Bonneville, Eric Samorodnitsky, Jharna Miya, Aharon G Freud, J Paul Monk, Steven K Clinton, Sameek Roychowdhury

Tumor heterogeneity and acquired drug resistance in FGFR2-fusion-positive cholangiocarcinoma through rapid research autopsy

通过快速研究尸检了解 FGFR2 融合阳性胆管癌的肿瘤异质性和获得性耐药性

Melanie A Krook #, Russell Bonneville #, Hui-Zi Chen, Julie W Reeser, Michele R Wing, Dorrelyn M Martin, Amy M Smith, Thuy Dao, Eric Samorodnitsky, Anoosha Paruchuri, Jharna Miya, Kaitlin R Baker, Lianbo Yu, Cynthia Timmers, Kristin Dittmar, Aharon G Freud, Patricia Allenby, Sameek Roychowdhury

Expression of channelrhodopsin-2 localized within the deep CA1 hippocampal sublayer in the Thy1 line 18 mouse

Thy1 18 号小鼠海马 CA1 深层亚层中通道视紫红质-2 的表达

Dobbins, Dorothy L; Klorig, David C; Smith, Thuy; Godwin, Dwayne W

Expression profiling reveals differential gene induction underlying specific and non-specific memory for pheromones in mice

表达谱分析揭示了小鼠对信息素的特异性和非特异性记忆背后不同的基因诱导机制。

Upadhya, Sudarshan C; Smith, Thuy K; Brennan, Peter A; Mychaleckyj, Josyf C; Hegde, Ashok N

Proteasome inhibition enhances the induction and impairs the maintenance of late-phase long-term potentiation

蛋白酶体抑制增强了晚期长时程增强作用的诱导,但损害了其维持。

Dong, Chenghai; Upadhya, Sudarshan C; Ding, Lan; Smith, Thuy K; Hegde, Ashok N